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The protective effect and potential mechanism of NRXN1 on learning and memory in ADHD rat models

https://doi.org/10.1016/j.expneurol.2021.113806
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1 of 44 checkable references need attention · checked 2026-07-23

At the dated check, the references listed below either did not resolve in Crossref or DataCite, or carried a retraction notice. Each one is shown with the registry record that put it there.

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References needing attention

marked retracted — notice via Crossref, record curated by Retraction Watch10.1016/j.neuron.2017.04.011
RETRACTED: Conditional Deletion of All Neurexins Defines Diversity of Essential Synaptic Organizer Functions for Neurexins
The 43 checked references that resolve
resolves10.1016/j.ejmg.2018.07.015
NRXN1 deletion syndrome; phenotypic and penetrance data from 34 families
resolves10.1177/0022219412437040
Learning and Memory Impairments in Children and Adolescents With Attention-Deficit/Hyperactivity Disorder
resolves10.1016/j.cell.2015.06.056
β-Neurexins Control Neural Circuits by Regulating Synaptic Endocannabinoid Signaling
resolves10.1523/JNEUROSCI.0599-17.2018
Rescue of Learning and Memory Deficits in the Human Nonsyndromic Intellectual Disability Cereblon Knock-Out Mouse Model by Targeting the AMP-Activated Protein Kinase–mTORC1 Translational Pathway
resolves10.1016/j.biopsych.2006.04.022
Methylphenidate Preferentially Increases Catecholamine Neurotransmission within the Prefrontal Cortex at Low Doses that Enhance Cognitive Function
resolves10.1093/brain/awx272
Epigenetic editing of the Dlg4/PSD95 gene improves cognition in aged and Alzheimer’s disease mice
resolves10.1016/j.nbd.2012.03.031
Mutations affecting synaptic levels of neurexin-1β in autism and mental retardation
resolves10.1083/jcb.96.5.1337
Synapsin I (protein I), a nerve terminal-specific phosphoprotein. I. Its general distribution in synapses of the central and peripheral nervous system demonstrated by immunofluorescence in frozen and plastic sections.
resolves10.1016/j.neures.2016.09.005
Functions of synapse adhesion molecules neurexin/neuroligins and neurodevelopmental disorders
resolves10.1016/j.neuron.2019.03.032
Alternative Splicing of Presynaptic Neurexins Differentially Controls Postsynaptic NMDA and AMPA Receptor Responses
resolves10.1074/jbc.270.41.23903
High Affinity Binding of α-Latrotoxin to Recombinant Neurexin Iα
resolves10.1037/bne0000024
Phenotypic characterization of nonsocial behavioral impairment in neurexin 1α knockout rats.
resolves10.1007/s00439-011-0975-z
Truncating mutations in NRXN2 and NRXN1 in autism spectrum disorders and schizophrenia
resolves10.1101/lm.028449.112
The roles of protein kinases in learning and memory
resolves10.1371/journal.pone.0067114
Altered Social Behaviours in Neurexin 1α Knockout Mice Resemble Core Symptoms in Neurodevelopmental Disorders
resolves10.1002/ajmg.a.34255
Compound heterozygous deletion of<i>NRXN1</i>causing severe developmental delay with early onset epilepsy in two sisters
resolves10.7150/ijms.4892
Increased Locomotor Activity and Non-Selective Attention and Impaired Learning Ability in SD Rats after Lentiviral Vector-Mediated RNA Interference of Homer 1a in the Brain
resolves10.1126/science.277.5331.1511
Binding of Neuroligins to PSD-95
resolves10.1038/ng1136
Mutations of the X-linked genes encoding neuroligins NLGN3 and NLGN4 are associated with autism
resolves10.1016/j.conb.2017.12.003
NLGN1 and NLGN2 in the prefrontal cortex: their role in memory consolidation and strengthening
resolves10.1016/j.yexcr.2019.06.014
Single cell analysis of autism patient with bi-allelic NRXN1-alpha deletion reveals skewed fate choice in neural progenitors and impaired neuronal functionality
resolves10.1101/lm.043133.116
Muscarinic acetylcholine receptors act in synergy to facilitate learning and memory
resolves10.1523/JNEUROSCI.3560-05.2005
N-Terminal Insertion and C-Terminal Ankyrin-Like Repeats of α-Latrotoxin Are Critical for Ca<sup>2</sup>+-Dependent Exocytosis
resolves10.1038/s41598-019-42635-7
A rare autism-associated MINT2/APBA2 mutation disrupts neurexin trafficking and synaptic function
resolves10.1016/j.jpsychires.2011.10.015
Mutation analysis of the NRXN1 gene in a Chinese autism cohort
resolves10.1101/lm.045369.117
α2* Nicotinic acetylcholine receptors influence hippocampus-dependent learning and memory in adolescent mice
resolves10.7554/eLife.46773
Neurexophilin4 is a selectively expressed α-neurexin ligand that modulates specific cerebellar synapses and motor functions
resolves10.1515/bjmg-2016-0031
Mutation analysis of the <i>NRXN1</i> gene in autism spectrum disorders
resolves10.1038/srep16410
Mice lacking the PSD-95–interacting E3 ligase, Dorfin/Rnf19a, display reduced adult neurogenesis, enhanced long-term potentiation and impaired contextual fear conditioning
resolves10.1016/j.chc.2014.05.014
Behavior Management for School-Aged Children with ADHD
resolves10.3389/fnsyn.2020.00008
Phosphorylation-Dependent Regulation of Ca2+-Permeable AMPA Receptors During Hippocampal Synaptic Plasticity
resolves10.1186/gb-2013-14-9-213
Neurexins
resolves10.1074/jbc.M114.595413
Dystroglycan Binding to α-Neurexin Competes with Neurexophilin-1 and Neuroligin in the Brain
resolves10.1006/geno.2002.6734
Analysis of the Human Neurexin Genes: Alternative Splicing and the Generation of Protein Diversity
resolves10.1016/j.conb.2019.03.007
Control of neural development and function by glial neuroligins
resolves10.1523/JNEUROSCI.1357-17.2017
Proteolytic Processing of Neurexins by Presenilins Sustains Synaptic Vesicle Release
resolves10.1083/jcb.200105003
A stoichiometric complex of neurexins and dystroglycan in brain
resolves10.1017/S0033291715002020
Disruptions of working memory and inhibition mediate the association between exposure to institutionalization and symptoms of attention deficit hyperactivity disorder
resolves10.2174/1570159X14666160414111821
Mitochondria and Synaptic Plasticity in the Mature and Aging Nervous System
resolves10.1007/978-3-540-74805-2_7
α-Latrotoxin and Its Receptors
resolves10.1002/mgg3.105
A common cognitive, psychiatric, and dysmorphic phenotype in carriers of <scp><i>NRXN1</i></scp> deletion
resolves10.1016/j.brainresbull.2020.03.015
Distinct long non-coding RNA and mRNA expression profiles in the hippocampus of an attention deficit hyperactivity disorder model in spontaneously hypertensive rats and control wistar Kyoto rats
The 4 references without a DOI — listed, not checked
no DOI — not checkedRole of NRXN-NLGN-SHANK pathway gene variations in the pathogenesis of autism spectrum disorders
no DOI — not checkedThe rat cortex in stereotaxic coordinates
no DOI — not checkedNeurexin-1alpha regulates neurite growth of rat hippocampal neurons
no DOI — not checkedInteractions between the hippocampus, prefrontal cortex, and amygdala support complex learning and memory
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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