At the dated check, the references listed below either did not resolve in
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The 33 checked references that resolve
resolves10.1038/ncomms7997APOBEC family mutational signatures are associated with poor prognosis translocations in multiple myeloma
resolves10.1038/ncomms3997Heterogeneity of genomic evolution and mutational profiles in multiple myeloma
resolves10.1200/JCO.2014.59.1503Mutational Spectrum, Copy Number Changes, and Outcome: Results of a Sequencing Study of Patients With Newly Diagnosed Myeloma
resolves10.1038/leu.2013.199Intraclonal heterogeneity is a critical early event in the development of myeloma and precedes the development of clinical symptoms
resolves10.1038/leu.2017.345Biological and prognostic impact of APOBEC-induced mutations in the spectrum of plasma cell dyscrasias and multiple myeloma cell lines
resolves10.1038/bcj.2017.94High somatic mutation and neoantigen burden are correlated with decreased progression-free survival in multiple myeloma
resolves10.1126/science.aaa6806High burden and pervasive positive selection of somatic mutations in normal human skin
resolves10.1016/j.celrep.2018.01.088Somatic Mutational Landscape of Splicing Factor Genes and Their Functional Consequences across 33 Cancer Types
resolves10.1038/s41375-018-0103-3Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms
resolves10.1101/181339Patterns of structural variation in human cancer
resolves10.1038/nature13115Constitutional and somatic rearrangement of chromosome 21 in acute lymphoblastic leukaemia
resolves10.1001/jamaoncol.2015.2010Treatment With Carfilzomib-Lenalidomide-Dexamethasone With Lenalidomide Extension in Patients With Smoldering or Newly Diagnosed Multiple Myeloma
resolves10.1038/leu.2014.70Promiscuous MYC locus rearrangements hijack enhancers but mostly super-enhancers to dysregulate MYC expression in multiple myeloma
resolves10.1002/gcc.20123Characterization of oncogene dysregulation in multiple myeloma by combined FISH and DNA microarray analyses
resolves10.1038/bcj.2017.12Bi-allelic inactivation is more prevalent at relapse in multiple myeloma, identifying RB1 as an independent prognostic marker
resolves10.1182/blood-2012-12-471888Characterization of IGH locus breakpoints in multiple myeloma indicates a subset of translocations appear to occur in pregerminal center B cells
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