Reference health
Natalizumab treatment perturbs memory‐ and marginal zone‐like B‐cell homing in secondary lymphoid organs in multiple sclerosis
42/42 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
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The 42 checked references that resolve
resolves10.1001/archneur.65.12.noc80051
Decrease in the Numbers of Dendritic Cells and CD4+ T Cells in Cerebral Perivascular Spaces Due to Natalizumab
resolves10.1002/ana.20858
Immune surveillance in multiple sclerosis patients treated with natalizumab
resolves10.1038/356063a0
Prevention of experimental autoimmune encephalomyelitis by antibodies against α4βl integrin
resolves10.1073/pnas.0808909106
β
<sub>1</sub>
integrins differentially control extravasation of inflammatory cell subsets into the CNS during autoimmunity
resolves10.1182/blood-2007-09-112052
Increased numbers of circulating hematopoietic stem/progenitor cells are chronically maintained in patients treated with the CD49d blocking antibody natalizumab
resolves10.1182/blood-2007-10-120329
The monoclonal anti–VLA-4 antibody natalizumab mobilizes CD34+ hematopoietic progenitor cells in humans
resolves10.1038/bmt.2009.381
CD49d blockade by natalizumab in patients with multiple sclerosis affects steady-state hematopoiesis and mobilizes progenitors with a distinct phenotype and function
resolves10.1212/01.wnl.0000327671.91357.96
Natalizumab disproportionately increases circulating pre-B and B cells in multiple sclerosis
resolves10.1038/bmt.2010.328
CD49d blockade by natalizumab therapy in patients with multiple sclerosis increases immature B-lymphocytes
resolves10.1016/j.jneuroim.2011.02.010
Novel mechanisms of immune modulation of natalizumab in multiple sclerosis patients
resolves10.1159/000302687
Effects of Natalizumab on Circulating B Cells, T Regulatory Cells and Natural Killer Cells
resolves10.1056/NEJMoa051782
Progressive Multifocal Leukoencephalopathy Complicating Treatment with Natalizumab and Interferon Beta-1a for Multiple Sclerosis
resolves10.1056/NEJMoa051847
Progressive Multifocal Leukoencephalopathy in a Patient Treated with Natalizumab
resolves10.1016/S1474-4422(10)70006-5
Immune responses to JC virus in patients with multiple sclerosis treated with natalizumab: a cross-sectional and longitudinal study
resolves10.1016/S1474-4422(10)70040-5
Progressive multifocal leukoencephalopathy and other disorders caused by JC virus: clinical features and pathogenesis
resolves10.1086/597126
Prevalence of Polyomavirus BK and JC Infection and Replication in 400 Healthy Blood Donors
resolves10.1146/annurev.med.080708.082655
Progressive Multifocal Leukoencephalopathy in Patients on Immunomodulatory Therapies
resolves10.1056/NEJM198802043180507
Involvement of JC Virus–Infected Mononuclear Cells from the Bone Marrow and Spleen in the Pathogenesis of Progressive Multifocal Leukoencephalopathy
resolves10.1002/jnr.490270405
Glial cells of the human developing brain and B cells of the immune system share a common DNA binding factor for recognition of the regulatory sequences of the human polyomavirus, JCV
resolves10.1128/JVI.70.10.7004-7012.1996
JC virus infection of hematopoietic progenitor cells, primary B lymphocytes, and tonsillar stromal cells: implications for viral latency
resolves10.1016/0042-6822(92)90909-9
Interaction of the human polyomavirus, JCV, with human B-lymphocytes
resolves10.1177/1352458510385834
CD34+ progenitor cells mobilized by natalizumab are not a relevant reservoir for JC virus
resolves10.1086/597117
Detection of JC Virus DNA and Proteins in the Bone Marrow of HIV‐Positive and HIV‐Negative Patients: Implications for Viral Latency and Neurotropic Transformation
resolves10.1086/344280
Traffic of JC Virus from Sites of Initial Infection to the Brain: The Path to Progressive Multifocal Leukoencephalopathy
resolves10.1002/jlb.65.4.428
Viral variant nucleotide sequences help expose leukocytic positioning in the JC virus pathway to the CNS
resolves10.1001/archneur.63.10.1383
Altered CD4+/CD8+ T-Cell Ratios in Cerebrospinal Fluid of Natalizumab-Treated Patients With Multiple Sclerosis
resolves10.4049/jimmunol.128.2.844
Differences in the migration of B and T lymphocytes: organ-selective localization <i>in vivo</i> and the role of lymphocyte-endothelial cell recognition.
resolves10.4049/jimmunol.160.10.5113
Intrinsic Differences in L-Selectin Expression Levels Affect T and B Lymphocyte Subset-Specific Recirculation Pathways
resolves10.1126/science.1071632
Integrin-Mediated Long-Term B Cell Retention in the Splenic Marginal Zone
resolves10.1038/337179a0
The mucosal vascular addressin is a tissue-specific endothelial cell adhesion molecule for circulating lymphocytes
resolves10.1084/jem.182.2.559
Analysis of mutations in immunoglobulin heavy chain variable region genes of microdissected marginal zone (MGZ) B cells suggests that the MGZ of human spleen is a reservoir of memory B cells.
resolves10.1177/1352458510383075
Adhesion molecules are promising candidates to establish surrogate markers for natalizumab treatment
resolves10.4049/jimmunol.1001413
Regulation of Follicular B Cell Differentiation by the Related E26 Transformation-Specific Transcription Factors PU.1, Spi-B, and Spi-C
resolves10.1099/vir.0.023184-0
Transcription factor Spi-B binds unique sequences present in the tandem repeat promoter/enhancer of JC virus and supports viral activity
resolves10.3109/13550289909021994
Detection and typing of JC virus in autopsy brains and extraneural organs of AIDS patients and non-immunocompromised individuals
resolves10.1128/JVI.00609-10
JC Virus Latency in the Brain and Extraneural Organs of Patients with and without Progressive Multifocal Leukoencephalopathy
resolves10.1002/ana.20703
Diagnostic criteria for multiple sclerosis: 2005 revisions to the “McDonald Criteria”
The 1 reference without a DOI — listed, not checked
no DOI — not checkedExpression of the mucosal vascular addressin, MAdCAM‐1, on sinus‐lining cells in the spleen
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