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Angiotensin II facilitates neointimal formation by increasing vascular smooth muscle cell migration: Involvement of APE/Ref-1-mediated overexpression of sphingosine-1-phosphate receptor 1

https://doi.org/10.1016/j.taap.2018.03.032
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38/38 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

3 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 38 checked references that resolve
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Angiotensin II revisited: new roles in inflammation, immunology and aging
resolves10.1089/ars.2008.2198
Transcriptional Regulatory Functions of Mammalian AP-Endonuclease (APE1/Ref-1), an Essential Multifunctional Protein
resolves10.1016/j.semcdb.2012.05.003
Redox balance dynamically regulates vascular growth and remodeling
resolves10.1016/j.jnutbio.2009.03.003
l-Carnitine attenuates angiotensin II-induced proliferation of cardiac fibroblasts: role of NADPH oxidase inhibition and decreased sphingosine-1-phosphate generation
resolves10.1016/j.immuni.2012.10.015
Angiotensin II Drives the Production of Tumor-Promoting Macrophages
resolves10.1016/j.drudis.2010.11.016
Angiotensin II receptors and drug discovery in cardiovascular disease
resolves10.1016/j.abb.2010.04.003
Angiotensin II-dependent growth of vascular smooth muscle cells requires transactivation of the epidermal growth factor receptor via a cytosolic phospholipase A2-mediated release of arachidonic acid
resolves10.1016/j.jvs.2007.05.055
Regulation of arterial lesions in mice depends on differential smooth muscle cell migration: A role for sphingosine-1-phosphate receptors
resolves10.1161/01.ATV.16.7.857
Angiotensin II-Induced Progression of Neointimal Thickening in the Balloon-Injured Rat Carotid Artery Is AT<sub>1</sub>Receptor Mediated
resolves10.1016/j.ajpath.2012.04.015
Fenofibrate Increases High-Density Lipoprotein and Sphingosine 1 Phosphate Concentrations Limiting Abdominal Aortic Aneurysm Progression in a Mouse Model
resolves10.1161/CIRCRESAHA.108.178699
Gene Transfer of Redox Factor-1 Inhibits Neointimal Formation
resolves10.1016/j.bbagen.2014.11.017
DJ-1 regulates the expression of renal (pro)renin receptor via reactive oxygen species-mediated epigenetic modification
resolves10.1016/j.atherosclerosis.2015.03.038
Carvacrol inhibits atherosclerotic neointima formation by downregulating reactive oxygen species production in vascular smooth muscle cells
resolves10.1007/s11010-011-1154-1
Regulation of metabolism and transport of sphingosine-1-phosphate in mammalian cells
resolves10.1016/j.tcb.2011.09.003
Sphingosine-1-phosphate signaling and its role in disease
resolves10.1152/ajpcell.00287.2006
Angiotensin II cell signaling: physiological and pathological effects in the cardiovascular system
resolves10.1093/cvr/cvw256
Sphingosine-1-phosphate signalling—a key player in the pathogenesis of Angiotensin II-induced hypertension
resolves10.1242/dev.094805
Sphingosine 1-phosphate signalling
resolves10.1007/s11906-014-0431-2
Angiotensin II and Vascular Injury
resolves10.1161/01.ATV.0000237569.95046.b9
Sphingosine Kinase–Dependent Activation of Endothelial Nitric Oxide Synthase by Angiotensin II
resolves10.1007/s12272-001-2109-y
APE1/Ref-1 promotes the effect of Angiotensin II on Ca2+-activated K+ channel in human endothelial cells via suppression of NADPH Oxidase
resolves10.1093/eurheartj/ehn603
Epigenetic histone acetylation modifiers in vascular remodelling: new targets for therapy in cardiovascular disease
resolves10.1146/annurev.biochem.78.072407.103733
Sphingosine 1-Phosphate Receptor Signaling
resolves10.1089/ars.2009.2852
Redox Control of Vascular Smooth Muscle Migration
resolves10.2152/jmi.57.12
Crucial role of renin-angiotensin system in the pathogenesis of atherosclerosis
resolves10.1186/1868-7083-4-7
On the epigenetics of vascular regulation and disease
resolves10.1111/j.1476-5381.2011.01260.x
Pharmacological relevance and potential of sphingosine 1‐phosphate in the vascular system
resolves10.1097/HJH.0b013e3283525124
Regulation of mouse-renin gene by apurinic/apyrimidinic-endonuclease 1 (APE1/Ref-1) via recruitment of histone deacetylase 1 corepressor complex
resolves10.1093/cvr/cvt268
Acetylation of cyclophilin A is required for its secretion and vascular cell activation
resolves10.1016/B978-0-12-405881-1.00002-1
Reactive Oxygen Species in the Activation of MAP Kinases
resolves10.1111/j.1582-4934.2010.01136.x
Vascular biology: the role of sphingosine 1‐phosphate in both the resting state and inflammation
resolves10.1038/emm.2014.42
APE1/Ref-1 as an emerging therapeutic target for various human diseases: phytochemical modulation of its functions
resolves10.1016/j.bbalip.2013.09.009
Sphingosine 1-phosphate (S1P) induces COX-2 expression and PGE2 formation via S1P receptor 2 in renal mesangial cells
resolves10.1016/j.atherosclerosis.2012.08.018
Angiotensin converting enzyme 2 and atherosclerosis
resolves10.1093/toxsci/kfr347
Antifungal Miconazole Induces Cardiotoxicity Via Inhibition of APE/Ref-1-Related Pathway in Rat Neonatal Cardiomyocytes
resolves10.1093/cvr/cvt274
DJ-1/park7 modulates vasorelaxation and blood pressure via epigenetic modification of endothelial nitric oxide synthase
resolves10.2174/157016111201140327155551
Epigenetics in Vascular Disease – Therapeutic Potential of New Agents
resolves10.1093/cvr/cvr003
Role of histone deacetylases in vascular cell homeostasis and arteriosclerosis
The 3 references without a DOI — listed, not checked
no DOI — not checkedACE2 deficiency enhances angiotensin II-mediated aortic profilin-1 expression, inflammation and peroxynitrite production
no DOI — not checkedSphingosine signaling and atherogenesis
no DOI — not checkedAngiotensin-(1-7) abrogates angiotensin II-induced proliferation, migration and inflammation in VSMCs through inactivation of ROS-mediated PI3K/Akt and MAPK/ERK signaling pathways
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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