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HSV-1 Biology and Life Cycle

https://doi.org/10.1007/978-1-4939-0428-0_1
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35/35 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

13 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 35 checked references that resolve
resolves10.1128/JVI.76.4.1537-1547.2002
Herpesvirus Assembly and Egress
resolves10.1099/vir.0.012492-0
Characterization of the herpes simplex virus (HSV)-1 tegument protein VP1-2 during infection with the HSV temperature-sensitive mutant tsB7
resolves10.1128/JVI.01913-12
A Network of Protein Interactions around the Herpes Simplex Virus Tegument Protein VP22
resolves10.1128/JVI.01239-07
Replication-Competent Herpes Simplex Virus 1 Isolates Selected from Cells Transfected with a Bacterial Artificial Chromosome DNA Lacking Only the U <sub>L</sub> 49 Gene Vary with Respect to the Defect in the U <sub>L</sub> 41 Gene Encoding Host Shutoff RNase
resolves10.1099/vir.0.048900-0
Regulation of alphaherpesvirus infections by the ICP0 family of proteins
resolves10.1128/JVI.78.5.2169-2178.2004
Role of ICP0 in the Strategy of Conquest of the Host Cell by Herpes Simplex Virus 1
resolves10.1080/15216540310001612237
The Role of DNA Recombination in Herpes Simplex Virus DNA Replication
resolves10.1093/emboj/cdf458
Visualization of parental HSV‐1 genomes and replication compartments in association with ND10 in live infected cells
resolves10.1111/cmi.12044
The way out: what we know and do not know about herpesvirus nuclear egress
resolves10.1016/j.virusres.2005.04.017
Towards an understanding of the molecular basis of herpes simplex virus latency
resolves10.1111/j.1574-6976.2011.00320.x
The molecular basis of herpes simplex virus latency
resolves10.1038/nrmicro1794
Chromatin control of herpes simplex virus lytic and latent infection
resolves10.1371/journal.ppat.1002852
HSV-1 Genome Subnuclear Positioning and Associations with Host-Cell PML-NBs and Centromeres Regulate LAT Locus Transcription during Latency in Neurons
resolves10.1038/nature07103
MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAs
resolves10.1128/JVI.00504-13
Mutational Inactivation of Herpes Simplex Virus 1 MicroRNAs Identifies Viral mRNA Targets and Reveals Phenotypic Effects in Culture
resolves10.1128/JVI.00652-12
Influence of Herpes Simplex Virus 1 Latency-Associated Transcripts on the Establishment and Maintenance of Latency in the ROSA26R Reporter Mouse Model
resolves10.1099/vir.0.2008/005066-0
A historical analysis of herpes simplex virus promoter activation in vivo reveals distinct populations of latently infected neurones
resolves10.1099/vir.0.034728-0
An investigation of herpes simplex virus promoter activity compatible with latency establishment reveals VP16-independent activation of immediate-early promoters in sensory neurones
resolves10.1093/infdis/jiq035
Persistent Genital Herpes Simplex Virus-2 Shedding Years Following the First Clinical Episode
resolves10.1038/nm.3103
Rapid host immune response and viral dynamics in herpes simplex virus-2 infection
resolves10.1128/JVI.00785-09
Epigenetic Modulation of Gene Expression from Quiescent Herpes Simplex Virus Genomes
resolves10.1128/JVI.02263-10
Reversal of Heterochromatic Silencing of Quiescent Herpes Simplex Virus Type 1 by ICP0
resolves10.1128/jvi.63.8.3513-3515.1989
Herpes simplex virus type 1 immediate-early protein Vmw110 reactivates latent herpes simplex virus type 2 in an in vitro latency system
resolves10.1099/vir.0.83391-0
Interferons and viruses: an interplay between induction, signalling, antiviral responses and virus countermeasures
resolves10.1073/pnas.1211302109
Nuclear IFI16 induction of IRF-3 signaling during herpesviral infection and degradation of IFI16 by the viral ICP0 protein
resolves10.1084/jem.20061792
Virus-specific CD8+ T cells accumulate near sensory nerve endings in genital skin during subclinical HSV-2 reactivation
resolves10.1038/nature12110
Immune surveillance by CD8αα+ skin-resident T cells in human herpes virus infection
resolves10.1038/375411a0
Herpes simplex virus turns off the TAP to evade host immunity
resolves10.1128/JVI.02265-10
Activities of ICP0 Involved in the Reversal of Silencing of Quiescent Herpes Simplex Virus 1
resolves10.1073/pnas.0707266104
Herpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST–REST complex
resolves10.1128/JVI.01940-08
The Two Functions of Herpes Simplex Virus 1 ICP0, Inhibition of Silencing by the CoREST/REST/HDAC Complex and Degradation of PML, Are Executed in Tandem
resolves10.1128/JVI.02950-12
Components of Promyelocytic Leukemia Nuclear Bodies (ND10) Act Cooperatively To Repress Herpesvirus Infection
resolves10.1038/nrmicro3015
Interplay between viruses and host sumoylation pathways
resolves10.1016/j.bbamcr.2008.08.004
New insights into the role of the subnuclear structure ND10 for viral infection
resolves10.1371/journal.ppat.1002123
SUMO Pathway Dependent Recruitment of Cellular Repressors to Herpes Simplex Virus Type 1 Genomes
The 13 references without a DOI — listed, not checked
no DOI — not checkedWeller SK (2011) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK
no DOI — not checkedKnipe DM, Howley PM, Griffin DE, Lamb RA, Martin MA, Roizman B, Strauss SE (2006) Fields virology. Lippincott Williams and Wilkins, Philadelphia, PA
no DOI — not checkedEisenberg RJ, Heldwein EE, Cohen GH, Krummenacher C (2011) Recent progress in understanding herpes simplex virus entry: relationship of structure to function. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 131–152
no DOI — not checkedDeLuca NA (2011) Functions and mechanism of action of the herpes simplex virus regulatory protein, ICP4. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 17–38
no DOI — not checkedSandri-Goldin RM (2011) The functions and activities of HSV-1 ICP27, a multifunctional regulator of gene expression. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 39–50
no DOI — not checkedEverett RD (2006) The roles of ICP0 during HSV-1 infection. In: Sandri-Goldin RM (ed) Alpha herpesviruses. Molecular and cellular biology. Caister Academic Press, Wymondham, pp 39–64
no DOI — not checkedEverett RD (2011) The role of ICP0 in counteracting intrinsic cellular resistance to virus infection. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 51–72
no DOI — not checkedRice SA (2011) Multiple roles of immediate-early protein ICP22 in HSV-1 replication. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 73–88
no DOI — not checkedWard SA, Weller SK (2011) HSV-1 DNA replication. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 89–112
no DOI — not checkedConway JF, Homa FL (2011) Nucleocapsid structure, assembly and DNA packaging of herpes simplex virus. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 175–194
no DOI — not checkedBloom DC, Kwiatkowski DL (2011) HSV-1 latency and the roles of LATs. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 295–316
no DOI — not checkedSobol PT, Mossman KL (2011) Mechanisms of subversion of type I interferon responses by alphaherpesviruses. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 219–336
no DOI — not checkedJerome KR (2011) Immunity to herpes simplex virus. In: Weller SK (ed) Alphaherpesviruses. Molecular virology. Caister Academic Press, Norfolk, UK, pp 331–350
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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