Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 34 checked references that resolve
resolves10.1007/BF00966084The structural and functional heterogeneity of glutamic acid decarboxylase: A review
resolves10.1083/jcb.118.2.309Membrane anchoring of the autoantigen GAD65 to microvesicles in pancreatic beta-cells by palmitoylation in the NH2-terminal domain.
resolves10.1074/jbc.270.5.2241Targeting of the 67-kDa Isoform of Glutamic Acid Decarboxylase to Intracellular Organelles Is Mediated by Its Interaction with the NH2-terminal Region of the 65-kDa Isoform of Glutamic Acid Decarboxylase
resolves10.1016/S0021-9258(18)54849-3Pancreatic beta cells express two autoantigenic forms of glutamic acid decarboxylase, a 65-kDa hydrophilic form and a 64-kDa amphiphilic form which can be both membrane-bound and soluble.
resolves10.1111/j.1471-4159.1991.tb08211.xTwo Forms of the γ‐Aminobutyric Acid Synthetic Enzyme Glutamate Decarboxylase Have Distinct Intraneuronal Distributions and Cofactor Interactions
resolves10.1002/j.1460-2075.1991.tb08069.xGABA and pancreatic beta‐cells: colocalization of glutamic acid decarboxylase (GAD) and GABA with synaptic‐like microvesicles suggests their role in GABA storage and secretion.
resolves10.1074/jbc.272.3.1548Phosphorylation of Serine Residues 3, 6, 10, and 13 Distinguishes Membrane Anchored from Soluble Glutamic Acid Decarboxylase 65 and Is Restricted to Glutamic Acid Decarboxylase 65α
resolves10.1006/bbrc.1996.1898Mice Lacking the 65 kDa Isoform of Glutamic Acid Decarboxylase (GAD65) Maintain Normal Levels of GAD67 and GABA in Their Brains but Are Susceptible to Seizures
resolves10.1073/pnas.96.4.1698Increased anxiety and altered responses to anxiolytics in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase
resolves10.1152/jn.1998.80.3.1327Analysis of Excitatory and Inhibitory Spontaneous Synaptic Activity in Mouse Retinal Ganglion Cells
resolves10.1073/pnas.94.12.6496Cleft palate and decreased brain γ-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase
resolves10.1073/pnas.94.21.11451Cleft palate in mice with a targeted mutation in the γ-aminobutyric acid-producing enzyme glutamic acid decarboxylase 67
resolves10.1038/375488a0Essential functions of synapsins I and II in synaptic vesicle regulation
resolves10.1038/375493a0Distinct pools of synaptic vesicles in neurotransmitter release
resolves10.1523/JNEUROSCI.19-02-00539.1999The<i>Caenorhabditis elegans</i>Gene<i>unc-25</i>Encodes Glutamic Acid Decarboxylase and Is Required for Synaptic Transmission But Not Synaptic Development
resolves10.1073/pnas.83.1.170Presynaptic transmitter content controls the number of quanta released at a neuro-neuronal cholinergic synapse.
resolves10.1085/jgp.61.3.342Quantal Mechanism of Transmitter Release during Progressive Depletion of the Presynaptic Stores at a Ganglionic Synapse
resolves10.1152/jn.1996.75.5.2089Block of glutamate decarboxylase decreases GABAergic inhibition at the crayfish synapses: possible role of presynaptic metabotropic mechanisms
resolves10.1038/355050a0Postsynaptic contribution to long-term potentiation revealed by the analysis of miniature synaptic currents
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