Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 50 checked references that resolve
resolves10.1038/nrm2240Hydrogen peroxide: a metabolic by-product or a common mediator of ageing signals?
resolves10.1099/mic.0.050823-0Superoxide dismutase C is required for intracellular survival and virulence of Burkholderia pseudomallei
resolves10.1038/nri3423Beyond oxidative stress: an immunologist's guide to reactive oxygen species
resolves10.1038/cdd.2014.16Oncogene-induced reactive oxygen species fuel hyperproliferation and DNA damage response activation
resolves10.1021/cr300163eCysteine-Mediated Redox Signaling: Chemistry, Biology, and Tools for Discovery
resolves10.1038/nature02075ATP-dependent reduction of cysteine–sulphinic acid by S. cerevisiae sulphiredoxin
resolves10.7554/eLife.41044Myocardial NADPH oxidase-4 regulates the physiological response to acute exercise
resolves10.1038/nrm3801Cellular mechanisms and physiological consequences of redox-dependent signalling
resolves10.1038/nrc3339Aiding and abetting roles of NOX oxidases in cellular transformation
resolves10.1038/ncomms5425Src-dependent impairment of autophagy by oxidative stress in a mouse model of Duchenne muscular dystrophy
resolves10.1002/pro.5560021013A structural model for the nucleotide binding domains of the flavocytochrome <i>b</i><sub>–245</sub> β‐chain
resolves10.1074/jbc.270.14.7853The Mechanism of Electron Donation to Molecular Oxygen by Phagocytic Cytochrome b558
resolves10.1111/febs.14190High‐resolution studies of hydride transfer in the ferredoxin:<scp>NADP</scp><sup>+</sup> reductase superfamily
resolves10.1111/febs.14757Tripping the light fantastic in membrane redox biology: linking dynamic structures to function in ER electron transfer chains
resolves10.1074/jbc.M109.058909Structure of the N-terminal Regulatory Domain of a Plant NADPH Oxidase and Its Functional Implications
resolves10.1042/BJ20070255Structures and metal-ion-binding properties of the Ca2+-binding helix–loop–helix EF-hand motifs
resolves10.1038/416291aKilling activity of neutrophils is mediated through activation of proteases by K+ flux
resolves10.1182/blood-2010-06-290171Myeloperoxidase is required for neutrophil extracellular trap formation: implications for innate immunity
resolves10.1038/ni.1992Germline CYBB mutations that selectively affect macrophages in kindreds with X-linked predisposition to tuberculous mycobacterial disease
resolves10.1089/ars.2010.3611Redox Regulation of Protein Tyrosine Phosphatases: Structural and Chemical Aspects
resolves10.1126/scisignal.2000448The Plant NADPH Oxidase RBOHD Mediates Rapid Systemic Signaling in Response to Diverse Stimuli
resolves10.1038/srep02084Sustained production of ROS triggers compensatory proliferation and is required for regeneration to proceed
resolves10.1038/ncb2659Amputation-induced reactive oxygen species are required for successful Xenopus tadpole tail regeneration
resolves10.1371/journal.pone.0054391ATP-Mediated Transactivation of the Epidermal Growth Factor Receptor in Airway Epithelial Cells Involves DUOX1-Dependent Oxidation of Src and ADAM17
resolves10.1038/nature10632Lyn is a redox sensor that mediates leukocyte wound attraction in vivo
resolves10.1038/s41467-018-07505-2Tumour-elicited neutrophils engage mitochondrial metabolism to circumvent nutrient limitations and maintain immune suppression
resolves10.1210/me.2010-0340ROS Signaling by NOX4 Drives Fibroblast-to-Myofibroblast Differentiation in the Diseased Prostatic Stroma
The 4 references without a DOI — listed, not checked
no DOI — not checkedHMDB 3.0—The Human Metabolome Database in 2013
no DOI — not checkedEvolution of the ferric reductase domain (FRD) superfamily: modularity, functional diversification, and signature motifs
no DOI — not checkedMolecular repair, wound healing, and carcinogenesis tumor production a possible overhealing?
no DOI — not checkedNADPH oxidase mediates microtubule alterations and diaphragm dysfunction in dystrophic mice
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