Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 50 checked references that resolve
resolves10.1038/nrn2915Goal-directed and habitual control in the basal ganglia: implications for Parkinson's disease
resolves10.1038/nature11466Dopaminergic neurons inhibit striatal output through non-canonical release of GABA
resolves10.7554/eLife.01936Midbrain dopamine neurons sustain inhibitory transmission using plasma membrane uptake of GABA, not synthesis
resolves10.1523/JNEUROSCI.4029-08.2009Single Nigrostriatal Dopaminergic Neurons Form Widely Spread and Highly Dense Axonal Arborizations in the Neostriatum
resolves10.1002/dvg.20405Deletion of GAD67 in dopamine receptor‐1 expressing cells causes specific motor deficits
resolves10.1016/j.plaphy.2007.05.007Higher accumulation of γ-aminobutyric acid induced by salt stress through stimulating the activity of diamine oxidases in Glycine max (L.) Merr. roots
resolves10.1016/j.plantsci.2012.06.001Hypothesis/review: Contribution of putrescine to 4-aminobutyrate (GABA) production in response to abiotic stress
resolves10.1038/nn.2777A neuroprotective role for polyamines in a Xenopus tadpole model of epilepsy
resolves10.1016/0091-3057(86)90017-1The effect of L-erythro-dihydroxyphenylserine injected into the lateral ventricle and the hypothalamus on the locomotor activity
resolves10.1016/S0736-5748(99)00002-7GABAergic system in the developing mammalian retina: dual sources of GABA at early stages of postnatal development
resolves10.1016/0306-4522(92)90345-3Characteristics of putrescine uptake and subsequent GABA formation in primary cultured astrocytes from normal C57BL/6J and epileptic DBA/2J mouse brain cortices
resolves10.1172/JCI72176Aldehyde dehydrogenase 1 defines and protects a nigrostriatal dopaminergic neuron subpopulation
resolves10.1073/pnas.91.16.7772High levels of a retinoic acid-generating dehydrogenase in the meso-telencephalic dopamine system.
resolves10.1111/jnc.12345Determinants of buildup of the toxic dopamine metabolite<scp>DOPAL</scp>in Parkinson's disease
resolves10.1128/MCB.23.13.4637-4648.2003Targeted Disruption of <i>Aldh1a1</i> (<i>Raldh1</i>) Provides Evidence for a Complex Mechanism of Retinoic Acid Synthesis in the Developing Retina
resolves10.1111/j.1530-0277.2009.00904.xAssociations and Interactions Between SNPs in the Alcohol Metabolizing Genes and Alcoholism Phenotypes in European Americans
resolves10.1111/j.1530-0277.2010.01346.xHaplotype-Based Study of the Association of Alcohol-Metabolizing Genes With Alcohol Dependence in Four Independent Populations
resolves10.1038/nn1579Neural systems of reinforcement for drug addiction: from actions to habits to compulsion
resolves10.1523/JNEUROSCI.2783-12.2012Ethanol-Mediated Facilitation of AMPA Receptor Function in the Dorsomedial Striatum: Implications for Alcohol Drinking Behavior
resolves10.1038/nn.3823Single rodent mesohabenular axons release glutamate and GABA
resolves10.1126/science.1250469GABA/glutamate co-release controls habenula output and is modified by antidepressant treatment
resolves10.1038/nn.3078A toolbox of Cre-dependent optogenetic transgenic mice for light-induced activation and silencing
resolves10.1152/jn.00476.2009Maintenance of Thalamic Epileptiform Activity Depends on the Astrocytic Glutamate-Glutamine Cycle
resolves10.1124/pr.111.005538Aldehyde Dehydrogenase Inhibitors: a Comprehensive Review of the Pharmacology, Mechanism of Action, Substrate Specificity, and Clinical Application
resolves10.1523/JNEUROSCI.3660-10.2010Fragile X Protein FMRP Is Required for Homeostatic Plasticity and Regulation of Synaptic Strength by Retinoic Acid
resolves10.1016/j.neuron.2011.12.036Distinct Neuronal Coding Schemes in Memory Revealed by Selective Erasure of Fast Synchronous Synaptic Transmission
resolves10.1038/sj.gt.3300938Recombinant adeno-associated virus purification using novel methods improves infectious titer and yield
resolves10.1038/2843Alcohol preference and sensitivity are markedly reduced in mice lacking dopamine D2 receptors
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