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Symptom‐relieving and neuroprotective effects of the phytocannabinoid Δ<sup>9</sup>‐THCV in animal models of Parkinson's disease

https://doi.org/10.1111/j.1476-5381.2011.01278.x
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Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

The 37 checked references that resolve
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Increased vulnerability to 6-hydroxydopamine lesion and reduced development of dyskinesias in mice lacking CB1 cannabinoid receptors
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The psychoactive plant cannabinoid, Δ<sup>9</sup>‐tetrahydrocannabinol, is antagonized by Δ<sup>8</sup>‐ and Δ<sup>9</sup>‐tetrahydrocannabivarin in mice<i>in vivo</i>
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Unilateral 6-hydroxydopamine lesions of nigrostriatal dopaminergic neurons increased CB1 receptor mRNA levels in the caudate-putamen
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Motor actions of cannabinoids in the basal ganglia output nuclei
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Effects of CB1 cannabinoid receptor modulating compounds on the hyperkinesia induced by high‐dose levodopa in the reserpine‐treated rat model of Parkinson's disease
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Parkinson's disease
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Evidence that the plant cannabinoid Δ<sup>9</sup>‐tetrahydrocannabivarin is a cannabinoid CB<sub>1</sub>and CB<sub>2</sub>receptor antagonist
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