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Role of accumbens BDNF and TrkB in cocaine-induced psychomotor sensitization, conditioned-place preference, and reinstatement in rats

https://doi.org/10.1007/s00213-008-1164-1
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38/38 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 38 checked references that resolve
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The Neurotrophins NT-4/5 and BDNF Augment Serotonin, Dopamine, and GABAergic Systems during Behaviorally Effective Infusions to the Substantia Nigra
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<i>In vivo</i> gene delivery of urokinase‐type plasminogen activator with regulatable lentivirus induces behavioural changes in chronic cocaine administration
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CD81‐induced behavioural changes during chronic cocaine administration: <i>in vivo</i> gene delivery with regulatable lentivirus
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Silencing dopamine D<sub>3</sub>‐receptors in the nucleus accumbens shell <i>in vivo</i> induces changes in cocaine‐induced hyperlocomotion
resolves10.1111/j.1471-4159.2004.02961.x
<i>In vivo</i> gene silencing of CD81 by lentiviral expression of small interference RNAs suppresses cocaine‐induced behaviour
resolves10.1111/j.1471-4159.2006.04013.x
Silencing urokinase in the ventral tegmental area <i>in vivo</i> induces changes in cocaine‐induced hyperlocomotion
resolves10.1016/0306-4522(95)00207-Y
Influence of neurotrophic factors on morphine- and cocaine-induced biochemical changes in the mesolimbic dopamine system
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Neurotrophic Mechanisms in Drug Addiction
resolves10.1016/S0959-4388(96)80017-1
Neurotrophins and activity-dependent development of the neocortex
resolves10.1016/0361-9230(93)90102-H
Extinction of cocaine-induced place approach in rats: A validation of the “biased” conditioning procedure
resolves10.1016/j.brainres.2005.11.099
Alterations in BDNF and trkB mRNAs following acute or sensitizing cocaine treatments and withdrawal
resolves10.1038/nn1929
Dynamic BDNF activity in nucleus accumbens with cocaine use increases self-administration and relapse
resolves10.1523/JNEUROSCI.23-03-00742.2003
Time-Dependent Increases in Brain-Derived Neurotrophic Factor Protein Levels within the Mesolimbic Dopamine System after Withdrawal from Cocaine: Implications for Incubation of Cocaine Craving
resolves10.1038/35075076
BDNF controls dopamine D3 receptor expression and triggers behavioural sensitization
resolves10.1038/sj.npp.1300192
Reduced Behavioral Effects of Cocaine in Heterozygous Brain-Derived Neurotrophic Factor (BDNF) Knockout Mice
resolves10.1523/JNEUROSCI.19-10-04110.1999
Enhancement of Locomotor Activity and Conditioned Reward to Cocaine by Brain-Derived Neurotrophic Factor
resolves10.1038/350230a0
BDNF is a neurotrophic factor for dopaminergic neurons of the substantia nigra
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Overlapping and distinct actions of the neurotrophins BDNF, NT-3, and NT-4/5 on cultured dopaminergic and GABAergic neurons of the ventral mesencephalon
resolves10.1093/emboj/19.6.1290
BDNF regulates eating behavior and locomotor activity in mice
resolves10.1038/sj.mp.4000749
Brain Derived Neurotrophic Factor (BDNF) gene variants association with age at onset and therapeutic response in schizophrenia
resolves10.1523/JNEUROSCI.19-14-05823.1999
Subcellular Localization of Full-Length and Truncated Trk Receptor Isoforms in Polarized Neurons and Epithelial Cells
resolves10.1016/j.brainres.2005.10.004
Rodent BDNF genes, novel promoters, novel splice variants, and regulation by cocaine
resolves10.1523/JNEUROSCI.5124-03.2004
A Single Infusion of Brain-Derived Neurotrophic Factor into the Ventral Tegmental Area Induces Long-Lasting Potentiation of Cocaine Seeking after Withdrawal
resolves10.1111/j.1460-9568.1996.tb01313.x
Spontaneous Behaviours of Rats are Differentially Affected by Substantia Nigra Infusions of Brain‐derived Neurotrophic Factor and Neurotrophin‐3
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Amphetamine increases tyrosine kinase-B receptor expression in the dorsal striatum
resolves10.1016/S0006-8993(02)03160-8
Brain-derived neurotrophic factor expression is increased in the rat amygdala, piriform cortex and hypothalamus following repeated amphetamine administration
resolves10.1016/S0896-6273(00)81154-X
Role for GDNF in Biochemical and Behavioral Adaptations to Drugs of Abuse
resolves10.1007/BF00427782
Reinforcing properties of morphine and naloxone revealed by conditioned place preferences: a procedural examination
resolves10.1016/S0166-4328(00)00239-4
Cocaine-induced conditioned place preference: reinstatement by priming injections of cocaine after extinction
resolves10.1016/S0959-4388(00)00208-7
Trk receptors: mediators of neurotrophin action
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Neurotrophin-3 Contributes to the Initiation of Behavioral Sensitization to Cocaine by Activating the Ras/Mitogen-Activated Protein Kinase Signal Transduction Cascade
resolves10.1038/nn1687
BDNF-dependent synaptic sensitization in midbrain dopamine neurons after cocaine withdrawal
resolves10.1038/nn0807-935
A role for BDNF in cocaine reward and relapse
resolves10.1073/pnas.091039298
Molecular mechanisms of cocaine reward: Combined dopamine and serotonin transporter knockouts eliminate cocaine place preference
resolves10.1016/S0079-6123(00)28016-3
Neurotrophins and activity-dependent plasticity
resolves10.1086/324467
Polysubstance Abuse–Vulnerability Genes: Genome Scans for Association, Using 1,004 Subjects and 1,494 Single-Nucleotide Polymorphisms
resolves10.1038/sj.mp.4000964
Cocaine, reward, movement and monoamine transporters
The 1 reference without a DOI — listed, not checked
no DOI — not checkedPaxinos G, Watson C (1998) The rat brain in stereotactic coordinates. New York, Academic
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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