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Constitutive AP-1 Activity and EBV Infection Induce PD-L1 in Hodgkin Lymphomas and Posttransplant Lymphoproliferative Disorders: Implications for Targeted Therapy

https://doi.org/10.1158/1078-0432.ccr-11-1942
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48/48 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 48 checked references that resolve
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The biology of Hodgkin's lymphoma
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Epstein-Barr Virus (EBV) LMP2A induces alterations in gene transcription similar to those observed in Reed-Sternberg cells of Hodgkin lymphoma
resolves10.1002/path.2384
The Epstein–Barr virus oncoprotein, latent membrane protein‐1, reprograms germinal centre B cells towards a Hodgkin's Reed–Sternberg‐like phenotype
resolves10.1182/blood-2010-11-320481
Viral induction and targeted inhibition of galectin-1 in EBV+ posttransplant lymphoproliferative disorders
resolves10.1073/pnas.0706017104
The AP1-dependent secretion of galectin-1 by Reed–Sternberg cells fosters immune privilege in classical Hodgkin lymphoma
resolves10.1158/1078-0432.CCR-07-4709
AP1-Dependent Galectin-1 Expression Delineates Classical Hodgkin and Anaplastic Large Cell Lymphomas from Other Lymphoid Malignancies with Shared Molecular Features
resolves10.1182/blood-2007-05-085159
PD-1–PD-1 ligand interaction contributes to immunosuppressive microenvironment of Hodgkin lymphoma
resolves10.1182/blood-2010-05-282780
Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma
resolves10.1146/annurev.immunol.26.021607.090331
PD-1 and Its Ligands in Tolerance and Immunity
resolves10.1016/j.immuni.2007.05.016
Programmed Death-1 Ligand 1 Interacts Specifically with the B7-1 Costimulatory Molecule to Inhibit T Cell Responses
resolves10.1084/jem.20090847
PD-L1 regulates the development, maintenance, and function of induced regulatory T cells
resolves10.1038/nature04444
Restoring function in exhausted CD8 T cells during chronic viral infection
resolves10.1038/nature05115
PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression
resolves10.1128/JVI.02021-06
Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression
resolves10.1038/nm1482
Upregulation of PD-1 expression on HIV-specific CD8+ T cells leads to reversible immune dysfunction
resolves10.1053/j.gastro.2008.03.037
Dynamic Programmed Death 1 Expression by Virus-Specific CD8 T Cells Correlates With the Outcome of Acute Hepatitis B
resolves10.1073/pnas.192461099
Involvement of PD-L1 on tumor cells in the escape from host immune system and tumor immunotherapy by PD-L1 blockade
resolves10.1002/cncr.24899
Tumor cell expression of programmed cell death‐1 ligand 1 is a prognostic factor for malignant melanoma
resolves10.1007/s00432-008-0364-8
B7-H1 up-regulated expression in human pancreatic carcinoma tissue associates with tumor progression
resolves10.1158/1078-0432.CCR-08-1608
Overexpression of PD-L1 Significantly Associates with Tumor Aggressiveness and Postoperative Recurrence in Human Hepatocellular Carcinoma
resolves10.1016/j.canlet.2008.03.056
Clinical significance and regulation of the costimulatory molecule B7-H1 in pancreatic cancer
resolves10.1158/1078-0432.CCR-06-2746
Clinical Significance and Therapeutic Potential of the Programmed Death-1 Ligand/Programmed Death-1 Pathway in Human Pancreatic Cancer
resolves10.1073/pnas.0611533104
Programmed cell death 1 ligand 1 and tumor-infiltrating CD8 <sup>+</sup> T lymphocytes are prognostic factors of human ovarian cancer
resolves10.1053/hupa.2001.29679
Inflammatory pseudotumor of lymph node and spleen: An entity biologically distinct from inflammatory myofibroblastic tumor
resolves10.1007/978-1-61779-080-5_5
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resolves10.1093/nar/gki354
TOUCAN 2: the all-inclusive open source workbench for regulatory sequence analysis
resolves10.1038/nprot.2008.73
Analyzing real-time PCR data by the comparative CT method
resolves10.1128/jvi.55.3.710-720.1985
Epstein-Barr virus-encoded protein found in plasma membranes of transformed cells
resolves10.1084/jem.20080809
Origin and pathogenesis of nodular lymphocyte–predominant Hodgkin lymphoma as revealed by global gene expression analysis
resolves10.1038/leu.2008.272
PD-1/PD-L1 expression in human T-cell leukemia virus type 1 carriers and adult T-cell leukemia/lymphoma patients
resolves10.1097/QAD.0b013e3282f4de23
Upregulation of PD-L1 on monocytes and dendritic cells by HIV-1 derived TLR ligands
resolves10.1016/j.jhep.2006.05.007
PD-L1 is induced in hepatocytes by viral infection and by interferon-α and -γ and mediates T cell apoptosis
resolves10.1093/emboj/16.21.6478
Epstein–Barr virus latent membrane protein‐1 triggers AP‐1 activity via the c‐Jun N‐terminal kinase cascade
resolves10.1093/emboj/18.11.3064
Latent membrane protein 1 of Epstein–Barr virus interacts with JAK3 and activates STAT proteins
resolves10.1016/j.cell.2011.02.013
Hallmarks of Cancer: The Next Generation
resolves10.1038/nature09754
MHC class II transactivator CIITA is a recurrent gene fusion partner in lymphoid cancers
resolves10.1200/JCO.2010.32.8401
Molecular Pathogenesis of Hodgkin's Lymphoma: Increasing Evidence of the Importance of the Microenvironment
resolves10.1093/emboj/cdf389
Aberrantly expressed c‐Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF‐κB
resolves10.1371/journal.pone.0012926
Programmed Death-1 Expression on Epstein Barr Virus Specific CD8+ T Cells Varies by Stage of Infection, Epitope Specificity, and T-Cell Receptor Usage
resolves10.1097/01.TP.0000163288.98419.0D
Characteristic Pattern of Chromosomal Imbalances in Posttransplantation Lymphoproliferative Disorders: Correlation with Histopathological Subcategories and EBV Status
resolves10.1111/j.1365-2141.2006.06114.x
Comparative genome‐wide profiling of post‐transplant lymphoproliferative disorders and diffuse large B‐cell lymphomas
resolves10.1038/sj.onc.1209151
Mutations of the tumor suppressor gene SOCS-1 in classical Hodgkin lymphoma are frequent and associated with nuclear phospho-STAT5 accumulation
resolves10.1182/blood-2009-02-204842
Regulation of JAK2 by miR-135a: prognostic impact in classic Hodgkin lymphoma
resolves10.1182/blood-2010-06-291310
JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphoma
resolves10.1158/1078-0432.CCR-10-2660
Programmed Death Ligand 1 Is Expressed by Non–Hodgkin Lymphomas and Inhibits the Activity of Tumor-Associated T Cells
resolves10.1182/blood-2006-11-059451
NPM-ALK oncogenic kinase promotes cell-cycle progression through activation of JNK/cJun signaling in anaplastic large-cell lymphoma
resolves10.1038/modpathol.2010.130
Increase of programmed death-1-expressing intratumoral CD8 T cells predicts a poor prognosis for nasopharyngeal carcinoma
The 1 reference without a DOI — listed, not checked
no DOI — not checkedRole of the transcription factor AP-1 in melanoma differentiation
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