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Epistatic effect of TLR3 and cGAS‐STING‐IKKε‐TBK1‐IFN signaling variants on colorectal cancer risk

https://doi.org/10.1002/cam4.2804
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1 of 40 checkable references need attention · checked 2026-07-23

At the dated check, the references listed below either did not resolve in Crossref or DataCite, or carried a retraction notice. Each one is shown with the registry record that put it there.

3 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

References needing attention

marked retracted — notice via Crossref, record curated by Retraction Watch10.1159/000320276
Inflammatory Bowel Disease as a Risk Factor for Colorectal Cancer
The 39 checked references that resolve
resolves10.3322/caac.21492
Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries
resolves10.5217/ir.2018.16.3.327
Genetic and epigenetic alterations of colorectal cancer
resolves10.1002/ijc.30583
Concordant and discordant familial cancer: Familial risks, proportions and population impact
resolves10.1038/nrc.2017.82
Genome-wide association studies of cancer: current insights and future perspectives
resolves10.1038/s41588-018-0286-6
Discovery of common and rare genetic risk variants for colorectal cancer
resolves10.7150/jca.15602
<i>XPG</i> Gene Polymorphisms Contribute to Colorectal Cancer Susceptibility: A Two-Stage Case-Control Study
resolves10.3390/genes7100073
Association of XPC Gene Polymorphisms with Colorectal Cancer Risk in a Southern Chinese Population: A Case-Control Study and Meta-Analysis
resolves10.1016/j.gendis.2016.03.004
Gut microbiota, inflammation and colorectal cancer
resolves10.1016/j.ccell.2018.03.004
The Intestinal Microbiota in Colorectal Cancer
resolves10.1016/j.cell.2008.05.009
Regulatory T Cells and Immune Tolerance
resolves10.4161/onci.27763
Toll-like receptor genetic variants and colorectal cancer
resolves10.2174/1566524033479726
Regulation of Dendritic Cell Function Through Toll-like Receptors
resolves10.1002/emmm.201100160
Regulation of innate immune signalling pathways by the tripartite motif (TRIM) family proteins
resolves10.1016/j.bbadis.2014.04.030
Interferon regulatory factors: at the crossroads of immunity, metabolism, and disease
resolves10.1196/annals.1443.020
Toll‐like Receptor and RIG‐1‐like Receptor Signaling
resolves10.1038/nature03326
Toll-like receptor 3 promotes cross-priming to virus-infected cells
resolves10.1074/jbc.M116.771964
Cytosolic DNA Promotes Signal Transducer and Activator of Transcription 3 (STAT3) Phosphorylation by TANK-binding Kinase 1 (TBK1) to Restrain STAT3 Activity
resolves10.1159/000452281
DNA-Sensing and Nuclease Gene Expressions as Markers for Colorectal Cancer Progression
resolves10.3390/ijms18112456
cGAS/STING Pathway in Cancer: Jekyll and Hyde Story of Cancer Immune Response
resolves10.3892/or.2013.2322
The expression and function of Toll-like receptors 3 and 9 in human colon carcinoma
resolves10.1002/ijc.26314
Toll‐like receptor genes and their association with colon and rectal cancer development and prognosis
resolves10.1016/j.ejca.2010.12.011
TLR-3 polymorphism is an independent prognostic marker for stage II colorectal cancer
resolves10.1371/journal.pone.0111061
Single Nucleotide Polymorphisms within Interferon Signaling Pathway Genes Are Associated with Colorectal Cancer Susceptibility and Survival
resolves10.1371/journal.pone.0192385
Investigation of single and synergic effects of NLRC5 and PD-L1 variants on the risk of colorectal cancer
resolves10.1038/s41598-017-08946-3
Association between taste receptor (TAS) genes and the perception of wine characteristics
resolves10.1093/jnci/djh075
Assessing the Probability That a Positive Report is False: An Approach for Molecular Epidemiology Studies
resolves10.1177/0394632017716048
Profile of gene expression of TLR-signaling pathways in colorectal cancer tissues
resolves10.1016/j.celrep.2013.05.009
The Innate Immune DNA Sensor cGAS Produces a Noncanonical Cyclic Dinucleotide that Activates Human STING
resolves10.1073/pnas.1705499114
cGAS is essential for cellular senescence
resolves10.1093/jnci/djy099
Novel Common Genetic Susceptibility Loci for Colorectal Cancer
resolves10.1093/annonc/mdv540
Implications of polygenic risk for personalised colorectal cancer screening
resolves10.1038/ng.2756
Systematic identification of trans eQTLs as putative drivers of known disease associations
resolves10.3748/wjg.v20.i8.1961
New genes emerging for colorectal cancer predisposition
resolves10.1093/hmg/ddu058
New insights into POLE and POLD1 germline mutations in familial colorectal cancer and polyposis
resolves10.14348/molcells.2014.2353
NF-κB in Cellular Senescence and Cancer Treatment
resolves10.1038/sj.onc.1209942
Nuclear factor-κB and inhibitor of κB kinase pathways in oncogenic initiation and progression
resolves10.1007/82_2010_102
NF-κB and Innate Immunity
resolves10.3892/or.2013.2884
Expression of early growth response-1 in colorectal cancer and its relation to tumor cell proliferation and apoptosis
resolves10.1136/gutjnl-2014-308227
Batf-dependent Th17 cells critically regulate IL-23 driven colitis-associated colon cancer
The 3 references without a DOI — listed, not checked
no DOI — not checkedToll‐like receptors and the tendency of normal mucous membrane to transform to polyp or colorectal cancer
no DOI — not checkedColorectal cancer and NF‐kappaB signaling pathway
no DOI — not checkedTranscription factor YY1 expression in human gastrointestinal cancer cells
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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