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Inflammation induced by incomplete radiofrequency ablation accelerates tumor progression and hinders PD-1 immunotherapy
47/47 checkable references clean · checked 2026-07-22
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 47 checked references that resolve
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Association of PD-1, PD-1 Ligands, and Other Features of the Tumor Immune Microenvironment with Response to Anti–PD-1 Therapy
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Predictive correlates of response to the anti-PD-L1 antibody MPDL3280A in cancer patients
resolves10.1158/1078-0432.CCR-15-1352
PD-1 Blockade Boosts Radiofrequency Ablation–Elicited Adaptive Immune Responses against Tumor
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Clinically feasible approaches to potentiating cancer cell-based immunotherapies
resolves10.2217/hep-2016-0008
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Hepatocellular carcinoma with sarcomatous change arising after radiofrequency ablation for well-differentiated hepatocellular carcinoma
resolves10.3748/wjg.v10.i8.1137
Rapid progression of hepatocellular carcinoma after Radiofrequency Ablation
resolves10.1007/s10147-003-0328-6
Rapid and aggressive recurrence accompanied by portal tumor thrombus after radiofrequency ablation for hepatocellular carcinoma
resolves10.1016/j.jhep.2010.04.040
CD95 is a key mediator of invasion and accelerated outgrowth of mouse colorectal liver metastases following radiofrequency ablation
resolves10.1002/jso.21570
Radiofrequency ablation of colorectal liver metastases induces an inflammatory response in distant hepatic metastases but not in local accelerated outgrowth
resolves10.1016/j.canlet.2018.02.024
Insufficient radiofrequency ablation promotes proliferation of residual hepatocellular carcinoma via autophagy
resolves10.1007/s00262-013-1514-7
In vivo immunological antitumor effect of OK-432-stimulated dendritic cell transfer after radiofrequency ablation
resolves10.1016/j.immuni.2018.03.018
Co-inhibitory Molecule B7 Superfamily Member 1 Expressed by Tumor-Infiltrating Myeloid Cells Induces Dysfunction of Anti-tumor CD8+ T Cells
resolves10.3389/fimmu.2017.01106
Translational Significance for Tumor Metastasis of Tumor-Associated Macrophages and Epithelial–Mesenchymal Transition
resolves10.1016/j.celrep.2015.06.024
CCL2 Promotes Colorectal Carcinogenesis by Enhancing Polymorphonuclear Myeloid-Derived Suppressor Cell Population and Function
resolves10.1038/cmi.2017.135
The chemokine MCP-1 (CCL2) in the host interaction with cancer: a foe or ally?
resolves10.1136/gutjnl-2015-310514
Targeting of tumour-infiltrating macrophages via CCL2/CCR2 signalling as a therapeutic strategy against hepatocellular carcinoma
resolves10.1148/radiol.2511072175
Radiofrequency Ablation Induces Antigen-presenting Cell Infiltration and Amplification of Weak Tumor-induced Immunity
resolves10.1053/j.gastro.2009.12.051
Radiofrequency Thermal Ablation for Hepatocellular Carcinoma Stimulates Autologous NK-Cell Response
resolves10.1136/gutjnl-2014-308286
Additive antitumour response to the rabbit VX2 hepatoma by combined radio frequency ablation and toll like receptor 9 stimulation
resolves10.1186/1479-5876-10-230
After insufficient radiofrequency ablation, tumor-associated endothelial cells exhibit enhanced angiogenesis and promote invasiveness of residual hepatocellular carcinoma
resolves10.1111/cas.13191
Effect of a hypoxic microenvironment after radiofrequency ablation on residual hepatocellular cell migration and invasion
resolves10.1038/s41388-018-0169-4
Stress-induced phosphoprotein 1 mediates hepatocellular carcinoma metastasis after insufficient radiofrequency ablation
resolves10.1097/SLA.0b013e3181a38ef5
Accelerated Perinecrotic Outgrowth of Colorectal Liver Metastases Following Radiofrequency Ablation is a Hypoxia-Driven Phenomenon
resolves10.1186/1479-5876-8-73
Low temperature of radiofrequency ablation at the target sites can facilitate rapid progression of residual hepatic VX2 carcinoma
resolves10.1186/1479-5876-11-273
Insufficient radiofrequency ablation promotes epithelial-mesenchymal transition of hepatocellular carcinoma cells through Akt and ERK signaling pathways
resolves10.1002/hep.26526
Sublethal heat treatment promotes epithelial-mesenchymal transition and enhances the malignant potential of hepatocellular carcinoma
resolves10.1126/scitranslmed.aan3464
The systemic response to surgery triggers the outgrowth of distant immune-controlled tumors in mouse models of dormancy
resolves10.1038/s41467-018-03966-7
Snail promotes ovarian cancer progression by recruiting myeloid-derived suppressor cells via CXCR2 ligand upregulation
resolves10.1053/j.seminoncol.2014.02.003
Myeloid-Derived Suppressor Cells in Cancer: Therapeutic, Predictive, and Prognostic Implications
resolves10.1158/1078-0432.CCR-15-0676
Myeloid Cells and Related Chronic Inflammatory Factors as Novel Predictive Markers in Melanoma Treatment with Ipilimumab
resolves10.1038/nature20554
Overcoming resistance to checkpoint blockade therapy by targeting PI3Kγ in myeloid cells
resolves10.1126/scitranslmed.3007974
Disruption of CXCR2-Mediated MDSC Tumor Trafficking Enhances Anti-PD1 Efficacy
resolves10.1038/ncomms13050
Cancer-associated fibroblast-secreted CXCL16 attracts monocytes to promote stroma activation in triple-negative breast cancers
resolves10.1038/s41467-017-01566-5
Host STING-dependent MDSC mobilization drives extrinsic radiation resistance
resolves10.1136/gutjnl-2017-313738
Targeting both tumour-associated CXCR2<sup>+</sup> neutrophils and CCR2<sup>+</sup> macrophages disrupts myeloid recruitment and improves chemotherapeutic responses in pancreatic ductal adenocarcinoma
resolves10.1007/s00330-012-2561-8
Early diffuse recurrence of hepatocellular carcinoma after percutaneous radiofrequency ablation: analysis of risk factors
resolves10.1016/S0221-0363(07)89927-6
Influence de la proximité des gros vaisseaux sur les résultats du traitement des carcinomes hépato-cellulaires par radiofréquence : une étude contrôlée
The 2 references without a DOI — listed, not checked
no DOI — not checkedPortolani, N. et al. Aggressive recurrence after radiofrequency ablation of liver neoplasms. Hepato-Gastroenterol. 50, 2179–2184 (2003).
no DOI — not checkedYoshimura, T., Liu, M., Chen, X., Li, L. & Wang, J. M. Crosstalk between tumor cells and macrophages in stroma renders tumor cells as the primary source of MCP-1/CCL2 in Lewis lung carcinoma. Front. Immunol. 6, 332 (2015).
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