Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 69 checked references that resolve
resolves10.1038/35019075Glycosyltransferase activity of Fringe modulates Notch–Delta interactions
resolves10.1128/JB.181.18.5636-5643.1999Isolation, Characterization, and Localization of a Capsule-Associated Gene,
<i>CAP10</i>
, of
<i>Cryptococcus neoformans</i>
resolves10.1016/j.cub.2006.09.031Lethal Giant Discs, a Novel C2-Domain Protein, Restricts Notch Activation during Endocytosis
resolves10.1242/dev.124.10.1919Notch signalling regulates <i>veinlet</i> expression and establishes boundaries between veins and interveins in the <i>Drosophila</i> wing
resolves10.1038/19083A presenilin-1-dependent γ-secretase-like protease mediates release of Notch intracellular domain
resolves10.1016/j.cell.2005.08.017Asymmetric Rab11 Endosomes Regulate Delta Recycling and Specify Cell Fate in the Drosophila Nervous System
resolves10.1242/dev.01448<i>Drosophila</i>
Deltex mediates Suppressor of Hairless-independent and late-endosomal activation of Notch signaling
resolves10.1016/j.devcel.2006.09.019The Drosophila Notch Inhibitor and Tumor Suppressor Gene lethal (2) giant discs Encodes a Conserved Regulator of Endosomal Trafficking
resolves10.1016/j.devcel.2005.06.010Sec15, a Component of the Exocyst, Promotes Notch Signaling during the Asymmetric Division of Drosophila Sensory Organ Precursors
resolves10.1038/383707a0Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia
resolves10.1242/dev.01074Notch signaling: control of cell communication and cell fate
resolves10.1016/j.ydbio.2004.01.032A hidden program in Drosophila peripheral neurogenesis revealed: fundamental principles underlying sensory organ diversity
resolves10.1242/dev.01789The roles of receptor and ligand endocytosis in regulating Notch signaling
resolves10.1101/gad.9.21.2598The neurogenic suppressor of hairless DNA-binding protein mediates the transcriptional activation of the enhancer of split complex genes triggered by Notch signaling.
resolves10.1038/ng0797-243Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1
resolves10.1074/jbc.M511974200Two Distinct Pathways for O-Fucosylation of Epidermal Growth Factor-like or Thrombospondin Type 1 Repeats
resolves10.1128/MCB.01655-05Leukemia-Associated Mutations within the NOTCH1 Heterodimerization Domain Fall into at Least Two Distinct Mechanistic Classes
resolves10.1074/jbc.275.13.9604Mammalian Notch1 Is Modified with Two Unusual Forms ofO-Linked Glycosylation Found on Epidermal Growth Factor-like Modules
resolves10.1016/S0076-6879(06)17008-1Methods for Analysis of O‐Linked Modifications on Epidermal Growth Factor‐Like and Thrombospondin Type 1 Repeats
resolves10.1038/ng0797-235Mutations in the human Jagged1 gene are responsible for Alagille syndrome
resolves10.1126/science.1108995Chaperone Activity of Protein
<i>O</i>
-Fucosyltransferase 1 Promotes Notch Receptor Folding
resolves10.1016/S0092-8674(00)80335-9Kuzbanian Controls Proteolytic Processing of Notch and Mediates Lateral Inhibition during Drosophila and Vertebrate Neurogenesis
resolves10.1242/dev.127.7.1373Ligand endocytosis drives receptor dissociation and activation in the Notch pathway
resolves10.1016/S0955-0674(00)00219-2Intracellular signaling from the endoplasmic reticulum to the nucleus: the unfolded protein response in yeast and mammals
resolves10.1074/jbc.M509552200Lunatic Fringe, Manic Fringe, and Radical Fringe Recognize Similar Specificity Determinants in O-Fucosylated Epidermal Growth Factor-like Repeats
resolves10.1242/dev.00679<i>neurotic</i>
, a novel maternal neurogenic gene, encodes an
<i>O</i>
-fucosyltransferase that is essential for Notch-Delta interactions
resolves10.1242/dev.02811The<i>O</i>-fucosyltransferase O-fut1 is an extracellular component that is essential for the constitutive endocytic trafficking of Notch in<i>Drosophila</i>
resolves10.1007/s000180300019O-fucose modifications of epidermal growth factor-like repeats and thrombospondin type 1 repeats: unusual modifications in unusual places
resolves10.1093/glycob/cwf085O-Glycosylation of EGF repeats: identification and initial characterization of a UDP-glucose: protein O-glucosyltransferase
resolves10.1074/jbc.M212221200Fringe Modifies O-Fucose on Mouse Notch1 at Epidermal Growth Factor-like Repeats within the Ligand-binding Site and the Abruptex Region
resolves10.1073/pnas.0831126100Protein
<i>O</i>
-fucosyltransferase 1 is an essential component of Notch signaling pathways
resolves10.1038/19091Presenilin is required for activity and nuclear access of Notch in Drosophila
resolves10.1016/j.gene.2005.08.027Cloning, expression and characterization of a novel human CAP10-like gene hCLP46 from CD34+ stem/progenitor cells
resolves10.1101/gad.10.6.672shotgun encodes Drosophila E-cadherin and is preferentially required during cell rearrangement in the neurectoderm and other morphogenetically active epithelia.
resolves10.1074/jbc.M701065200O-Fucosylation of Thrombospondin Type 1 Repeats in ADAMTS-like-1/Punctin-1 Regulates Secretion
resolves10.1242/dev.01413<i>Drosophila</i>
Epsin mediates a select endocytic pathway that DSL ligands must enter to activate Notch
resolves10.1242/dev.01860Distinct roles for Mind bomb, Neuralized and Epsin in mediating DSL endocytosis and signaling in<i>Drosophila</i>
resolves10.1074/jbc.273.14.8112Purification and Characterization of a GDP-fucose:Polypeptide Fucosyltransferase from Chinese Hamster Ovary Cells
resolves10.1038/19096Neurogenic phenotypes and altered Notch processing in Drosophila Presenilin mutants
resolves10.1073/pnas.1832753100Mapping
<i>Drosophila</i>
mutations with molecularly defined
<i>P</i>
element insertions
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