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TSLP-activated dendritic cells induce an inflammatory T helper type 2 cell response through OX40 ligand

https://doi.org/10.1084/jem.20051135
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39/39 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

4 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 39 checked references that resolve
resolves10.1016/S1074-7613(00)80533-6
Cytokines Induce the Development of Functionally Heterogeneous T Helper Cell Subsets
resolves10.1146/annurev.iy.07.040189.001045
TH1 and TH2 Cells: Different Patterns of Lymphokine Secretion Lead to Different Functional Properties
resolves10.1016/S0091-6749(96)70197-5
Interleukin-10 regulation in normal subjects and patients with asthma
resolves10.1046/j.1365-2222.2001.01118.x
IL‐10: a key regulator of allergic disease
resolves10.1172/JCI117966
Interleukin-10 inhibits antigen-induced cellular recruitment into the airways of sensitized mice.
resolves10.1067/mai.2002.127512
CD4 T-helper cells engineered to produce IL-10 prevent allergen-induced airway hyperreactivity and inflammation
resolves10.1165/ajrcmb.21.5.3755
Interleukin-10 Gene Transfer to the Airway Regulates Allergic Mucosal Sensitization in Mice
resolves10.1038/90667
Pulmonary dendritic cells producing IL-10 mediate tolerance induced by respiratory exposure to antigen
resolves10.1146/annurev.immunol.15.1.297
INDUCTION OF TH1 AND TH2 CD4<sup>+</sup>T CELL RESPONSES:The Alternative Approaches
resolves10.1084/jem.180.4.1195
Th2 cell clonal anergy as a consequence of partial activation.
resolves10.1016/S0952-7915(97)80192-6
CD4+ T-cell subsets in autoimmunity
resolves10.1084/jem.183.3.979
The T cell-B cell interaction via OX40-OX40L is necessary for the T cell-dependent humoral immune response.
resolves10.1016/j.coi.2003.09.003
The master regulators of allergic inflammation: dendritic cells in Th2 sensitization
resolves10.1182/blood.V92.9.3338
OX40 Costimulation Enhances Interleukin-4 (IL-4) Expression at Priming and Promotes the Differentiation of Naive Human CD4+ T Cells Into High IL-4–Producing Effectors
resolves10.1084/jem.191.2.375
Critical Contribution of Ox40 Ligand to T Helper Cell Type 2 Differentiation in Experimental Leishmaniasis
resolves10.1084/jem.193.3.387
Development of Allergic Inflammation in a Murine Model of Asthma Is Dependent on the Costimulatory Receptor Ox40
resolves10.1038/nri954
The lineage decisions of helper T cells
resolves10.1016/S1074-7613(01)00191-1
OX40 Promotes Bcl-xL and Bcl-2 Expression and Is Essential for Long-Term Survival of CD4 T Cells
resolves10.1146/annurev.immunol.23.021704.115839
TNF/TNFR FAMILY MEMBERS IN COSTIMULATION OF T CELL RESPONSES
resolves10.1084/jem.183.6.2559
Interleukin-12 primes human CD4 and CD8 T cell clones for high production of both interferon-gamma and interleukin-10.
resolves10.4049/jimmunol.157.3.1127
IL-12 induces human T cells secreting IL-10 with IFN-γ
resolves10.1084/jem.184.2.473
Differentiation and stability of T helper 1 and 2 cells derived from naive human neonatal CD4+ T cells, analyzed at the single-cell level.
resolves10.4049/jimmunol.152.10.4775
In vitro maturation of human neonatal CD4 T lymphocytes. II. Cytokines present at priming modulate the development of lymphokine production.
resolves10.1006/cyto.1997.0193
INTERLEUKIN 12 AND B7/CD28 INTERACTION SYNERGISTICALLY UPREGULATE INTERLEUKIN 10 PRODUCTION BY HUMAN T CELLS
resolves10.1016/S1074-7613(02)00278-9
In the Absence of IL-12, CD4+ T Cell Responses to Intracellular Pathogens Fail to Default to a Th2 Pattern and Are Host Protective in an IL-10−/− Setting
resolves10.4049/jimmunol.171.11.5751
TNF Receptor-Associated Factor 6 Deficiency during Hemopoiesis Induces Th2-Polarized Inflammatory Disease
resolves10.1038/ni712
Toll-like receptors control activation of adaptive immune responses
resolves10.4049/jimmunol.171.12.6954
Paradoxical Dampening of Anti-Islet Self-Reactivity but Promotion of Diabetes by OX40 Ligand
resolves10.1002/1521-4141(200010)30:10<2815::AID-IMMU2815>3.0.CO;2-#
Contribution of OX40/OX40 ligand interaction to the pathogenesis of rheumatoid arthritis
resolves10.4049/jimmunol.167.5.2991
Critical Involvement of OX40 Ligand Signals in the T Cell Priming Events During Experimental Autoimmune Encephalomyelitis
resolves10.1152/ajpgi.00450.2002
Therapeutic effect of anti-OX40L and anti-TNF-α MAbs in a murine model of chronic colitis
resolves10.1006/smim.1998.0146
OX-40: life beyond the effector T cell stage
resolves10.4049/jimmunol.172.11.6735
A Signal through OX40 (CD134) Allows Anergic, Autoreactive T Cells to Acquire Effector Cell Functions
resolves10.4049/jimmunol.171.11.5997
OX40-Mediated Memory T Cell Generation Is TNF Receptor-Associated Factor 2 Dependent
resolves10.1084/jem.190.7.953
Tumor Necrosis Factor α Is a Critical Component of Interleukin 13–Mediated Protective T Helper Cell Type 2 Responses during Helminth Infection
resolves10.1067/mai.2003.1554
TNF-α contributes to the development of allergic rhinitis in mice
resolves10.1111/j.1365-2249.2004.02649.x
Early up-regulation of Th2 cytokines and late surge of Th1 cytokines in an atopic dermatitis model
resolves10.1038/ni1122
Induction of T helper type 1–like regulatory cells that express Foxp3 and protect against airway hyper-reactivity
resolves10.1038/ni1048
Human thymic stromal lymphopoietin promotes dendritic cell–mediated CD4+ T cell homeostatic expansion
The 4 references without a DOI — listed, not checked
no DOI — not checked2023072513272926700_BIB4
no DOI — not checked2023072513272926700_BIB13
no DOI — not checked2023072513272926700_BIB37
no DOI — not checked2023072513272926700_BIB38
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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