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Cerebral Hypoperfusion: A New Pathophysiologic Concept in Multiple Sclerosis?
70/70 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
9 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 70 checked references that resolve
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Relapsing and remitting multiple sclerosis: Pathology of the newly forming lesion
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Monoclonal antibody treatment exposes three mechanisms underlying the clinical course of multiple sclerosis
resolves10.1093/brain/awl370
Autologous haematopoietic stem cell transplantation fails to stop demyelination and neurodegeneration in multiple sclerosis
resolves10.1080/01616412.1983.11739631
Cerebral Blood Flow and Red Cell Delivery in Normal Subjects and in Multiple Sclerosis
resolves10.1136/jnnp.47.11.1182
Studies on regional cerebral oxygen utilisation and cognitive function in multiple sclerosis.
resolves10.1159/000116926
Regional Cerebral Blood Flow in Multiple Sclerosis Measured by Single Photon Emission Tomography with Technetium-99m Hexamethyl-propyleneamine Oxime
resolves10.1007/BF03164835
Clinical significance of reduced cerebral metabolism in multiple sclerosis: A combined PET and MRI study
resolves10.1148/radiol.2313030996
Microvascular Abnormality in Relapsing-Remitting Multiple Sclerosis: Perfusion MR Imaging Findings in Normal-appearing White Matter
resolves10.1016/j.neuroimage.2006.08.008
Pattern of hemodynamic impairment in multiple sclerosis: Dynamic susceptibility contrast perfusion MR imaging at 3.0 T
resolves10.1016/j.jns.2008.12.036
White matter hemodynamic abnormalities precede sub-cortical gray matter changes in multiple sclerosis
resolves10.1002/mrm.24194
White matter and deep gray matter hemodynamic changes in multiple sclerosis patients with clinically isolated syndrome
resolves10.1002/(SICI)1522-2586(200005)11:5<495::AID-JMRI5>3.0.CO;2-S
Dynamic susceptibility contrast MR imaging of plaque development in multiple sclerosis: Application of an extended blood-brain barrier leakage correction
resolves10.1093/brain/awh007
Changes in cerebral perfusion precede plaque formation in multiple sclerosis: a longitudinal perfusion MRI study
resolves10.1136/jnnp.2008.157164
Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis
resolves10.1212/WNL.0b013e318212a901
Prevalence, sensitivity, and specificity of chronic cerebrospinal venous insufficiency in MS
resolves10.1002/jmri.21758
Diminished visibility of cerebral venous vasculature in multiple sclerosis by susceptibility‐weighted imaging at 3.0 Tesla
resolves10.1136/jnnp.2010.223479
No association of abnormal cranial venous drainage with multiple sclerosis: a magnetic resonance venography and flow-quantification study
resolves10.1038/jcbfm.2013.95
Concomitant Analysis of Arterial, Venous, and CSF Flows using Phase-Contrast MRI: A Quantitative Comparison Between MS Patients and Healthy Controls
resolves10.1136/jnnp-2013-305612
Reduced grey matter perfusion without volume loss in early relapsing-remitting multiple sclerosis
resolves10.1016/j.pneurobio.2006.12.003
N-Acetylaspartate in the CNS: From neurodiagnostics to neurobiology
resolves10.1038/jcbfm.2011.193
White-Matter Astrocytes, Axonal Energy Metabolism, and Axonal Degeneration in Multiple Sclerosis
resolves10.1371/journal.pone.0011625
Reduced NAA-Levels in the NAWM of Patients with MS Is a Feature of Progression. A Study with Quantitative Magnetic Resonance Spectroscopy at 3 Tesla
resolves10.1177/1352458513477228
Cerebral white matter blood flow and energy metabolism in multiple sclerosis
resolves10.1007/s00415-009-5047-0
Intracortical excitability in patients with relapsing–remitting and secondary progressive multiple sclerosis
resolves10.1002/ana.20736
Mitochondrial dysfunction as a cause of axonal degeneration in multiple sclerosis patients
resolves10.1038/jcbfm.2008.72
Hypoperfusion of the Cerebral White Matter in Multiple Sclerosis: Possible Mechanisms and Pathophysiological Significance
resolves10.1097/00041327-200103000-00011
Increased Endothelin-1 Plasma Levels in Patients With Multiple Sclerosis
resolves10.1159/000069085
Extraocular Blood Flow and Endothelin-1 Plasma Levels in Patients with Multiple Sclerosis
resolves10.1073/pnas.1222560110
Cerebral hypoperfusion in multiple sclerosis is reversible and mediated by endothelin-1
resolves10.1016/S1734-1140(10)70240-1
Role of endothelin-1 receptor blockers on hemodynamic parameters and oxidative stress
resolves10.1016/S1474-4422(09)70043-2
Virtual hypoxia and chronic necrosis of demyelinated axons in multiple sclerosis
resolves10.1155/S1110724303305029
Hypoperfusion, Mitochondria Failure, Oxidative Stress, and Alzheimer Disease
resolves10.1016/S0006-8993(01)03223-1
Axonal damage and demyelination in the white matter after chronic cerebral hypoperfusion in the rat
resolves10.1111/j.1750-3639.2003.tb00485.x
Molecular Changes in Normal Appearing White Matter in Multiple Sclerosis are Characteristic of Neuroprotective Mechanisms Against Hypoxic Insult
resolves10.1161/01.STR.0000221308.94473.14
White Matter Lesions in an Unselected Cohort of the Elderly
resolves10.1136/jnnp.69.1.48
Reduced cerebral blood flow in white matter in ischaemic leukoaraiosis demonstrated using quantitative exogenous contrast based perfusion MRI
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Oligodendrocyte injury in multiple sclerosis: a role for p53
resolves10.1002/1531-8249(200006)47:6<707::AID-ANA3>3.0.CO;2-Q
Heterogeneity of multiple sclerosis lesions: Implications for the pathogenesis of demyelination
resolves10.1093/jnen/62.1.25
Preferential Loss of Myelin-Associated Glycoprotein Reflects Hypoxia-Like White Matter Damage in Stroke and Inflammatory Brain Diseases
resolves10.1111/j.1552-6569.2011.00585.x
The Relationship between Normal Cerebral Perfusion Patterns and White Matter Lesion Distribution in 1,249 Patients with Multiple Sclerosis
resolves10.1016/j.mri.2013.03.016
Abnormalities of cerebral blood flow in multiple sclerosis: A pseudocontinuous arterial spin labeling MRI study
resolves10.1177/1352458513495936
Hypoperfusion and T1-hypointense lesions in white matter in multiple sclerosis
resolves10.1038/sj.jcbfm.9600504
Perfusion Magnetic Resonance Imaging Correlates of Neuropsychological Impairment in Multiple Sclerosis
resolves10.3174/ajnr.A3148
Robust Perfusion Deficits in Cognitively Impaired Patients with Secondary-Progressive Multiple Sclerosis
resolves10.3174/ajnr.A3060
Decreased Frontal Lobe Gray Matter Perfusion in Cognitively Impaired Patients with Secondary-Progressive Multiple Sclerosis Detected by the Bookend Technique
resolves10.1016/j.jns.2005.06.018
Subcortical vascular cognitive impairment: Similarities and differences with multiple sclerosis
resolves10.1016/S0031-6997(25)06809-7
Endothelins: molecular biology, biochemistry, pharmacology, physiology, and pathophysiology.
resolves10.1042/CS20060043
(Peri)vascular production and action of pro-inflammatory cytokines in brain pathology
resolves10.1006/pupt.1998.0145
Adenosine-Induced Inhibition of Basal Endothelin-1 Production from Guinea-pig Tracheal Epithelial Cells: a Mechanism Involving the Release of cAMP
The 9 references without a DOI — listed, not checked
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no DOI — not checkedbibr31-jcbfm.2015.131
no DOI — not checkedbibr43-jcbfm.2015.131
no DOI — not checkedbibr62-jcbfm.2015.131
no DOI — not checkedbibr63-jcbfm.2015.131
no DOI — not checkedbibr70-jcbfm.2015.131
no DOI — not checkedbibr72-jcbfm.2015.131
no DOI — not checkedbibr74-jcbfm.2015.131
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