At the dated check, the references listed below either did not resolve in
Crossref or DataCite, or carried a retraction notice. Each one is shown with the
registry record that put it there.
The 42 checked references that resolve
resolves10.1136/bmj.1.5237.1434Two Families Showing Interaction of Haemoglobin C or Thalassaemia with High Foetal Haemoglobin in Adults
resolves10.1182/blood.V21.3.261.261Hereditary Persistence of Fetal Hemoglobin: A Study of 79 Affected Persons in 15 Negro Families in Baltimore
resolves10.1038/178792a0A Specific Chemical Difference Between the Globins of Normal Human and Sickle-Cell Anæmia Hæmoglobin
resolves10.1038/ng2108A QTL influencing F cell production maps to a gene encoding a zinc-finger protein on chromosome 2p15
resolves10.1073/pnas.0711566105Genome-wide association study shows
<i>BCL11A</i>
associated with persistent fetal hemoglobin and amelioration of the phenotype of β-thalassemia
resolves10.1073/pnas.0804799105DNA polymorphisms at the
<i>BCL11A</i>
,
<i>HBS1L-MYB</i>
, and β-
<i>globin</i>
loci associate with fetal hemoglobin levels and pain crises in sickle cell disease
resolves10.1126/science.1165409Human Fetal Hemoglobin Expression Is Regulated by the Developmental Stage-Specific Repressor
<i>BCL11A</i>
resolves10.1038/nature08243Developmental and species-divergent globin switching are driven by BCL11A
resolves10.1101/gad.1897310Transcriptional silencing of γ-globin by BCL11A involves long-range interactions and cooperation with SOX6
resolves10.1182/blood-2010-08-300723Therapeutic levels of fetal hemoglobin in erythroid progeny of β-thalassemic CD34+ cells after lentiviral vector-mediated gene transfer
resolves10.1038/ni925Bcl11a is essential for normal lymphoid development
resolves10.1073/pnas.1006774107Chemical genetic strategy identifies histone deacetylase 1 (HDAC1) and HDAC2 as therapeutic targets in sickle cell disease
resolves10.1126/science.1152092Treatment of Sickle Cell Anemia Mouse Model with iPS Cells Generated from Autologous Skin
resolves10.1126/science.1178178Therapeutic Silencing of MicroRNA-122 in Primates with Chronic Hepatitis C Virus Infection
resolves10.1073/pnas.1109522108Combination therapy utilizing shRNA knockdown and an optimized resistant transgene for rescue of diseases caused by misfolded proteins
resolves10.1073/pnas.92.12.5655Use of yeast artificial chromosomes (YACs) in studies of mammalian development: production of beta-globin locus YAC mice carrying human globin developmental mutants.
resolves10.1182/blood.V90.11.4602The Human β Globin Locus Introduced by YAC Transfer Exhibits a Specific and Reproducible Pattern of Developmental Regulation in Transgenic Mice
resolves10.1002/gene.10161<i>vavCre</i> Transgenic mice: A tool for mutagenesis in hematopoietic and endothelial lineages
resolves10.1073/pnas.0509322103Methyl binding domain protein 2 mediates γ-globin gene silencing in adult human βYAC transgenic mice
resolves10.1158/0008-5472.CAN-05-4225Histone Deacetylase Inhibitors Suppress the Inducibility of Nuclear Factor-κB by Tumor Necrosis Factor-α Receptor-1 Down-regulation
resolves10.1073/pnas.0712051105HDAC inhibitor reduces cytokine storm and facilitates induction of chimerism that reverses lupus in anti-CD3 conditioning regimen
resolves10.1182/blood-2009-02-204990The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice
resolves10.1007/BF02982061A New Anti-Hemoglobin F Antibody Against Synthetic Peptides for the Detection of F-Cell Precursors (F-blasts) in Bone Marrow
resolves10.1101/gad.1627208<i>Rb</i> intrinsically promotes erythropoiesis by coupling cell cycle exit with mitochondrial biogenesis
resolves10.1101/gad.1861209Transcriptional competence and the active marking of tissue-specific enhancers by defined transcription factors in embryonic and induced pluripotent stem cells
resolves10.1073/pnas.90.23.11381Germ-line transmission and developmental regulation of a 150-kb yeast artificial chromosome containing the human beta-globin locus in transgenic mice.
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