Reference health
PARPi Triggers the STING-Dependent Immune Response and Enhances the Therapeutic Efficacy of Immune Checkpoint Blockade Independent of BRCAness
24/24 checkable references clean · checked 2026-07-22
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 24 checked references that resolve
resolves10.1056/NEJMoa0900212
Inhibition of Poly(ADP-Ribose) Polymerase in Tumors from
<i>BRCA</i>
Mutation Carriers
resolves10.1056/NEJMoa1105535
Olaparib Maintenance Therapy in Platinum-Sensitive Relapsed Ovarian Cancer
resolves10.1038/nature03443
Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase
resolves10.1038/nature03445
Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy
resolves10.1016/S1470-2045(11)70214-5
Olaparib in patients with recurrent high-grade serous or poorly differentiated ovarian carcinoma or triple-negative breast cancer: a phase 2, multicentre, open-label, non-randomised study
resolves10.1056/NEJMoa1611310
Niraparib Maintenance Therapy in Platinum-Sensitive, Recurrent Ovarian Cancer
resolves10.1093/nar/gkn730
Cell Line Data Base: structure and recent improvements towards molecular authentication of human cell lines
resolves10.1186/s12915-016-0292-z
The Golgi apparatus acts as a platform for TBK1 activation after viral RNA sensing
resolves10.1038/nature12306
cGAS produces a 2′-5′-linked cyclic dinucleotide second messenger that activates STING
resolves10.1016/j.molonc.2012.12.007
BRCA1/2 mutation analysis in 41 ovarian cell lines reveals only one functionally deleterious BRCA1 mutation
resolves10.1093/jnci/djw199
Activation of STING-Dependent Innate Immune Signaling By S-Phase-Specific DNA Damage in Breast Cancer
resolves10.1158/0008-5472.CAN-11-4136
NF-κB Hyperactivation in Tumor Tissues Allows Tumor-Selective Reprogramming of the Chemokine Microenvironment to Enhance the Recruitment of Cytolytic T Effector Cells
resolves10.1038/nm1699
Endothelin B receptor mediates the endothelial barrier to T cell homing to tumors and disables immune therapy
resolves10.1038/nature23470
Mitotic progression following DNA damage enables pattern recognition within micronuclei
resolves10.1016/j.ccr.2012.02.022
Accessories to the Crime: Functions of Cells Recruited to the Tumor Microenvironment
resolves10.1016/j.ctrv.2017.11.007
CXCL9, CXCL10, CXCL11/CXCR3 axis for immune activation – A target for novel cancer therapy
resolves10.4049/jimmunol.1201018
Tumor-Infiltrating Monocytic Myeloid-Derived Suppressor Cells Mediate CCR5-Dependent Recruitment of Regulatory T Cells Favoring Tumor Growth
resolves10.1136/gutjnl-2016-312078
Myeloid cells are required for PD-1/PD-L1 checkpoint activation and the establishment of an immunosuppressive environment in pancreatic cancer
resolves10.3389/fimmu.2017.00086
Human Tumor-Infiltrating Myeloid Cells: Phenotypic and Functional Diversity
The 1 reference without a DOI — listed, not checked
no DOI — not checkedImmunosuppressive tumor-infiltrating myeloid cells mediate adaptive immune resistance via a PD-1/PD-L1 mechanism in glioblastoma
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