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Cardiac and adipose tissue abnormalities but not diabetes in mice deficient in GLUT4

https://doi.org/10.1038/377151a0
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18/18 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

5 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 18 checked references that resolve
resolves10.1007/978-1-4615-2910-1_10
Insulin Resistance and the Pathogenesis of Non-Insulin Dependent Diabetes Mellitus: Cellular and Molecular Mechanisms
resolves10.2337/diacare.13.3.209
Molecular Physiology of Glucose Transporters
resolves10.1016/S0021-9258(19)38295-X
Human facilitative glucose transporters. Isolation, functional characterization, and gene localization of cDNAs encoding an isoform (GLUT5) expressed in small intestine, kidney, muscle, and adipose tissue and an unusual glucose transporter pseudogene-like sequence (GLUT6).
resolves10.1172/JCI115724
Facilitative glucose transporters: regulatory mechanisms and dysregulation in diabetes.
resolves10.1172/JCI113485
Role of glucose transporters in the cellular insulin resistance of type II non-insulin-dependent diabetes mellitus.
resolves10.1073/pnas.86.8.2535
A glucose transport protein expressed predominately in insulin-responsive tissues.
resolves10.1042/bj2950287
Glucose transporters and <i>in vivo</i> glucose uptake in skeletal and cardiac muscle: fasting, insulin stimulation and immunoisolation studies of GLUT1 and GLUT4
resolves10.1083/jcb.126.5.1123
Insulin resistance, diabetes, and the insulin-regulated trafficking of GLUT-4.
resolves10.1073/pnas.91.2.728
Mice lacking N-acetylglucosaminyltransferase I activity die at mid-gestation, revealing an essential role for complex or hybrid N-linked carbohydrates.
resolves10.2337/diab.41.2.187
Glucose Transporter Levels in Tissues of Spontaneously Diabetic Zucker <i>fa</i>/<i>fa</i> Rat (ZDF/drt) and Viable Yellow Mouse (<i>Avy</i>/<i>a</i>)
resolves10.2337/diab.39.6.712
Differential Regulation of Two Glucose Transporters in Rat Liver by Fasting and Refeeding and by Diabetes and Insulin Treatment
resolves10.1016/S0021-9258(18)41516-5
Adipose cell hyperplasia and enhanced glucose disposal in transgenic mice overexpressing GLUT4 selectively in adipose tissue.
resolves10.1126/science.1439783
What if Minkowski Had Been Ageusic? An Alternative Angle on Diabetes
resolves10.2337/diacare.14.3.173
Insulin Resistance: A Multifaceted Syndrome Responsible for NIDDM, Obesity, Hypertension, Dyslipidemia, and Atherosclerotic Cardiovascular Disease
resolves10.2337/diacare.14.6.470
Hyperinsulinemia or Increased Sympathetic Drive as Links for Obesity and Hypertension
resolves10.1242/jcs.81.1.143
Localization of low abundance dna sequences in tissue sections by <i>in situ</i> hybridization
resolves10.1172/JCI115254
Decreased in vivo glucose uptake but normal expression of GLUT1 and GLUT4 in skeletal muscle of diabetic rats.
resolves10.1038/372186a0
Alternative pathway of insulin signalling in mice with targeted disruption of the IRS-1 gene
The 5 references without a DOI — listed, not checked
no DOI — not checkedLitwan, S. E. et al. Am. J. Physiol. 258, H551–H556 (1990).
no DOI — not checkedBressler, R. & Goldman, S. Cardioscience 4, 133–142 (1993).
no DOI — not checkedRobertson, E. J. Teratocarcinomas and Embryonic Stem Cells: A Practical Approach (IRL, Cambridge, 1987).
no DOI — not checkedSambrook, J., Fritsch, E. F. & Maniatis, T. Molecular Cloning: A Laboratory Manual 2nd edn (Cold Spring Harbor Lab. Press, NY, 1989).
no DOI — not checkedGarvey, W. T., Hardin, D., Juhaszova, M. & Dominguez, J. H. Am. J. Physiol. 264, H837–H844 (1993).
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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