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T3 and T4 decrease ROS levels and increase endothelial nitric oxide synthase expression in the myocardium of infarcted rats
44/44 checkable references clean · checked 2026-08-09
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 44 checked references that resolve
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Progression of heart failure after myocardial infarction in the rat
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Time course of hydrogen peroxide–thioredoxin balance and its influence on the intracellular signalling in myocardial infarction
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Redox-sensitive prosurvival and proapoptotic protein expression in the myocardial remodeling post-infarction in rats
resolves10.1152/ajpheart.01162.2006
Thiol-based mechanisms of the thioredoxin and glutaredoxin systems: implications for diseases in the cardiovascular system
resolves10.1038/sj.bjp.0706853
Thioredoxin reduces post‐ischemic myocardial apoptosis by reducing oxidative/nitrative stress
resolves10.1006/bbrc.1998.8695
Studies on the Mechanism of Oxidative Modification of Human Glyceraldehyde-3-phosphate Dehydrogenase by Glutathione: Catalysis by Glutaredoxin
resolves10.1161/01.CIR.100.3.292
Endothelial Dysfunction in Chronic Myocardial Infarction Despite Increased Vascular Endothelial Nitric Oxide Synthase and Soluble Guanylate Cyclase Expression
resolves10.1161/hc3601.094298
Endothelial Nitric Oxide Synthase Limits Left Ventricular Remodeling After Myocardial Infarction in Mice
resolves10.1016/j.ijcard.2011.05.078
Adipose tissue-derived stem cells embedded with eNOS restore cardiac function in acute myocardial infarction model
resolves10.1126/science.1079368
Mitochondrial Biogenesis in Mammals: The Role of Endogenous Nitric Oxide
resolves10.1111/j.1582-4934.2010.01014.x
Early long-term L-T3 replacement rescues mitochondria and prevents ischemic cardiac remodelling in rats
resolves10.1007/s00395-008-0697-0
Long-term thyroid hormone administration reshapes left ventricular chamber and improves cardiac function after myocardial infarction in rats
resolves10.1016/j.metabol.2013.05.008
Thyroid hormone improves the mechanical performance of the post-infarcted diabetic myocardium: A response associated with up-regulation of Akt/mTOR and AMPK activation
resolves10.1016/j.mce.2014.04.010
Cardioprotective effects of thyroid hormones in a rat model of myocardial infarction are associated with oxidative stress reduction
resolves10.1097/00000658-195411000-00006
EXPERIMENTAL MYOCARDIAL INFARCTION. I. A. METHOD OF CORONARY OCCLUSION IN SMALL ANIMALS
resolves10.1016/j.mce.2006.02.005
Myocardial antioxidant enzyme activities and concentration and glutathione metabolism in experimental hyperthyroidism
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A função mecânica do miocárdio remanescente a um infarto do miocárdio é normal durante o período de cicatrização, embora exista insuficiência cardíaca
resolves10.1016/j.lfs.2010.10.008
Bone marrow derived cells decrease inflammation but not oxidative stress in an experimental model of acute myocardial infarction
resolves10.1177/030089168507100305
Comparison of Lipid Peroxidation and Myocardial Damage Induced by Adriamycin and 4′-Epiadriamycin in Mice
resolves10.1021/tx00026a012
Evaluation of the probe 2',7'-dichlorofluorescin as an indicator of reactive oxygen species formation and oxidative stress
resolves10.1016/S0076-6879(99)01068-X
[6] Measuring nitric oxide production in human clinical studies
resolves10.1038/227680a0
Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4
resolves10.1210/en.2007-0043
Thyroid Function Disturbance and Type 3 Iodothyronine Deiodinase Induction after Myocardial Infarction in Rats—A Time Course Study
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Triiodothyronine (T3) Effects on Cardiovascular System in Patients with Heart Failure
resolves10.1104/pp.106.077073
Reactive Species and Antioxidants. Redox Biology Is a Fundamental Theme of Aerobic Life
resolves10.1021/bi011459o
Glutathionylation of the p50 Subunit of NF-κB: a Mechanism for Redox-Induced Inhibition of DNA Binding
resolves10.1007/BF01906378
Left ventricular systolic wall stress as a primary determinant of myocardial oxygen consumption: Comparative studies in patients with normal left ventricular function, with pressure and volume overload and with coronary heart disease
resolves10.1016/j.mce.2010.12.018
Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system
resolves10.1161/01.CIR.81.3.772
Endothelium-dependent dilation of the coronary microvasculature is impaired in dilated cardiomyopathy.
resolves10.1097/01.fjc.0000140206.81804.33
Thyroid Hormone Analog, DITPA, Improves Endothelial Nitric Oxide and Beta-Adrenergic Mediated Vasorelaxation after Myocardial Infarction
resolves10.1152/ajpcell.1996.271.3.C869
Endothelin-1-evoked arachidonic acid release: a Ca(2+)-dependent pathway
resolves10.1093/cvr/cvn098
Transcriptional control of mitochondrial biogenesis: the central role of PGC-1
resolves10.1007/s11010-014-2292-z
Modulation of apoptosis by sulforaphane is associated with PGC-1α stimulation and decreased oxidative stress in cardiac myoblasts
resolves10.1089/ars.2009.2940
PGC-1α Regulates Expression of Myocardial Mitochondrial Antioxidants and Myocardial Oxidative Stress After Chronic Systolic Overload
resolves10.1152/ajpendo.00436.2004
Thyroid hormone regulates tubulin expression in mammalian liver. Effects of deleting thyroid hormone receptor-α or -β
The 2 references without a DOI — listed, not checked
no DOI — not checkedKlein D, Kern RM, Sokol RZ (1995) A method for quantification and correction of proteins after transfer to immobilization membranes. Biochem Mol Biol Int 36:59–66
no DOI — not checkedSamuel JL, Marotte F, Delcayre C, Rappaport L (1986) Microtubule reorganization is related to rate of heart myocyte hypertrophy in rat. Am J Physiol 251:1118–1125
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