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Correction of Sickle Cell Disease in Adult Mice by Interference with Fetal Hemoglobin Silencing

https://doi.org/10.1126/science.1211053
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1 of 43 checkable references need attention · checked 2026-07-26

At the dated check, the references listed below either did not resolve in Crossref or DataCite, or carried a retraction notice. Each one is shown with the registry record that put it there.

References needing attention

does not resolve to a known work10.1073/pnas.011404098
The 42 checked references that resolve
resolves10.1136/bmj.1.5237.1434
Two Families Showing Interaction of Haemoglobin C or Thalassaemia with High Foetal Haemoglobin in Adults
resolves10.1182/blood.V21.3.261.261
Hereditary Persistence of Fetal Hemoglobin: A Study of 79 Affected Persons in 15 Negro Families in Baltimore
resolves10.1038/178792a0
A Specific Chemical Difference Between the Globins of Normal Human and Sickle-Cell Anæmia Hæmoglobin
resolves10.1056/NEJM199406093302303
Mortality In Sickle Cell Disease -- Life Expectancy and Risk Factors for Early Death
resolves10.1038/ng2108
A QTL influencing F cell production maps to a gene encoding a zinc-finger protein on chromosome 2p15
resolves10.1073/pnas.0711566105
Genome-wide association study shows <i>BCL11A</i> associated with persistent fetal hemoglobin and amelioration of the phenotype of β-thalassemia
resolves10.1073/pnas.0804799105
DNA polymorphisms at the <i>BCL11A</i> , <i>HBS1L-MYB</i> , and β- <i>globin</i> loci associate with fetal hemoglobin levels and pain crises in sickle cell disease
resolves10.1126/science.1165409
Human Fetal Hemoglobin Expression Is Regulated by the Developmental Stage-Specific Repressor <i>BCL11A</i>
resolves10.1038/nature08243
Developmental and species-divergent globin switching are driven by BCL11A
resolves10.1101/gad.1897310
Transcriptional silencing of γ-globin by BCL11A involves long-range interactions and cooperation with SOX6
resolves10.1182/blood-2010-08-300723
Therapeutic levels of fetal hemoglobin in erythroid progeny of β-thalassemic CD34+ cells after lentiviral vector-mediated gene transfer
resolves10.1182/blood-2010-11-316893
Transcriptional regulation of fetal to adult hemoglobin switching: new therapeutic opportunities
resolves10.1182/blood-2003-05-1442
A mouse model for visualization and conditional mutations in the erythroid lineage
resolves10.1038/ni925
Bcl11a is essential for normal lymphoid development
resolves10.1073/pnas.79.14.4428
5-Azacytidine stimulates fetal hemoglobin synthesis in anemic baboons.
resolves10.1056/NEJM198212093072401
5-Azacytidine Selectively Increases γ-Globin Synthesis in a Patient with β<sup>+</sup>Thalassemia
resolves10.1073/pnas.1006774107
Chemical genetic strategy identifies histone deacetylase 1 (HDAC1) and HDAC2 as therapeutic targets in sickle cell disease
resolves10.1126/science.278.5339.873
Knockout-Transgenic Mouse Model of Sickle Cell Disease
resolves10.1126/science.278.5339.876
Transgenic Knockout Mice with Exclusively Human Sickle Hemoglobin and Sickle Cell Disease
resolves10.1126/science.1065806
Correction of Sickle Cell Disease in Transgenic Mouse Models by Gene Therapy
resolves10.1182/blood.V97.2.410
Second generation knockout sickle mice: the effect of HbF
resolves10.1126/science.1152092
Treatment of Sickle Cell Anemia Mouse Model with iPS Cells Generated from Autologous Skin
resolves10.1001/archinte.1910.00050330050003
PECULIAR ELONGATED AND SICKLE-SHAPED RED BLOOD CORPUSCLES IN A CASE OF SEVERE ANEMIA
resolves10.1016/j.cell.2006.07.007
Knocking down Disease with siRNAs
resolves10.1126/science.1178178
Therapeutic Silencing of MicroRNA-122 in Primates with Chronic Hepatitis C Virus Infection
resolves10.1073/pnas.1109522108
Combination therapy utilizing shRNA knockdown and an optimized resistant transgene for rescue of diseases caused by misfolded proteins
resolves10.1126/scitranslmed.3002594
A Tale of Two SCIDs
resolves10.1038/nature08543
Direct inhibition of the NOTCH transcription factor complex
resolves10.1016/j.cbpa.2010.03.022
A complex task? Direct modulation of transcription factors with small molecules
resolves10.1073/pnas.92.12.5655
Use of yeast artificial chromosomes (YACs) in studies of mammalian development: production of beta-globin locus YAC mice carrying human globin developmental mutants.
resolves10.1182/blood.V90.11.4602
The Human β Globin Locus Introduced by YAC Transfer Exhibits a Specific and Reproducible Pattern of Developmental Regulation in Transgenic Mice
resolves10.1002/gene.10161
<i>vavCre</i> Transgenic mice: A tool for mutagenesis in hematopoietic and endothelial lineages
resolves10.1126/science.7660125
Inducible Gene Targeting in Mice
resolves10.1073/pnas.0509322103
Methyl binding domain protein 2 mediates γ-globin gene silencing in adult human βYAC transgenic mice
resolves10.1158/0008-5472.CAN-05-4225
Histone Deacetylase Inhibitors Suppress the Inducibility of Nuclear Factor-κB by Tumor Necrosis Factor-α Receptor-1 Down-regulation
resolves10.1073/pnas.0712051105
HDAC inhibitor reduces cytokine storm and facilitates induction of chimerism that reverses lupus in anti-CD3 conditioning regimen
resolves10.1182/blood-2009-02-204990
The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice
resolves10.1007/BF02982061
A New Anti-Hemoglobin F Antibody Against Synthetic Peptides for the Detection of F-Cell Precursors (F-blasts) in Bone Marrow
resolves10.1101/gad.1627208
<i>Rb</i> intrinsically promotes erythropoiesis by coupling cell cycle exit with mitochondrial biogenesis
resolves10.1101/gad.1861209
Transcriptional competence and the active marking of tissue-specific enhancers by defined transcription factors in embryonic and induced pluripotent stem cells
resolves10.1186/gb-2003-4-5-p3
DAVID: Database for Annotation, Visualization, and Integrated Discovery
resolves10.1073/pnas.90.23.11381
Germ-line transmission and developmental regulation of a 150-kb yeast artificial chromosome containing the human beta-globin locus in transgenic mice.
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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