Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 59 checked references that resolve
resolves10.1016/S0140-6736(16)30054-XTrends in adult body-mass index in 200 countries from 1975 to 2014: a pooled analysis of 1698 population-based measurement studies with 19·2 million participants
resolves10.1210/er.2008-0021Bisphenol-A and the Great Divide: A Review of Controversies in the Field of Endocrine Disruption
resolves10.1016/j.envres.2014.08.008Urinary BPA measurements in children and mothers from six European member states: Overall results and determinants of exposure
resolves10.1289/ehp.1408989Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes
resolves10.1001/2012.jama.11461Association Between Urinary Bisphenol A Concentration and Obesity Prevalence in Children and Adolescents
resolves10.1007/BF02977657Differential regulation of thyroid hormone receptor-mediated function by endocrine disruptors
resolves10.1016/j.jsbmb.2008.02.006Dietary xenoestrogens differentially impair 3T3-L1 preadipocyte differentiation and persistently affect leptin synthesis
resolves10.1038/oby.2009.419Environmental Endocrine Disruptors Promote Adipogenesis in the 3T3‐L1 Cell Line through Glucocorticoid Receptor Activation
resolves10.1038/nutd.2013.43Bisphenol A induces differentiation of human preadipocytes in the absence of glucocorticoid and is inhibited by an estrogen-receptor antagonist
resolves10.1093/toxsci/kfi088Bisphenol A Accelerates Terminal Differentiation of 3T3-L1 Cells into Adipocytes through the Phosphatidylinositol 3-Kinase Pathway
resolves10.1371/journal.pone.0031109Rapid Insulinotropic Action of Low Doses of Bisphenol-A on Mouse and Human Islets of Langerhans: Role of Estrogen Receptor β
resolves10.1016/j.tox.2007.11.006Neonatal co-exposure to low doses of an ortho-PCB (PCB 153) and methyl mercury exacerbate defective developmental neurobehavior in mice
resolves10.1007/s00216-015-8492-xOccurrence of specific environmental risk factors in brain tissues of sudden infant death and sudden intrauterine unexpected death victims assessed with gas chromatography–tandem mass spectrometry
resolves10.1016/j.jsbmb.2006.06.028The mammary gland response to estradiol: Monotonic at the cellular level, non-monotonic at the tissue-level of organization?
resolves10.1371/journal.pone.0179583Low-dose exposure to bisphenols A, F and S of human primary adipocyte impacts coding and non-coding RNA profiles
resolves10.1254/jphs.FM0070034Life Style-Related Diseases of the Digestive System: Endocrine Disruptors Stimulate Lipid Accumulation in Target Cells Related to Metabolic Syndrome
resolves10.1073/pnas.1417731112Low-dose exposure to bisphenol A and replacement bisphenol S induces precocious hypothalamic neurogenesis in embryonic zebrafish
resolves10.3390/ijerph14091059Distribution of Non-Persistent Endocrine Disruptors in Two Different Regions of the Human Brain
resolves10.1016/j.envres.2017.03.030Assumed non-persistent environmental chemicals in human adipose tissue; matrix stability and correlation with levels measured in urine and serum
resolves10.1016/j.envint.2015.02.015Accumulation of 19 environmental phenolic and xenobiotic heterocyclic aromatic compounds in human adipose tissue
resolves10.1289/ehp.1408258Early-Life Bisphenol A Exposure and Child Body Mass Index: A Prospective Cohort Study
resolves10.1289/ehp.1306866Nano GO Consortium—A Team Science Approach to Assess Engineered Nanomaterials: Reliable Assays and Methods
resolves10.1038/jes.2014.36Exposure to bisphenol A among school children in eastern China: A multicenter cross-sectional study
resolves10.1002/hep.24685Low Doses of Bisphenol A Induce Gene Expression Related to Lipid Synthesis and Trigger Triglyceride Accumulation in Adult Mouse Liver
resolves10.1289/ehp.1001993Bisphenol A Exposure during Pregnancy Disrupts Glucose Homeostasis in Mothers and Adult Male Offspring
resolves10.1289/ehp.9710570Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol.
resolves10.1210/en.2015-1926Bisphenol A Promotes Adiposity and Inflammation in a Nonmonotonic Dose-response Way in 5-week-old Male and Female C57BL/6J Mice Fed a Low-calorie Diet
resolves10.1371/journal.pone.0155237Human Exposures to Bisphenol A, Bisphenol F and Chlorinated Bisphenol A Derivatives and Thyroid Function
resolves10.1016/j.bbrc.2012.08.065Molecular docking of bisphenol A and its nitrated and chlorinated metabolites onto human estrogen-related receptor-gamma
resolves10.1021/tx025548tMetabolism and Kinetics of Bisphenol A in Humans at Low Doses Following Oral Administration
resolves10.1289/ehp.0800376Bisphenol A Data in NHANES Suggest Longer than Expected Half-Life, Substantial Nonfood Exposure, or Both
The 8 references without a DOI — listed, not checked
no DOI — not checkedHeindel, J. J. et al. Parma consensus statement on metabolic disruptors. Environ. Heal. A Glob. Access Sci. Source 14, 54 (2015).
no DOI — not checkedSwaab, D. In The Human Hypothalamus: Basic and Clinical Aspects 79, 476 (Elsevier, 2003).
no DOI — not checkedSwaab, D. F. In Handbook of Clinical Neurology 80 (Elsevier, 2004).
no DOI — not checkedThe MAK-Collection for Occupational Health and Safety, Part IV: Biomonitoring Methods. in (eds. Angerer, J. & Hartwig, A.) 12, 169–184 (Wiley-VCH Verlag GmbH & Co. KGaA, 2010).
no DOI — not checkedDowle, M., Srinivasan, A., Short, T. & Lianoglou, S. data.table: Extension of Data.frame. (2015).
no DOI — not checkedR. Studio: Integrated Development Environment for R. (2015).
no DOI — not checkedR: A Language and Environment for Statistical Computing. (R Foundation for Statistical Computing, 2017).
no DOI — not checkedYoshida, K. & Bohn., J. tableone: Create ‘Table 1’ to Describe Baseline Characteristics. (2015).
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