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Amyloid β-peptide and oxidative cellular injury in Alzheimer’s disease

84/84 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

7 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 84 checked references that resolve

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β-Amyloid and the Pathogenesis of Alzheimer's Disease

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Hydrogen peroxide mediates amyloid β protein toxicity

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Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca2+ homeostasis and cell death

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β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease

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Candidate Gene for the Chromosome 1 Familial Alzheimer's Disease Locus

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Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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Secreted Forms of β-Amyloid Precursor Protein Protect Hippocampal Neurons against Amyloid β-Peptide-Induced Oxidative Injury

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β-Amyloid Peptide Free Radical Fragments Initiate Synaptosomal Lipoperoxidation in a Sequence-Specific Fashion: Implications to Alzheimer′s Disease

resolves10.1016/0024-3205(94)00390-4

Role of protein oxidation in aging and in age-associated neurodegenerative diseases

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Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

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Soluble β-Amyloid Induction of Alzheimer's Phenotype for Human Fibroblast K + Channels

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Tumor necrosis factors alpha and beta protect neurons against amyloid beta-peptide toxicity: evidence for involvement of a kappa B-binding factor and attenuation of peroxide and Ca2+ accumulation.

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Neurotrophic Factors Attenuate Glutamate‐Induced Accumulation of Peroxides, Elevation of Intracellular Ca2+ Concentration, and Neurotoxicity and Increase Antioxidant Enzyme Activities in Hippocampal Neurons

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Tumor necrosis factors protect neurons against metabolic-excitotoxic insults and promote maintenance of calcium homeostasis

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Activation of K+ channels and suppression of neuronal activity by secreted β-amyloid-precursor protein

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β-Amyloid peptide-derived, oxygen-dependent free radicals inhibit glutamate uptake in cultured astrocytes: implications for Alzheimerʼs disease

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Amyloid β‐Protein Induces Its Own Production in Cultured Degenerating Cerebrovascular Smooth Muscle Cells

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Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state

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A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease.

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Deposits of Aβ fibrils are not toxic to cortical and hippocampal neurons in vitro

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Isozymes of the Na+/K+-ATPase

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The Ca2+ pump of the plasma membrane.

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Cholesterol oxidation reduces Ca2+ + Mg2+-ATPase activity, interdigitation, and increases fluidity of brain synaptic plasma membranes

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Oxidation of membrane cholesterol alters active and passive transsarcolemmal calcium movement.

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Autopsy Samples of Alzheimer's Cortex Show Increased Peroxidation In Vitro

resolves10.1111/j.1749-6632.1994.tb44404.x

Alzheimer's Disease: Membrane‐Associated Metabolic Changesa

resolves10.1016/0006-8993(90)91355-K

β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

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Potentiation of NMDA receptor currents by arachidonic acid

resolves10.1046/j.1471-4159.1994.62010372.x

Rapid Communication: Ca2+ Channel Blockers Attenuate β‐Amyloid Peptide Toxicity to Cortical Neurons in Culture

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β‐Amyloid Neurotoxicity in Human Cortical Culture Is Not Mediated by Excitotoxins

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Anticonvulsants attenuate amyloid β-peptide neurotoxicity, Ca2+ deregulation, and cytoskeletal pathology

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The Intracellular Component of Cellular 3‐(4,5‐Dimethylthiazol‐2‐yl)‐2,5‐Diphenyltetrazolium Bromide (MTT) Reduction Is Specifically Inhibited by β‐Amyloid Peptides

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Cholinergic and serotonergic stimulation of phosphoinositide hydrolysis is decreased in Alzheimer's disease

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Amyloid beta-peptide disrupts carbachol-induced muscarinic cholinergic signal transduction in cortical neurons.

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Cholinergic Neural Regulation of Regional Cerebral Blood Flow

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Chapter 29 Nitric oxide in the central nervous system

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β-Amyloid 25–35 activates nitric oxide synthase in a neuronal clone

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Inhibition of purified (Na+,K+)-ATPase activity from porcine cerebral cortex by NO generating drugs

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Pathological alterations of the cerebral microvasculature in Alzheimer's disease and related dementing disorders

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Morphometric comparison of hippocampal microvasculature in ageing and demented people: Diameters and densities

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The relationship between senile plaques and cerebral blood vessels in Alzheimer’s disease and senile dementia

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Differential degeneration of the cerebral microvasculature in Alzheimerʼs disease

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Degeneration of vascular muscle cells in cerebral amyloid angiopathy of Alzheimer disease

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Microvascular pathology and vascular basement membrane components in Alzheimer's disease

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Passage of human amyloid β-protein 1–40 across the murine blood-brain barrier

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Characterization of Alzheimer amyloid precursor protein transcripts in platelets and megakarocytes

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Excessive production of amyloid beta-protein by peripheral cells of symptomatic and presymptomatic patients carrying the Swedish familial Alzheimer disease mutation.

resolves10.1073/pnas.86.19.7606

Interleukin 1 regulates synthesis of amyloid beta-protein precursor mRNA in human endothelial cells.

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Neuritic plaques and cerebrovascular amyloid in Alzheimer disease are antigenically related.

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beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease.

resolves10.1111/j.1471-4159.1989.tb07399.x

Reduced Glucose Transporter at the Blood‐Brain Barrier and in Cerebral Cortex in Alzheimer Disease

resolves10.1038/380168a0

β-Amyloid-mediated vasoactivity and vascular endothelial damage

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Microglial interleukin-lα expression in human head injury: Correlations with neuronal and neuritic β-amyloid precursor protein expression

resolves10.1016/0006-8993(92)90380-R

β-Amyloid stimulates glial cells in vitro to produce growth factors that accumulate in senile plaques in Alzheimer's disease

resolves10.1073/pnas.86.19.7611

Brain interleukin 1 and S-100 immunoreactivity are elevated in Down syndrome and Alzheimer disease.

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Active microglia, sick astroglia and Alzheimer type dementias

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Glial cytokines in Alzheimer's disease: Review and pathogenic implications

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Interleukin-1 beta attenuates excitatory amino acid-induced neurodegeneration in vitro: involvement of nerve growth factor

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Interleukin-1 injected into mammalian brain stimulates astrogliosis and neovascularization

resolves10.1016/0306-4522(94)90547-9

β-Amyloid-induced changes in cultured astrocytes parallel reactive astrocytosis associated with senile plaques in Alzheimer's disease

resolves10.1016/0006-8993(93)91698-R

Synthetic Alzheimer amyloid β/A4 peptides enhance production of complement C3 component by cultured microglial cells

resolves10.1002/jnr.490400407

Complement and cytokine gene expression in cultured microglia derived from postmortem human brains

resolves10.1007/978-1-4615-3432-7_9

Contrasting Effects of Specific Lymphokines on the Survival of Hippocampal Neurons in Culture

resolves10.1016/0006-8993(93)90068-X

β-Amyloid peptides induce degeneration of cultured rat microglia

resolves10.1016/0922-4106(95)90006-3

Differential regulation by β-amyloid peptides of intracellular free Ca2+ concentration in cultured rat microglia

resolves10.4049/jimmunol.152.7.3578

Involvement of the cystine transport system xc- in the macrophage-induced glutamate-dependent cytotoxicity to neurons.

resolves10.1038/nm0796-788

Altered neuronal and microglial responses to excitotoxic and ischemic brain injury in mice lacking TNF receptors

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Molecular profile of reactive astrocytes—Implications for their role in neurologic disease

resolves10.1006/exnr.1993.1202

Neuritic Involvement within bFGf Immunopositive Plaques of Alzheimer's Disease

resolves10.1006/exnr.1994.1185

Regional Differences in Reactive Gliosis Induced by Substrate-Bound β-Amyloid

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Glutamate uptake inhibition by oxygen free radicals in rat cortical astrocytes

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β‐Amyloid protein enhances the mitogen‐induced calcium response in circulating human lymphocytes

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Alzheimer beta A4-amyloid protein precursor in immunocompetent cells.

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β-amyloid peptide decreases membrane fluidity

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Alterations of intracellular calcium regulation during aging and Alzheimer's disease in nonneuronal cells

resolves10.1016/0006-8993(94)90223-2

Staurosporine and K-252 compounds protect hippocampal neurons against amyloid β-peptide toxicity and oxidative injury

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Ceramide Protects Hippocampal Neurons Against Excitotoxic and Oxidative Insults, and Amyloid β‐Peptide Toxicity

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Linoleic acid and TNF-alpha cross-amplify oxidative injury and dysfunction of endothelial cells.

The 7 references without a DOI — listed, not checked

no DOI — not checkedCopani A., Koh J.-Y., and Cotman C. W. (1991) β-amyloid increases neuronal susceptibility to injury by glucose deprivation.Neuroreport 543, 139–147.

no DOI — not checkedBlanc E. M., Toborek M., Mark R. J., and Mattson M. P. (1996) Amyloid β-peptide disrupts barrier and transport functions and induces apoptosis in vascular endothelial cells.J. Neurosci., submitted.

no DOI — not checkedZarow C., Chui H. C., Hsu E., and Perlmutter L. S. (1989) Correlation between microangiopathy and neuronal loss in Alzheimer’s disease.Soc. Neurosci. Abstr. 15, 1376.

no DOI — not checkedFrautschy S. A., Cole G. M., and Baird A. (1992) Phagocytosis and deposition of vascular β-amyloid in rat brains injected with alzheimer β-amyloid.Am. J. Neuropathol. 140, 1389–1399.

no DOI — not checkedStewart P. A., Hayakawa K., Akers M.-A., and Vinters H. V. (1992) A morphometric study of the blood-brain barrier in Alzheimer disease.Lab. Invest. 67, 734–742.

no DOI — not checkedCacabelos R., Alvarez X. A., Fernandez-Novoa L., Franco A., Mangues R., Pellicer A., and Nishimura T. (1994) Brain interleukin-1 beta in Alzheimer’s disease and vascular demintia.Methods Find. Exp. Clin. Pharmacol. 16, 141–151.

no DOI — not checkedGalindo M. F., Prehn J. H. M., Bindokas V. P., and Miller R. J. (1994) Potential role of TGFβ-1 in Alzheimer’s disease; regulation of APP expression and inhibition of βAP neurotoxicity.Soc. Neurosci. Abstr. 14, 1248.

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