Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
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The 62 checked references that resolve
resolves10.1177/026988110001400418Evidence that long-lasting potentiation of amygdala efferents in the right hemisphere underlies pharmacological stressor (FG-7142) induced lasting increases in anxiety-like behaviour: role of GABA tone in initiation of brain and behavioural changes
resolves10.1038/12146Anxiety: at the intersection of genes and experience
resolves10.1006/bbrc.1996.1898Mice Lacking the 65 kDa Isoform of Glutamic Acid Decarboxylase (GAD65) Maintain Normal Levels of GAD67 and GABA in Their Brains but Are Susceptible to Seizures
resolves10.1073/pnas.94.12.6496Cleft palate and decreased brain γ-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase
resolves10.1176/appi.ajp.161.2.195Psychobiological Mechanisms of Resilience and Vulnerability: Implications for Successful Adaptation to Extreme Stress
resolves10.1523/JNEUROSCI.3301-04.2005Regulation of Gephyrin and GABA<sub>A</sub>Receptor Binding within the Amygdala after Fear Acquisition and Extinction
resolves10.1038/12207Decreased GABAA-receptor clustering results in enhanced anxiety and a bias for threat cues
resolves10.1101/lm.51305Effects of post-training hippocampal injections of midazolam on fear conditioning
resolves10.1038/sj.npp.1300478Increased Fear Learning Coincides with Neuronal Dysinhibition and Facilitated LTP in the Basolateral Amygdala following Benzodiazepine Withdrawal in Rats
resolves10.1074/jbc.274.52.37200The Hydrophilic Isoform of Glutamate Decarboxylase, GAD67, Is Targeted to Membranes and Nerve Terminals Independent of Dimerization with the Hydrophobic Membrane-anchored Isoform, GAD65
resolves10.1073/pnas.96.4.1698Increased anxiety and altered responses to anxiolytics in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase
resolves10.1038/nrn2207Long-term synaptic plasticity in hippocampal interneurons
resolves10.1152/jn.1997.77.1.341Similar Inhibitory Processes Dominate the Responses of Cat Lateral Amygdaloid Projection Neurons to Their Various Afferents
resolves10.1152/jn.1997.77.1.353Synaptic and Synaptically Activated Intrinsic Conductances Underlie Inhibitory Potentials in Cat Lateral Amygdaloid Projection Neurons In Vivo
resolves10.1006/meth.2001.1262Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method
resolves10.1038/29312Calcium-permeable AMPA receptors mediate long-term potentiation in interneurons in the amygdala
resolves10.1037/0735-7044.111.4.683Functional inactivation of the lateral and basal nuclei of the amygdala by muscimol infusion prevents fear conditioning to an explicit conditioned stimulus and to contextual stimuli.
resolves10.1002/cne.21185Postsynaptic targets of somatostatin‐containing interneurons in the rat basolateral amygdala
resolves10.1177/1073858405284360Regulation of Cortical Interneurons by Neurotrophins: From Development to Cognitive Disorders
resolves10.1523/JNEUROSCI.2260-05.2005Previous Stress Facilitates Fear Memory, Attenuates GABAergic Inhibition, and Increases Synaptic Plasticity in the Rat Basolateral Amygdala
resolves10.1038/nn1732Generalization of amygdala LTP and conditioned fear in the absence of presynaptic inhibition
resolves10.1111/j.1471-4159.2005.03250.xProteolytic degradation of glutamate decarboxylase mediates disinhibition of hippocampal CA3 pyramidal cells in cathepsin D‐deficient mice
resolves10.1101/lm.39401Identification of Genes Expressed in the Amygdala During the Formation of Fear Memory
resolves10.1016/S0304-3940(02)00387-7Reduction of extracellular GABA in the mouse amygdala during and following confrontation with a conditioned fear stimulus
resolves10.1523/JNEUROSCI.20-23-08909.2000Putative Cortical and Thalamic Inputs Elicit Convergent Excitation in a Population of GABAergic Interneurons of the Lateral Amygdala
resolves10.1073/pnas.96.22.12911The role of the synthetic enzyme GAD65 in the control of neuronal γ-aminobutyric acid release
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