Reference health

Targeted complement inhibition salvages stressed neurons and inhibits neuroinflammation after stroke in mice

https://doi.org/10.1126/scitranslmed.aao6459
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38/38 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 38 checked references that resolve
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Complement in disease: a defence system turning offensive
resolves10.3389/fimmu.2015.00417
Complement in the Homeostatic and Ischemic Brain
resolves10.1111/imr.12470
Injury site‐specific targeting of complement inhibitors for treating stroke
resolves10.1186/1742-2094-9-137
Complement activation in the injured central nervous system: another dual-edged sword?
resolves10.1186/s12974-017-0894-6
Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complement
resolves10.4049/jimmunol.1102132
Pathogenic Natural Antibodies Propagate Cerebral Injury Following Ischemic Stroke in Mice
resolves10.1161/CIRCULATIONAHA.114.010482
Targeting Pathogenic Postischemic Self-Recognition by Natural IgM to Protect Against Posttransplantation Cardiac Reperfusion Injury
resolves10.1159/000108676
Induction of Early-Response Genes after Focal Ischemia
resolves10.1016/j.neuroscience.2008.06.038
Neurodegeneration and cellular stress in the retina and optic nerve in rat cerebral ischemia and hypoperfusion models
resolves10.1073/pnas.0400347101
Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury
resolves10.1038/sj.jcbfm.9600153
Reappearance of Hippocampal CA1 Neurons after Ischemia is Associated with Recovery of Learning and Memory
resolves10.1161/01.STR.30.12.2752
Recommendations for Standards Regarding Preclinical Neuroprotective and Restorative Drug Development
resolves10.1056/NEJMoa0804656
Thrombolysis with Alteplase 3 to 4.5 Hours after Acute Ischemic Stroke
resolves10.1186/s12974-015-0464-8
Modulation of post-stroke degenerative and regenerative processes and subacute protection by site-targeted inhibition of the alternative pathway of complement
resolves10.1161/01.STR.31.6.1223
Medical Complications After Stroke
resolves10.1161/01.STR.0000240409.68739.2b
Stroke Propagates Bacterial Aspiration to Pneumonia in a Model of Cerebral Ischemia
resolves10.1002/ana.24146
<i>Streptococcus pneumoniae</i> worsens cerebral ischemia via interleukin 1 and platelet glycoprotein Ibα
resolves10.1161/STROKEAHA.111.633255
Atrophy of Spared Gray Matter Tissue Predicts Poorer Motor Recovery and Rehabilitation Response in Chronic Stroke
resolves10.1038/nrn3710
Microglial phagocytosis of live neurons
resolves10.1073/pnas.1308679110
Phagocytosis executes delayed neuronal death after focal brain ischemia
resolves10.1523/JNEUROSCI.1498-12.2012
Galectin-3 Is Required for Resident Microglia Activation and Proliferation in Response to Ischemic Injury
resolves10.1038/nn.3599
Identification of a unique TGF-β–dependent molecular and functional signature in microglia
resolves10.1038/ncomms11499
Microglia protect against brain injury and their selective elimination dysregulates neuronal network activity after stroke
resolves10.1523/JNEUROSCI.1922-07.2007
CNS-Derived Interleukin-4 Is Essential for the Regulation of Autoimmune Inflammation and Induces a State of Alternative Activation in Microglial Cells
resolves10.1038/nrn2735
Plasticity during stroke recovery: from synapse to behaviour
resolves10.1146/annurev-neuro-061010-113810
The Complement System: An Unexpected Role in Synaptic Pruning During Development and Disease
resolves10.18632/oncotarget.4844
Complementing regeneration
resolves10.4049/jimmunol.142.2.582
The murine complement receptor gene family. Analysis of mCRY gene products and their homology to human CR1.
resolves10.1126/science.285.5427.595
Neuronal Protection in Stroke by an sLe <sup>x</sup> -Glycosylated Complement Inhibitory Protein
resolves10.3758/BF03193146
G*Power 3: A flexible statistical power analysis program for the social, behavioral, and biomedical sciences
resolves10.1172/JCI25208
Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection
resolves10.1038/jcbfm.1988.59
Quantitative Evaluation of Vascular Permeability in the Gerbil Brain after Transient Ischemia Using Evans Blue Fluorescence
resolves10.1523/JNEUROSCI.2197-17.2018
Identifying the Role of Complement in Triggering Neuroinflammation after Traumatic Brain Injury
resolves10.1523/JNEUROSCI.6195-10.2011
FACS Identifies Unique Cocaine-Induced Gene Regulation in Selectively Activated Adult Striatal Neurons
resolves10.1016/j.jneumeth.2011.08.045
FACS purification of immunolabeled cell types from adult rat brain
resolves10.1073/pnas.012669199
The classical pathway is the dominant complement pathway required for innate immunity to <i>Streptococcus pneumoniae</i> infection in mice
resolves10.1128/IAI.00963-09
Cigarette Smoke Exposure Impairs Pulmonary Bacterial Clearance and Alveolar Macrophage Complement-Mediated Phagocytosis of <i>Streptococcus pneumoniae</i>
resolves10.1038/nbt1385
Direct multiplexed measurement of gene expression with color-coded probe pairs
The 1 reference without a DOI — listed, not checked
no DOI — not checkedP. Rioux, TP-10 (AVANT immunotherapeutics). Curr. Opin. Investig. Drugs 2, 364–371 (2001).
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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