Reference health
Remodelling of human atrial K+ currents but not ion channel expression by chronic β-blockade
31/31 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
6 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 31 checked references that resolve
resolves10.4161/chan.4.3.11479
α1-Adrenoreceptors regulate only the caveolae-located subpopulation of cardiac K<sub>V</sub>4 channels
resolves10.1093/eurheartj/ehq278
Guidelines for the management of atrial fibrillation: The Task Force for the Management of Atrial Fibrillation of the European Society of Cardiology (ESC)
resolves10.1161/01.CIR.0000145659.80212.6A
L-Type Ca
<sup>2+</sup>
Current Downregulation in Chronic Human Atrial Fibrillation Is Associated With Increased Activity of Protein Phosphatases
resolves10.1016/S0008-6363(99)00034-6
Ionic targets for drug therapy and atrial fibrillation-induced electrical remodeling: insights from a mathematical model
resolves10.1016/0167-5273(96)02605-8
The shape of human atrial action potential accounts for different frequency-related changes in vitro
resolves10.1007/s00395-003-0409-8
Remodeling of cardiomyocyte ion channels in human atrial fibrillation
resolves10.1098/rsta.2006.1765
Contributions of inwardly rectifying K
<sup>+</sup>
currents to repolarization assessed using mathematical models of human ventricular myocytes
resolves10.1161/CIRCULATIONAHA.104.506857
Human Atrial Ion Channel and Transporter Subunit Gene-Expression Remodeling Associated With Valvular Heart Disease and Atrial Fibrillation
resolves10.1161/01.RES.87.11.1026
Role of the Calcium-Independent Transient Outward Current
<i>I</i>
<sub>to1</sub>
in Shaping Action Potential Morphology and Duration
resolves10.1007/s00210-007-0174-6
Pharmacological evidence for altered src kinase regulation of I
Ca,L in patients with chronic atrial fibrillation
resolves10.1161/01.CIR.0000029747.53262.5C
Human Cardiac Inwardly-Rectifying K
<sup>+</sup>
Channel Kir
<sub>2.1b</sub>
Is Inhibited by Direct Protein Kinase C-Dependent Regulation in Human Isolated Cardiomyocytes and in an Expression System
resolves10.1172/JCI118358
beta-Adrenergic modulation of the inwardly rectifying potassium channel in isolated human ventricular myocytes. Alteration in channel response to beta-adrenergic stimulation in failing human hearts.
resolves10.1002/j.1460-2075.1996.tb00437.x
TWIK‐1, a ubiquitous human weakly inward rectifying K+ channel with a novel structure.
resolves10.1161/01.RES.78.5.903
Adrenergic Modulation of Ultrarapid Delayed Rectifier K<sup>+</sup>Current in Human Atrial Myocytes
resolves10.1016/j.ehj.2004.06.002
Expert consensus document on ?-adrenergic receptor blockersThe Task Force on Beta-Blockers of the European Society of Cardiology
resolves10.1016/j.cardiores.2005.11.013
Involvement of lipid rafts and caveolae in cardiac ion channel function
resolves10.1152/physrev.00014.2006
Arrhythmogenic Ion-Channel Remodeling in the Heart: Heart Failure, Myocardial Infarction, and Atrial Fibrillation
resolves10.1111/j.1469-7793.2000.t01-1-00285.x
Molecular basis of functional voltage‐gated K<sup>+</sup> channel diversity in the mammalian myocardium
resolves10.1016/j.yjmcc.2009.07.013
Molecular determinants of cardiac transient outward potassium current (Ito) expression and regulation
resolves10.1529/biophysj.105.060459
Ionic Determinants of Functional Reentry in a 2-D Model of Human Atrial Cells During Simulated Chronic Atrial Fibrillation
resolves10.1161/01.RES.48.6.804
Adaptation to prolonged beta-blockade of rabbit atrial, purkinje, and ventricular potentials, and of papillary muscle contraction. Time-course of development of and recovery from adaptation.
resolves10.1016/j.yjmcc.2006.01.012
Anti-adrenergic effects of endothelin on human atrial action potentials are potentially anti-arrhythmic
resolves10.1016/S0022-2828(03)00012-9
The L-type Ca2+-channel subunits α1C and β2 are not downregulated in atrial myocardium of patients with chronic atrial fibrillation
resolves10.1016/S0008-6363(01)00380-7
The contribution of ionic currents to changes in refractoriness of human atrial myocytes associated with chronic atrial fibrillation
resolves10.1016/S0008-6363(03)00263-3
Chronic beta-adrenoceptor blockade and human atrial cell electrophysiology: evidence of pharmacological remodelling
resolves10.1016/j.pharmthera.2011.02.002
Mechanisms of termination and prevention of atrial fibrillation by drug therapy
The 6 references without a DOI — listed, not checked
no DOI — not checkedBru-Mercier G, Deroubaix E, Rousseau D, Coulombe A, Renaud J-F (2002) Depressed transient outward potassium current density in catecholamine-depleted rat ventricular myocytes. Am J Physiol 282:H1237–H1247
no DOI — not checkedCourtemanche M, Ramirez RJ, Nattel S (1998) Ionic mechanisms underlying human atrial action potential properties: insights from a mathematical model. Am J Physiol 275:H301–H321
no DOI — not checkedEscande D, Coulombe A, Faivre JF, Coraboeuf E (1987) Two types of transient outward currents in adult human atrial cells. Am J Physiol 252:H142–H148
no DOI — not checkedFermini B, Wang Z, Duan D, Nattel S (1992) Differences in rate dependence of transient outward current in rabbit and human atrium. Am J Physiol 263:H1747–H1754
no DOI — not checkedKong W, Po S, Yamagishi T, Ashen MD, Stetten G, Tomaselli GF (1998) Isolation and characterization of the human gene encoding I to: further diversity by alternative mRNA splicing. Am J Physiol 275:H1963–H1970
no DOI — not checkedLiu QY, Rosen MR, McKinnon D, Robinson RB (1998) Sympathetic innervation modulates repolarizing K+ currents in rat epicardial myocytes. Am J Physiol 274:H915–H922
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