Reference health

Neuroendocrine immunology of fibromyalgia

https://doi.org/10.1111/j.1749-6632.2009.05344.x
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1 of 48 checkable references need attention · checked 2026-07-22

At the dated check, the references listed below either did not resolve in Crossref or DataCite, or carried a retraction notice. Each one is shown with the registry record that put it there.

10 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

References needing attention

does not resolve to a known work10.1002/1520-6394(2000)12:1 <2::AID-DA2>3.0.CO;2-4
The 47 checked references that resolve
resolves10.1002/art.1780330203
The american college of rheumatology 1990 criteria for the classification of fibromyalgia
resolves10.1016/S0049-0172(96)80011-4
Familial aggregation in the fihromyalgia syndrome
resolves10.1002/art.20042
Family study of fibromyalgia
resolves10.1007/s00296-002-0260-5
Significance of catechol-O-methyltransferase gene polymorphism in fibromyalgia syndrome
resolves10.1006/nbdi.1999.0262
The T102C Polymorphism of the 5-HT2A-Receptor Gene in Fibromyalgia
resolves10.1038/sj.mp.4001568
Erratum: An association between fibromyalgia and the dopamine D4 receptor exon III repeat polymorphism and relationship to novelty seeking personality traits
resolves10.2217/14622416.8.1.67
The Genetics of Fibromyalgia Syndrome
resolves10.1016/S1521-6942(03)00035-4
Chronic widespread pain and fibromyalgia: what we know, and what we need to know
resolves10.1007/s11916-002-0049-y
Rheumatic mimics and selected triggers of fibromyalgia
resolves10.1126/science.273.5276.749
Why Stress Is Bad for Your Brain
resolves10.1007/s11916-002-0046-1
Peripheral and central sensitization in fibromyalgia: Pathogenetic role
resolves10.1016/S0304-3959(98)00154-7
Windup leads to characteristics of central sensitization
resolves10.1016/S0304-3959(00)00432-2
Abnormal sensitization and temporal summation of second pain (wind-up) in patients with fibromyalgia syndrome
resolves10.1002/art.1780371106
Elevated cerebrospinal fluid levels of substance p in patients with the fibromyalgia syndrome
resolves10.1046/j.1526-4637.2001.01030.x
Evidence for Abnormal Pain Processing in Fibromyalgia Syndrome
resolves10.1016/S0304-3959(00)00284-0
Changes in the concentrations of amino acids in the cerebrospinal fluid that correlate with pain in patients with fibromyalgia: implications for nitric oxide pathways
resolves10.1016/j.neubiorev.2003.11.008
Descending modulation of pain
resolves10.1016/0028-3908(93)90076-F
Monoaminergic control of the release of calcitonin gene-related peptide- and substance P-like materials from rat spinal cord slices
resolves10.1002/art.1780401103
The neuroscience and endocrinology of fibromyalgia
resolves10.1016/S0889-8529(01)00004-4
The hypothalamic–pituitary–adrenal axis in the pathogenesis of rheumatic diseases
resolves10.1007/s003930050242
Secretory pattern of GH, TSH, thyroid hormones, ACTH, cortisol, FSH, and LH in patients with fibromyalgia syndrome following systemic injection of the relevant hypothalamic-releasing hormones
resolves10.1016/S0002-9343(99)00074-1
Reduced hypothalamic-pituitary and sympathoadrenal responses to hypoglycemia in women with fibromyalgia syndrome
resolves10.1046/j.1365-2265.2001.01373.x
A comparison between low‐dose (1 µg), standard‐dose (250 µg) ACTH stimulation tests and insulin tolerance test in the evaluation of hypothalamo–pituitary–adrenal axis in primary fibromyalgia syndrome
resolves10.1002/1529-0131(200004)43:4<872::AID-ANR19>3.0.CO;2-T
Responses of the sympathetic nervous system and the hypothalamic–pituitary–adrenal axis to interleukin-6: A pilot study in fibromyalgia
resolves10.1080/030097400750001743-1
Neuroendocrine and hormonal perturbations and relations to the serotonergic system in fibromyalgia patients
resolves10.1016/S0889-857X(01)00009-6
Evaluation and management of endocrine dysfunction in fibromyalgia
resolves10.1007/s11916-002-0050-5
Neuroendocrine abnormalities in fibromyalgia
resolves10.1016/S0306-9877(02)00294-3
A metabolic basis for fibromyalgia and its related disorders: the possible role of resistance to thyroid hormone
resolves10.1016/S0049-0172(00)80008-6
Autonomic nervous system dysfunction may explain the multisystem features of fibromyalgia
resolves10.1007/s11916-996-0012-4
Fibromyalgia as a sympathetically maintained pain syndrome
resolves10.1016/j.tips.2006.12.003
Neuropeptide tyrosine and pain
resolves10.1016/j.it.2004.08.005
More sympathy for autoimmunity with neuropeptide Y?
resolves10.1159/000026321
Differential Effects of Neuropeptides on Cytokine Production by Mouse Helper T Cell Subsets
resolves10.1073/pnas.95.21.12544
Neuropeptides, by direct interaction with T cells, induce cytokine secretion and break the commitment to a distinct T helper phenotype
resolves10.1016/S0950-3579(96)80022-7
Neurohormonal perturbations in fibromyalgia
resolves10.1002/art.1780370812
The prevalence and clinical impact of fibromyalgia in systemic lupus erythematosus
resolves10.1177/096120339400300310
Fibromyalgia Syndrome and Disease Activity in Systemic Lupus Erythematosus
resolves10.3109/03009748909095399
Fibromyalgia Features in Patients with Primary Sjögren's Syndrome: Evidence of a Relationship with Psychological Depression
resolves10.1007/s00296-007-0413-7
Is there a predisposition for the development of autoimmune diseases in patients with fibromyalgia? Retrospective analysis with long term follow-up
resolves10.1007/s10067-006-0237-y
The frequency of thyroid antibodies in fibromyalgia patients and their relationship with symptoms
resolves10.1080/08916930701620035
Anti-polymer antibodies are inversely correlated with pain and fatigue severity in patients with fibromyalgia syndrome
resolves10.7326/0003-4819-108-6-909_1
Fibromyalgia and Interleukin-2 Therapy for Malignancy
resolves10.1093/rheumatology/40.7.743
Cytokines play an aetiopathogenetic role in fibromyalgia: a hypothesis and pilot study
resolves10.1002/art.22026
Reduced levels of antiinflammatory cytokines in patients with chronic widespread pain
resolves10.1097/AJP.0b013e31817e13a3
The Role of IL-8 in Patients With Fibromyalgia
resolves10.3181/0808-RM-254
Plasma Cytokine Fluctuations over Time in Healthy Controls and Patients with Fibromyalgia
resolves10.1159/000135622
Neuroendocrine Dysfunction in Sjögren’s Syndrome
The 10 references without a DOI — listed, not checked
no DOI — not checkedFibromyalgia syndrome (FM) and nonarticular tenderness in relatives of patients with FM
no DOI — not checkedSerotonin levels, pain threshold, and fibromyalgia symptoms in the general population
no DOI — not checkedAltered reactivity of the hypothalamic‐pituitary‐adrenal axis in the primary fibromyalgia syndrome
no DOI — not checkedDiurnal hormone variation in fibromyalgia syndrome: a comparison with rheumatoid arthritis
no DOI — not checkedAltered reactivity of the hypothalamic‐pituitary‐adrenal axis in the primary fibromyalgia syndrome
no DOI — not checkedSleep disturbances, fibromyalgia and primary Sjögren's syndrome
no DOI — not checkedSevere rheumatoid arthritis (RA), worse outcomes, comorbid illness, and sociodermographic disadvantage characterize RA patients with fibromyalgia
no DOI — not checkedAntipolymer antibodies in a Danish fibromyalgia patients
no DOI — not checkedCytokine patterns in fibromyalgia and their correlation with clinical manifestations
no DOI — not checkedThe fibromyalgia syndrome: review of clinical presentation, pathogenesis, outcome measures, and treatment
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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