Reference health
Rescue of behavioral and EEG deficits in male and female Mecp2-deficient mice by delayed Mecp2 gene reactivation
51/51 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
The 51 checked references that resolve
resolves10.1038/13810
Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2
resolves10.1523/JNEUROSCI.1316-12.2012
MeCP2 Is Critical for Maintaining Mature Neuronal Networks and Global Brain Anatomy during Late Stages of Postnatal Brain Development and in the Mature Adult Brain
resolves10.1093/hmg/dds208
Postnatal inactivation reveals enhanced requirement for MeCP2 at distinct age windows
resolves10.1016/j.mcn.2004.07.006
MECP2 is progressively expressed in post-migratory neurons and is involved in neuronal maturation rather than cell fate decisions
resolves10.31887/DCNS.2012.14.3/jneul
The relationship of Rett syndrome and <i>MECP2</i> disorders to autism
resolves10.1016/S0887-8994(02)00624-0
Identification of MeCP2 mutations in a series of females with autistic disorder
resolves10.1038/85899
A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome
resolves10.1038/85906
Deficiency of methyl-CpG binding protein-2 in CNS neurons results in a Rett-like phenotype in mice
resolves10.1016/S0896-6273(02)00768-7
Mice with Truncated MeCP2 Recapitulate Many Rett Syndrome Features and Display Hyperacetylation of Histone H3
resolves10.1093/brain/awl022
Mecp2 deficiency is associated with learning and cognitive deficits and altered gene activity in the hippocampal region of mice
resolves10.1007/s00109-010-0704-4
Readthrough of nonsense mutations in Rett syndrome: evaluation of novel aminoglycosides and generation of a new mouse model
resolves10.1038/nn.2997
Rett syndrome mutation MeCP2 T158A disrupts DNA binding, protein stability and ERP responses
resolves10.1016/j.brainres.2007.08.039
Ube3a mRNA and protein expression are not decreased in Mecp2 mutant mice
resolves10.1242/dmm.011007
Preclinical research in Rett syndrome: setting the foundation for translational success
resolves10.1523/JNEUROSCI.2159-12.2012
Brain Activity Mapping in
<i>Mecp2</i>
Mutant Mice Reveals Functional Deficits in Forebrain Circuits, Including Key Nodes in the Default Mode Network, that are Reversed with Ketamine Treatment
resolves10.1371/journal.pone.0035396
Daily Rhythmic Behaviors and Thermoregulatory Patterns Are Disrupted in Adult Female MeCP2-Deficient Mice
resolves10.1523/JNEUROSCI.3156-09.2009
Pathophysiology of Locus Ceruleus Neurons in a Mouse Model of Rett Syndrome
resolves10.1523/JNEUROSCI.2623-05.2006
Learning and Memory and Synaptic Plasticity Are Impaired in a Mouse Model of Rett Syndrome
resolves10.1073/pnas.0506071102
Reduced cortical activity due to a shift in the balance between excitation and inhibition in a mouse model of Rett Syndrome
resolves10.1016/j.nbd.2009.12.018
Alterations of cortical and hippocampal EEG activity in MeCP2-deficient mice
resolves10.1523/JNEUROSCI.4225-08.2009
MeCP2-Mediated Transcription Repression in the Basolateral Amygdala May Underlie Heightened Anxiety in a Mouse Model of Rett Syndrome
resolves10.1016/j.neuron.2007.08.018
MeCP2 Controls Excitatory Synaptic Strength by Regulating Glutamatergic Synapse Number
resolves10.1002/hipo.20389
The MeCP2‐null mouse hippocampus displays altered basal inhibitory rhythms and is prone to hyperexcitability
resolves10.1093/hmg/dds406
Female Mecp2+/− mice display robust behavioral deficits on two different genetic backgrounds providing a framework for pre-clinical studies
resolves10.1093/brain/aws096
Morphological and functional reversal of phenotypes in a mouse model of Rett syndrome
resolves10.1006/dbio.2002.0597
Efficient Recombination in Diverse Tissues by a Tamoxifen-Inducible Form of Cre: A Tool for Temporally Regulated Gene Activation/Inactivation in the Mouse
resolves10.1523/JNEUROSCI.0057-11.2011
MeCP2 Is Critical within HoxB1-Derived Tissues of Mice for Normal Lifespan
resolves10.1093/hmg/dds433
Selective preservation of MeCP2 in catecholaminergic cells is sufficient to improve the behavioral phenotype of male and female Mecp2-deficient mice
resolves10.1016/j.expneurol.2012.09.005
Regional MeCP2 expression levels in the female MeCP2-deficient mouse brain correlate with specific behavioral impairments
resolves10.1111/j.1601-183X.2009.00551.x
Early postnatal behavioral changes in the <i>Mecp2</i>‐308 truncation mouse model of Rett syndrome
resolves10.1093/hmg/ddn026
Targeted delivery of an Mecp2 transgene to forebrain neurons improves the behavior of female Mecp2-deficient mice
resolves10.1073/pnas.0912257106
Loss of MeCP2 in aminergic neurons causes cell-autonomous defects in neurotransmitter synthesis and specific behavioral abnormalities
resolves10.1016/S0387-7604(01)00351-5
Neurobiology of Rett syndrome: a genetic disorder of synapse development
resolves10.1016/j.nbd.2007.04.005
Mecp2 deficiency leads to delayed maturation and altered gene expression in hippocampal neurons
resolves10.1002/ana.10633
Olfactory biopsies demonstrate a defect in neuronal development in Rett's syndrome
resolves10.1152/jn.00800.2010
Network hyperexcitability in hippocampal slices from<i>Mecp2</i>mutant mice revealed by voltage-sensitive dye imaging
resolves10.1203/01.pdr.0000238302.84552.d0
Autonomic Nervous System Dysregulation: Breathing and Heart Rate Perturbation During Wakefulness in Young Girls with Rett Syndrome
resolves10.2144/00291bm09
Preparation of PCR-Quality Mouse Genomic DNA with Hot Sodium Hydroxide and Tris (HotSHOT)
resolves10.1016/j.jneumeth.2008.03.001
A glue-based, screw-free method for implantation of intra-cranial electrodes in young mice
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