Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 52 checked references that resolve
resolves10.1074/jbc.M114.610758The Functional Differences between Pro-survival and Pro-apoptotic B Cell Lymphoma 2 (Bcl-2) Proteins Depend on Structural Differences in Their Bcl-2 Homology 3 (BH3) Domains
resolves10.1038/cdd.2011.17The role of Bcl-2 and its pro-survival relatives in tumourigenesis and cancer therapy
resolves10.18632/oncotarget.15751Genetic polymorphisms of Bcl-2 promoter in cancer susceptibility and prognosis: a meta-analysis
resolves10.1101/gr.9.8.677dbSNP—Database for Single Nucleotide Polymorphisms and Other Classes of Minor Genetic Variation
resolves10.1038/srep15496A parabolic model of drag coefficient for storm surge simulation in the South China Sea
resolves10.1093/carcin/bgm172Single-nucleotide polymorphisms at the TP53-binding or responsive promoter regions of BAX and BCL2 genes and risk of squamous cell carcinoma of the head and neck
resolves10.1007/s00726-011-1108-7In silico investigation of molecular mechanism of laminopathy caused by a point mutation (R482W) in lamin A/C protein
resolves10.1016/j.compbiolchem.2022.107669In-silico analysis of non-synonymous single nucleotide polymorphisms in human β-defensin type 1 gene reveals their impact on protein-ligand binding sites
resolves10.7554/eLife.20352Computationally designed high specificity inhibitors delineate the roles of BCL2 family proteins in cancer
resolves10.3390/cells9051287Inhibition of Anti-Apoptotic Bcl-2 Proteins in Preclinical and Clinical Studies: Current Overview in Cancer
resolves10.1038/nprot.2009.86Predicting the effects of coding non-synonymous variants on protein function using the SIFT algorithm
resolves10.1093/nar/gks539SIFT web server: predicting effects of amino acid substitutions on proteins
resolves10.1002/prot.22229Improving NMR protein structure quality by Rosetta refinement: A molecular replacement study
resolves10.1021/acsomega.0c01295Novel Approach for Characterizing Propofol Binding Affinities to Serum Albumins from Different Species
resolves10.1093/nar/gkaa275MusiteDeep: a deep-learning based webserver for protein post-translational modification site prediction and visualization
resolves10.1186/gb-2008-9-s1-s4GeneMANIA: a real-time multiple association network integration algorithm for predicting gene function
resolves10.1002/prot.10379Evaluation of protein docking predictions using <i>Hex</i> 3.1 in CAPRI rounds 1 and 2
resolves10.1002/jcc.540130805Settle: An analytical version of the SHAKE and RATTLE algorithm for rigid water models
resolves10.3389/fchem.2021.736509Scaffold Searching of FDA and EMA-Approved Drugs Identifies Lead Candidates for Drug Repurposing in Alzheimer’s Disease
resolves10.1098/rsif.2015.0915Correction to ‘Biophysics of protein evolution and evolutionary protein biophysics’
resolves10.1093/nar/gkw937The STRING database in 2017: quality-controlled protein–protein association networks, made broadly accessible
resolves10.1504/IJCBDD.2013.056801Multidrug resistance protein P-gp interaction with nanoparticles (fullerenes and carbon nanotube) to assess their drug delivery potential: a theoretical molecular docking study
resolves10.1371/journal.pcbi.1007485Alterations of the interactome of Bcl-2 proteins in breast cancer at the transcriptional, mutational and structural level
resolves10.1073/pnas.1015328108Arginine methylation of BCL-2 antagonist of cell death (BAD) counteracts its phosphorylation and inactivation by Akt
resolves10.1167/iovs.63.3.2The NLRP1 Inflammasome Induces Pyroptosis in Human Corneal Epithelial Cells
resolves10.1073/pnas.0809414106Mechanism of Bcl-2 and Bcl-X
<sub>L</sub>
inhibition of NLRP1 inflammasome: Loop domain-dependent suppression of ATP binding and oligomerization
resolves10.1016/j.jmgm.2017.11.011Exploring the selectivity of inhibitor complexes with Bcl-2 and Bcl-XL: A molecular dynamics simulation approach
resolves10.1002/mgg3.910Docking‐based approach for identification of mutations that disrupt binding between Bcl‐2 and Bax proteins: Inducing apoptosis in cancer cells
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