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Sirtuin 5 as a novel target to blunt blood–brain barrier damage induced by cerebral ischemia/reperfusion injury

https://doi.org/10.1016/j.ijcard.2017.12.060
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Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

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The 37 checked references that resolve
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Biochemical Markers for Blood-Brain Barrier Dysfunction in Acute Ischemic Stroke Correlate with Evolution and Outcome
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Matrix Metalloproteinase-2-Mediated Occludin Degradation and Caveolin-1-Mediated Claudin-5 Redistribution Contribute to Blood–Brain Barrier Damage in Early Ischemic Stroke Stage
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Normobaric hyperoxia attenuates early blood–brain barrier disruption by inhibiting MMP‐9‐mediated occludin degradation in focal cerebral ischemia
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Matrix Metalloproteinase-Mediated Disruption of Tight Junction Proteins in Cerebral Vessels is Reversed by Synthetic Matrix Metalloproteinase Inhibitor in Focal Ischemia in Rat
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Gene Silencing of the Mitochondrial Adaptor p66 <sup>Shc</sup> Suppresses Vascular Hyperglycemic Memory in Diabetes
resolves10.1093/eurheartj/ehv140
Post-ischaemic silencing of p66<sup>Shc</sup>reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke
resolves10.1038/ncomms1852
Targeted suppression of claudin-5 decreases cerebral oedema and improves cognitive outcome following traumatic brain injury
resolves10.1038/ncomms10523
Rapid endothelial cytoskeletal reorganization enables early blood–brain barrier disruption and long-term ischaemic reperfusion brain injury
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resolves10.1038/nrd4685
Clinical experiences with systemically administered siRNA-based therapeutics in cancer
resolves10.1038/nature14580
Genetic compensation induced by deleterious mutations but not gene knockdowns
resolves10.1073/pnas.1621174114
Endothelium-targeted overexpression of heat shock protein 27 ameliorates blood–brain barrier disruption after ischemic brain injury
resolves10.1136/neurintsurg-2013-010780
Admission neutrophil–lymphocyte ratio predicts 90 day outcome after endovascular stroke therapy
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Early Neutrophilia Is Associated With Volume of Ischemic Tissue in Acute Stroke
resolves10.1007/s12640-017-9707-z
Neutrophil-To-Lymphocyte Ratio Predicts 3-Month Outcome of Acute Ischemic Stroke
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Formyl-Peptide Receptor 2/3/Lipoxin A <sub>4</sub> Receptor Regulates Neutrophil-Platelet Aggregation and Attenuates Cerebral Inflammation
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The glucagon-like peptide-1 receptor agonist exendin-4 ameliorates warfarin-associated hemorrhagic transformation after cerebral ischemia
resolves10.1007/s12035-015-9583-6
bFGF Protects Against Blood-Brain Barrier Damage Through Junction Protein Regulation via PI3K-Akt-Rac1 Pathway Following Traumatic Brain Injury
resolves10.1016/j.brainresbull.2016.06.002
Ghrelin attenuates brain injury in septic mice via PI3K/Akt signaling activation
resolves10.1002/jcb.24118
The ubiquitin–proteasome pathway regulates claudin 5 degradation
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The Tight Junction-specific Protein Occludin Is a Functional Target of the E3 Ubiquitin-protein Ligase Itch
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Biphasic Modulation of Paracellular Claudin-5 Expression in Mouse Brain Endothelial Cells Is Mediated through the Phosphoinositide-3-Kinase/AKT Pathway
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Cerebral microvascular changes in permeability and tight junctions induced by hypoxia-reoxygenation
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Hypoxia Selectively Disrupts Brain Microvascular Endothelial Tight Junction Complexes Through a Hypoxia‐Inducible Factor‐1 (HIF‐1) Dependent Mechanism
The 2 references without a DOI — listed, not checked
no DOI — not checkedHeart disease and stroke Statistics-2016 update: a report from the American Heart Association
no DOI — not checkedet al
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