Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 48 checked references that resolve
resolves10.1182/blood-2003-04-1018Epstein-Barr Virus (EBV) LMP2A induces alterations in gene transcription similar to those observed in Reed-Sternberg cells of Hodgkin lymphoma
resolves10.1002/path.2384The Epstein–Barr virus oncoprotein, latent membrane protein‐1, reprograms germinal centre B cells towards a Hodgkin's Reed–Sternberg‐like phenotype
resolves10.1182/blood-2010-11-320481Viral induction and targeted inhibition of galectin-1 in EBV+ posttransplant lymphoproliferative disorders
resolves10.1073/pnas.0706017104The AP1-dependent secretion of galectin-1 by Reed–Sternberg cells fosters immune privilege in classical Hodgkin lymphoma
resolves10.1158/1078-0432.CCR-07-4709AP1-Dependent Galectin-1 Expression Delineates Classical Hodgkin and Anaplastic Large Cell Lymphomas from Other Lymphoid Malignancies with Shared Molecular Features
resolves10.1182/blood-2010-05-282780Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma
resolves10.1016/j.immuni.2007.05.016Programmed Death-1 Ligand 1 Interacts Specifically with the B7-1 Costimulatory Molecule to Inhibit T Cell Responses
resolves10.1084/jem.20090847PD-L1 regulates the development, maintenance, and function of induced regulatory T cells
resolves10.1038/nature04444Restoring function in exhausted CD8 T cells during chronic viral infection
resolves10.1038/nature05115PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression
resolves10.1128/JVI.02021-06Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression
resolves10.1038/nm1482Upregulation of PD-1 expression on HIV-specific CD8+ T cells leads to reversible immune dysfunction
resolves10.1053/j.gastro.2008.03.037Dynamic Programmed Death 1 Expression by Virus-Specific CD8 T Cells Correlates With the Outcome of Acute Hepatitis B
resolves10.1073/pnas.192461099Involvement of PD-L1 on tumor cells in the escape from host immune system and tumor immunotherapy by PD-L1 blockade
resolves10.1002/cncr.24899Tumor cell expression of programmed cell death‐1 ligand 1 is a prognostic factor for malignant melanoma
resolves10.1007/s00432-008-0364-8B7-H1 up-regulated expression in human pancreatic carcinoma tissue associates with tumor progression
resolves10.1158/1078-0432.CCR-08-1608Overexpression of PD-L1 Significantly Associates with Tumor Aggressiveness and Postoperative Recurrence in Human Hepatocellular Carcinoma
resolves10.1158/1078-0432.CCR-06-2746Clinical Significance and Therapeutic Potential of the Programmed Death-1 Ligand/Programmed Death-1 Pathway in Human Pancreatic Cancer
resolves10.1073/pnas.0611533104Programmed cell death 1 ligand 1 and tumor-infiltrating CD8
<sup>+</sup>
T lymphocytes are prognostic factors of human ovarian cancer
resolves10.1053/hupa.2001.29679Inflammatory pseudotumor of lymph node and spleen: An entity biologically distinct from inflammatory myofibroblastic tumor
resolves10.1093/nar/gki354TOUCAN 2: the all-inclusive open source workbench for regulatory sequence analysis
resolves10.1084/jem.20080809Origin and pathogenesis of nodular lymphocyte–predominant Hodgkin lymphoma as revealed by global gene expression analysis
resolves10.1038/leu.2008.272PD-1/PD-L1 expression in human T-cell leukemia virus type 1 carriers and adult T-cell leukemia/lymphoma patients
resolves10.1016/j.jhep.2006.05.007PD-L1 is induced in hepatocytes by viral infection and by interferon-α and -γ and mediates T cell apoptosis
resolves10.1093/emboj/16.21.6478Epstein–Barr virus latent membrane protein‐1 triggers AP‐1 activity via the c‐Jun N‐terminal kinase cascade
resolves10.1093/emboj/18.11.3064Latent membrane protein 1 of Epstein–Barr virus interacts with JAK3 and activates STAT proteins
resolves10.1038/nature09754MHC class II transactivator CIITA is a recurrent gene fusion partner in lymphoid cancers
resolves10.1200/JCO.2010.32.8401Molecular Pathogenesis of Hodgkin's Lymphoma: Increasing Evidence of the Importance of the Microenvironment
resolves10.1093/emboj/cdf389Aberrantly expressed c‐Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF‐κB
resolves10.1371/journal.pone.0012926Programmed Death-1 Expression on Epstein Barr Virus Specific CD8+ T Cells Varies by Stage of Infection, Epitope Specificity, and T-Cell Receptor Usage
resolves10.1097/01.TP.0000163288.98419.0DCharacteristic Pattern of Chromosomal Imbalances in Posttransplantation Lymphoproliferative Disorders: Correlation with Histopathological Subcategories and EBV Status
resolves10.1038/sj.onc.1209151Mutations of the tumor suppressor gene SOCS-1 in classical Hodgkin lymphoma are frequent and associated with nuclear phospho-STAT5 accumulation
resolves10.1182/blood-2010-06-291310JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphoma
resolves10.1158/1078-0432.CCR-10-2660Programmed Death Ligand 1 Is Expressed by Non–Hodgkin Lymphomas and Inhibits the Activity of Tumor-Associated T Cells
resolves10.1182/blood-2006-11-059451NPM-ALK oncogenic kinase promotes cell-cycle progression through activation of JNK/cJun signaling in anaplastic large-cell lymphoma
resolves10.1038/modpathol.2010.130Increase of programmed death-1-expressing intratumoral CD8 T cells predicts a poor prognosis for nasopharyngeal carcinoma
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