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Mechanism of extrasynaptic dopamine signaling in Caenorhabditis elegans
48/48 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 48 checked references that resolve
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Coupling of Human D-1 Dopamine Receptors to Different Guanine Nucleotide Binding Proteins. EVIDENCE THAT D-1 DOPAMINE RECEPTORS CAN COUPLE TO BOTH Gs AND Go
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The D2 dopamine receptor isoforms signal through distinct Gi alpha proteins to inhibit adenylyl cyclase. A study with site-directed mutant Gi alpha proteins.
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C. elegans Locomotory Rate Is Modulated by the Environment through a Dopaminergic Pathway and by Experience through a Serotonergic Pathway
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Cloning and characterization of a <i>Caenorhabditis elegans</i> D2‐like dopamine receptor
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Patterning of dopaminergic neurotransmitter identity among <i>Caenorhabditis elegans</i> ray sensory neurons by a TGFβfamily signaling pathway and a <i>Hox</i> gene
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Dopamine modulates the plasticity of mechanosensory responses in Caenorhabditis elegans
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Levamisole-resitant mutants of the nematode Caenorhabditis elegans appear to lack pharmacological acetylcholine receptors
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Neuronal Control of Locomotion in C. elegans Is Modified by a Dominant Mutation in the GLR-1 Ionotropic Glutamate Receptor
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Neurotoxin-induced degeneration of dopamine neurons in
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LIM homeobox gene-dependent expression of biogenic amine receptors in restricted regions of the C. elegans nervous system
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The structure of the nervous system of the nematode
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The <i>C. elegans</i> NeuroD homolog <i>cnd-1</i> functions in multiple aspects of motor neuron fate specification
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Coordinated Transcriptional Regulation of the<i>unc-25</i>Glutamic Acid Decarboxylase and the<i>unc-47</i>GABA Vesicular Transporter by the<i>Caenorhabditis elegans</i>UNC-30 Homeodomain Protein
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Homologous and unique G protein alpha subunits in the nematode Caenorhabditis elegans.
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Participation of the Protein G
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Antagonism between Goalpha and Gqalpha in Caenorhabditis elegans: the RGS protein EAT-16 is necessary for Goalpha signaling and regulates Gqalpha activity
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Two RGS proteins that inhibit Gαo and Gαq signaling in C. elegans neurons require a Gβ5-like subunit for function
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Calcium/Calmodulin-Dependent Protein Kinase II Regulates <i>Caenorhabditis elegans</i> Locomotion in Concert With a Go/Gq Signaling Network
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Mutations in a C. elegans Gqα Gene Disrupt Movement, Egg Laying, and Viability
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EGL-10 Regulates G Protein Signaling in the C. elegans Nervous System and Shares a Conserved Domain with Many Mammalian Proteins
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Goα and Diacylglycerol Kinase Negatively Regulate the Gqα Pathway in C. elegans
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Immunocytochemical localization of D1 and D2 dopamine receptors in the basal ganglia of the rat: Light and electron microscopy
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Prolonged and Extrasynaptic Excitatory Action of Dopamine Mediated by D1 Receptors in the Rat Striatum<i>In Vivo</i>
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Facilitation of Synaptic Transmission by EGL-30 Gqα and EGL-8 PLCβ
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Most central nervous system D2 dopamine receptors are coupled to their effectors by Go
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In vivo release of [3H]γ-aminobutyric acid in the rat neostriatum—II. Opposing effects of D1 and D2 dopamine receptor stimulation in the dorsal caudate putamen
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Regulation of Dopamine D1 and D2 Receptors on Striatal Acetylcholine Release in Rats
The 2 references without a DOI — listed, not checked
no DOI — not checkedMcNamara, F.N. et al. Congenic D1A Dopamine receptor mutants: ethologically based resolution of behavioral topology indicates genetic background as a determinant of knockout phenotype. Neuropharmacology 28, 86–99 (2003).
no DOI — not checkedWang, H.Y., Undie, A.S. & Friedman, E. Evidence for the coupling of Gq protein to D1-like dopamine sites in rat striatum: possible role in dopamine-mediated inositol phosphate formation. Mol. Pharmacol. 48, 988–994 (1995).
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