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MEF2C Ablation in Endothelial Cells Reduces Retinal Vessel Loss and Suppresses Pathologic Retinal Neovascularization in Oxygen-Induced Retinopathy
52/52 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
5 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 52 checked references that resolve
resolves10.1007/s10633-009-9181-x
The effects of oxygen stresses on the development of features of severe retinopathy of prematurity: knowledge from the 50/10 OIR model
resolves10.1172/JCI42142
Retinopathy of prematurity: understanding ischemic retinal vasculopathies at an extreme of life
resolves10.1167/iovs.08-2584
Through The Eyes of a Child: Understanding Retinopathy through ROP The Friedenwald Lecture
resolves10.1007/s10633-009-9201-x
Understanding ischemic retinopathies: emerging concepts from oxygen-induced retinopathy
resolves10.1006/dbio.1999.9307
The Transcription Factor MEF2C-Null Mouse Exhibits Complex Vascular Malformations and Reduced Cardiac Expression of Angiopoietin 1 and VEGF
resolves10.1242/dev.125.22.4565
Requirement of the MADS-box transcription factor MEF2C for vascular development
resolves10.1126/science.276.5317.1404
Control of Mouse Cardiac Morphogenesis and Myogenesis by Transcription Factor MEF2C
resolves10.1006/dbio.1995.0002
Regulation of Muscle Differentiation by the MEF2 Family of MADS Box Transcription Factors
resolves10.1167/iovs.08-1760
Vascular Endothelial Growth Factor Induces MEF2C and MEF2-Dependent Activity in Endothelial Cells
resolves10.1002/gene.20152
Generation of conditionalMef2cloxP/loxP mice for temporal- and tissue-specific analyses
resolves10.1002/dvdy.20828
MEF2C is required for the normal allocation of cells between the ventricular and sinoatrial precursors of the primary heart field
resolves10.1038/nprot.2009.187
Quantification of oxygen-induced retinopathy in the mouse: a model of vessel loss, vessel regrowth and pathological angiogenesis
resolves10.1002/dvdy.1200
NG2 proteoglycan is expressed exclusively by mural cells during vascular morphogenesis
resolves10.1167/iovs.11-8002
Reduced Retinal Neovascularization, Vascular Permeability, and Apoptosis in Ischemic Retinopathy in the Absence of Prolyl Hydroxylase-1 Due to the Prevention of Hyperoxia-Induced Vascular Obliteration
resolves10.1091/mbc.e04-11-1029
Cathepsin B Regulates the Intrinsic Angiogenic Threshold of Endothelial Cells
resolves10.1128/MCB.01275-09
Phospholipase Cγ Activation Drives Increased Production of Autotaxin in Endothelial Cells and Lysophosphatidic Acid-Dependent Regression
resolves10.1016/S0002-9440(10)63741-8
Transcription Factor Ets-1 Mediates Ischemia- and Vascular Endothelial Growth Factor-Dependent Retinal Neovascularization
resolves10.1161/01.ATV.0000248731.55781.87
Tissue Factor Pathway Inhibitor-2 Is Upregulated by Vascular Endothelial Growth Factor and Suppresses Growth Factor-Induced Proliferation of Endothelial Cells
resolves10.1074/jbc.M510217200
Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice
resolves10.1073/pnas.0802876105
Transcription factor MEF2C influences neural stem/progenitor cell differentiation and maturation
<i>in vivo</i>
resolves10.1016/j.devcel.2007.03.007
The Transcription Factor MEF2C Is Required for Craniofacial Development
resolves10.1038/ni.1609
Transcription factor Mef2c is required for B cell proliferation and survival after antigen receptor stimulation
resolves10.1038/nm1591
Increased dietary intake of ω-3-polyunsaturated fatty acids reduces pathological retinal angiogenesis
resolves10.1182/blood-2010-10-311589
Ischemic neurons prevent vascular regeneration of neural tissue by secreting semaphorin 3A
resolves10.1007/s00417-009-1116-4
Kinetics of retinal vaso-obliteration and neovascularisation in the oxygen-induced retinopathy (OIR) mouse model
resolves10.1038/nm1095-1024
Vascular endothelial growth factor acts as a survival factor for newly formed retinal vessels and has implications for retinopathy of prematurity
resolves10.1167/iovs.10-6978
Altered Expression of the Transcription Factor Mef2c during Retinal Degeneration in<i>Rpe65</i><sup>–/–</sup>Mice
resolves10.1016/j.ajpath.2010.12.031
Novel Protective Properties of IGFBP-3 Result in Enhanced Pericyte Ensheathment, Reduced Microglial Activation, Increased Microglial Apoptosis, and Neuronal Protection after Ischemic Retinal Injury
resolves10.1161/CIRCULATIONAHA.107.698316
Heme Oxygenase-1 and Carbon Monoxide in Vascular Pathobiology
resolves10.1172/JCI4165
Oxidative stress causes enhanced endothelial cell injury in human heme oxygenase-1 deficiency
resolves10.1002/gene.1030
Characterization of a novel EGFP reporter mouse to monitor Cre recombination as demonstrated by a Tie2 Cre mouse line
resolves10.1161/CIRCRESAHA.109.199059
Insulin-Like Growth Factor Binding Protein-3 Mediates Vascular Repair by Enhancing Nitric Oxide Generation
resolves10.1073/pnas.0702031104
IGFBP3 suppresses retinopathy through suppression of oxygen-induced vessel loss and promotion of vascular regrowth
resolves10.1523/JNEUROSCI.3210-09.2010
Norrin Promotes Vascular Regrowth after Oxygen-Induced Retinal Vessel Loss and Suppresses Retinopathy in Mice
resolves10.1002/dvdy.10412
Thrombospondin‐1–deficient mice exhibit increased vascular density during retinal vascular development and are less sensitive to hyperoxia‐mediated vessel obliteration
resolves10.1016/S0002-9440(10)62284-5
Inhibition of Tumor Necrosis Factor-α Improves Physiological Angiogenesis and Reduces Pathological Neovascularization in Ischemic Retinopathy
resolves10.1167/iovs.07-1356
Activated NAD(P)H Oxidase from Supplemental Oxygen Induces Neovascularization Independent of VEGF in Retinopathy of Prematurity Model
resolves10.1016/S0002-9440(10)63001-5
Inhibition of NAD(P)H Oxidase Activity Blocks Vascular Endothelial Growth Factor Overexpression and Neovascularization during Ischemic Retinopathy
resolves10.1016/j.freeradbiomed.2004.09.008
Redox-dependent effects of nitric oxide on microvascular integrity in oxygen-induced retinopathy
resolves10.1038/nm1336
Trans-arachidonic acids generated during nitrative stress induce a thrombospondin-1–dependent microvascular degeneration
The 5 references without a DOI — listed, not checked
no DOI — not checkedOxygen-induced retinopathy in the mouse
no DOI — not checkedQuantitative assessment of the integrity of the blood-retinal barrier in mice
no DOI — not checkedErythropoietin deficiency decreases vascular stability in mice
no DOI — not checkedInhibition of NAD(P)H oxidase reduces apoptosis and avascular retina in an animal model of retinopathy of prematurity
no DOI — not checkedReduced severity of oxygen-induced retinopathy in eNOS-deficient mice
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