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B7-H3 Silencing Increases Paclitaxel Sensitivity by Abrogating Jak2/Stat3 Phosphorylation

https://doi.org/10.1158/1535-7163.mct-11-0072
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40/40 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 40 checked references that resolve
resolves10.4049/jimmunol.172.4.2352
Molecular Characterization of Human 4Ig-B7-H3, a Member of the B7 Family with Four Ig-Like Domains
resolves10.1038/85339
B7-H3: A costimulatory molecule for T cell activation and IFN-γ production
resolves10.1002/eji.200839028
B7‐H3 is a potent inhibitor of human T‐cell activation: No evidence for B7‐H3 and TREML2 interaction
resolves10.1038/ni967
The B7 family member B7-H3 preferentially down-regulates T helper type 1–mediated immune responses
resolves10.1073/pnas.1434299100
B7x: A widely expressed B7 family member that inhibits T cell activation
resolves10.1097/SLA.0b013e3181f1939d
B7–H3 Ligand Expression by Primary Breast Cancer and Associated With Regional Nodal Metastasis
resolves10.1158/1078-0432.CCR-08-0536
Tumor Cell and Tumor Vasculature Expression of B7-H3 Predict Survival in Clear Cell Renal Cell Carcinoma
resolves10.1158/1078-0432.CCR-08-0731
T-Cell Coregulatory Molecule Expression in Urothelial Cell Carcinoma: Clinicopathologic Correlations and Association with Survival
resolves10.1073/pnas.0709802104
B7-H3 and B7x are highly expressed in human prostate cancer and associated with disease spread and poor outcome
resolves10.1038/sj.bjc.6605375
Clinical importance of B7-H3 expression in human pancreatic cancer
resolves10.2174/156800908785133141
The Immunoregulatory Protein Human B7H3 is a Tumor-Associated Antigen that Regulates Tumor Cell Migration and Invasion
resolves10.1101/gad.828901
Human breast cancer cells generated by oncogenic transformation of primary mammary epithelial cells
resolves10.1007/978-0-387-74039-3_9
Molecular Mechanisms of ErbB2-Mediated Breast Cancer Chemoresistance
resolves10.1016/j.molmed.2006.11.001
The STAT3 oncogene as a predictive marker of drug resistance
resolves10.1038/cr.2008.18
STAT3 as a target for inducing apoptosis in solid and hematological tumors
resolves10.1038/sj.onc.1204990
Inhibition of constitutively active Stat3 suppresses growth of human ovarian and breast cancer cells
resolves10.1038/nrm909
STATs: transcriptional control and biological impact
resolves10.1073/pnas.0604042103
STAT3: A multifaceted oncogene
resolves10.1158/0008-5472.CAN-05-0317
Malignant Transformation but not Normal Cell Growth Depends on Signal Transducer and Activator of Transcription 3
resolves10.1016/j.canlet.2007.08.019
Inhibition of Stat3 increases doxorubicin sensitivity in a human metastatic breast cancer cell line
resolves10.1158/1078-0432.CCR-04-1752
Persistent Activation of Stat3 Signaling Induces Survivin Gene Expression and Confers Resistance to Apoptosis in Human Breast Cancer Cells
resolves10.1182/blood-2002-07-2130
Inhibition of STAT3 signaling induces apoptosis and decreases survivin expression in primary effusion lymphoma
resolves10.1016/S1074-7613(00)80011-4
Constitutive Activation of Stat3 Signaling Confers Resistance to Apoptosis in Human U266 Myeloma Cells
resolves10.1172/JCI9940
Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression
resolves10.1172/JCI3785
Requirement of Stat3 but not Stat1 activation for epidermal growth factor receptor- mediated cell growth In vitro.
resolves10.1073/pnas.97.8.4227
Constitutive activation of Stat3 signaling abrogates apoptosis in squamous cell carcinogenesis <i>in vivo</i>
resolves10.1038/sj.onc.1205859
Roles of activated Src and Stat3 signaling in melanoma tumor cell growth
resolves10.1038/sj.onc.1206479
Activation of Stat3 by receptor tyrosine kinases and cytokines regulates survival in human non-small cell carcinoma cells
resolves10.1200/JCO.2003.02.063
Improved Outcomes From Adding Sequential Paclitaxel but Not From Escalating Doxorubicin Dose in an Adjuvant Chemotherapy Regimen for Patients With Node-Positive Primary Breast Cancer
resolves10.1016/j.bbrc.2005.07.075
Evaluation of potential Stat3-regulated genes in human breast cancer
resolves10.1097/00001813-200507000-00002
Targeting Stat3 in cancer therapy
resolves10.1002/ijc.20371
Downregulation of the antiapoptotic MCL‐1 protein and apoptosis in MA‐11 breast cancer cells induced by an anti‐epidermal growth factor receptor–<i>Pseudomonas</i> exotoxin a immunotoxin
resolves10.1158/1541-7786.MCR-07-0080
Down-Regulation of Mcl-1 by Small Interfering RNA Sensitizes Resistant Melanoma Cells to Fas-Mediated Apoptosis
resolves10.1007/s00280-008-0697-7
Inducing apoptosis and enhancing chemosensitivity to Gemcitabine via RNA interference targeting Mcl-1 gene in pancreatic carcinoma cell
resolves10.1158/1078-0432.CCR-08-0954
Mitotic Deregulation by Survivin in ErbB2-Overexpressing Breast Cancer Cells Contributes to Taxol Resistance
resolves10.1016/j.ejca.2006.06.013
Predictive value of HER-2 and Topoisomerase IIα in response to primary doxorubicin in breast cancer
resolves10.4161/cbt.3.5.810
Estrogen Receptor Expression and Sensitivity to Paclitaxel in Breast Cancer
resolves10.1038/73432
Systematic variation in gene expression patterns in human cancer cell lines
resolves10.1158/0008-5472.CAN-3299-2
Lineage Infidelity of MDA-MB-435 Cells
resolves10.1158/0008-5472.CAN-09-1528
MDA-MB-435 and M14 Cell Lines: Identical but not M14 Melanoma?
The 2 references without a DOI — listed, not checked
no DOI — not checkedB7-H3 and B7-H4 expression in non-small-cell lung cancer
no DOI — not checkedConstitutive activation of Stat3 in human prostate tumors and cell lines: direct inhibition of Stat3 signaling induces apoptosis of prostate cancer cells
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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