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Memory Reconsolidation

https://doi.org/10.1007/7854_2016_463
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The 185 checked references that resolve
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Phase-Dependent Molecular Requirements for Memory Reconsolidation: Differential Roles for Protein Synthesis and Protein Kinase A Activity
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The IκB Kinase Regulates Chromatin Structure during Reconsolidation of Conditioned Fear Memories
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PKMζ maintains memories by regulating GluR2-dependent AMPA receptor trafficking
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Post-Retrieval Extinction Attenuates Cocaine Memories
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Preventing the return of fear in humans using reconsolidation update mechanisms
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Amnesia: A Function of the Temporal Relation of Footshock to Electroconvulsive Shock
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Neural Signature of Reconsolidation Impairments by Propranolol in Humans
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PKMζ Maintains Spatial, Instrumental, and Classically Conditioned Long-Term Memories
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Enhancement of Consolidated Long-Term Memory by Overexpression of Protein Kinase Mζ in the Neocortex
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Reconsolidation may incorporate state-dependency into previously consolidated memories
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Removal of Perineuronal Nets in the Medial Prefrontal Cortex Impairs the Acquisition and Reconsolidation of a Cocaine-Induced Conditioned Place Preference Memory
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Memory Reconsolidation and Extinction Have Distinct Temporal and Biochemical Signatures
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The consolidation of new but not reactivated memory requires hippocampal C/EBPβ
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Induction of the learning and plasticity‐associated gene <i>Zif268</i> following exposure to a discrete cocaine‐associated stimulus
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Molecular mechanisms of memory reconsolidation
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Bidirectional behavioral plasticity of memory reconsolidation depends on amygdalar protein kinase A
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Cellular and systems mechanisms of memory strength as a constraint on auditory fear reconsolidation
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Differential Involvement of Brain-Derived Neurotrophic Factor in Reconsolidation and Consolidation of Conditioned Taste Aversion Memory
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Extracellular Signal-Regulated Kinase in the Basolateral Amygdala, but not the Nucleus Accumbens Core, is Critical for Context-Response-Cocaine Memory Reconsolidation in Rats
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Older and stronger object memories are selectively destabilized by reactivation in the presence of new information
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On the Dynamic Nature of the Engram: Evidence for Circuit-Level Reorganization of Object Memory Traces following Reactivation
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