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Genomic Complexity Profiling Reveals That HORMAD1 Overexpression Contributes to Homologous Recombination Deficiency in Triple-Negative Breast Cancers
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The 52 checked references that resolve
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Mutation in Brca2 stimulates error‐prone homology‐directed repair of DNA double‐strand breaks occurring between repeated sequences
resolves10.1093/jnci/djg050
Germline BRCA1 Mutations and a Basal Epithelial Phenotype in Breast Cancer
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Inhibition of Poly(ADP-Ribose) Polymerase in Tumors from
<i>BRCA</i>
Mutation Carriers
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Allele-specific copy number analysis of tumor samples with aneuploidy and tumor heterogeneity
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Patterns of genomic loss of heterozygosity predict homologous recombination repair defects in epithelial ovarian cancer
resolves10.1158/2159-8290.CD-11-0206
Telomeric Allelic Imbalance Indicates Defective DNA Repair and Sensitivity to DNA-Damaging Agents
resolves10.1158/0008-5472.CAN-12-1470
Ploidy and Large-Scale Genomic Instability Consistently Identify Basal-like Breast Carcinomas with
<i>BRCA1/2</i>
Inactivation
resolves10.1158/1078-0432.CCR-12-0857
Profiles of Genomic Instability in High-Grade Serous Ovarian Cancer Predict Treatment Outcome
resolves10.1093/annonc/mdq624
An aCGH classifier derived from BRCA1-mutated breast cancer and benefit of high-dose platinum-based chemotherapy in HER2-negative breast cancer patients
resolves10.1016/j.ygyno.2012.03.006
BRCA1/2 mutations and expression: Response to platinum chemotherapy in patients with advanced stage epithelial ovarian cancer
resolves10.1158/2159-8290.CD-12-0049
Loss of 53BP1 Causes PARP Inhibitor Resistance in
<i>Brca1</i>
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resolves10.1371/journal.pgen.1000702
Mouse HORMAD1 and HORMAD2, Two Conserved Meiotic Chromosomal Proteins, Are Depleted from Synapsed Chromosome Axes with the Help of TRIP13 AAA-ATPase
resolves10.1016/j.yexcr.2009.08.007
A novel mammalian HORMA domain-containing protein, HORMAD1, preferentially associates with unsynapsed meiotic chromosomes
resolves10.1038/ncb2213
Meiotic homologue alignment and its quality surveillance are controlled by mouse HORMAD1
resolves10.1101/gr.137570.112
Integrative analysis of genome-wide loss of heterozygosity and monoallelic expression at nucleotide resolution reveals disrupted pathways in triple-negative breast cancer
resolves10.1016/j.cell.2010.11.055
Massive Genomic Rearrangement Acquired in a Single Catastrophic Event during Cancer Development
resolves10.1128/MCB.23.2.733-743.2003
Extensive Loss of Heterozygosity Is Suppressed during Homologous Repair of Chromosomal Breaks
resolves10.1038/nature10983
The genomic and transcriptomic architecture of 2,000 breast tumours reveals novel subgroups
resolves10.1200/JCO.2014.57.0085
Phase II Study of Gemcitabine, Carboplatin, and Iniparib As Neoadjuvant Therapy for Triple-Negative and <i>BRCA1</i>/<i>2</i> Mutation–Associated Breast Cancer With Assessment of a Tumor-Based Measure of Genomic Instability: PrECOG 0105
resolves10.1186/1471-2164-13-619
Molecular characterisation of cell line models for triple-negative breast cancers
resolves10.3892/ol.2012.1093
Platinum-based chemotherapy in triple-negative breast cancer: A meta-analysis
resolves10.1186/1471-2164-14-643
Integrated genomic analysis of triple-negative breast cancers reveals novel microRNAs associated with clinical and molecular phenotypes and sheds light on the pathways they control
resolves10.1016/S0968-0004(98)01257-2
The HORMA domain: a common structural denominator in mitotic checkpoints, chromosome synapsis and DNA repair
resolves10.1093/mutage/geq052
Molecular mechanisms of micronucleus, nucleoplasmic bridge and nuclear bud formation in mammalian and human cells
resolves10.1371/journal.pgen.1004005
Down-Regulation of Rad51 Activity during Meiosis in Yeast Prevents Competition with Dmc1 for Repair of Double-Strand Breaks
resolves10.1534/genetics.111.130674
Genetic Evidence That Synaptonemal Complex Axial Elements Govern Recombination Pathway Choice in Mice
resolves10.1091/mbc.e05-05-0465
Partner Choice during Meiosis Is Regulated by Hop1-promoted Dimerization of Mek1
resolves10.1101/gad.13.20.2633
XRCC3 promotes homology-directed repair of DNA damage in mammalian cells
resolves10.1007/978-1-61779-998-3_27
I-SceI-Based Assays to Examine Distinct Repair Outcomes of Mammalian Chromosomal Double Strand Breaks
resolves10.1073/pnas.0806092105
High sensitivity of BRCA1-deficient mammary tumors to the PARP inhibitor AZD2281 alone and in combination with platinum drugs
resolves10.1038/nature03445
Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy
resolves10.1158/1078-0432.CCR-13-1391
BMN 673, a Novel and Highly Potent PARP1/2 Inhibitor for the Treatment of Human Cancers with DNA Repair Deficiency
resolves10.1200/JCO.2014.55.2984
Phase III Study of Iniparib Plus Gemcitabine and Carboplatin Versus Gemcitabine and Carboplatin in Patients With Metastatic Triple-Negative Breast Cancer
resolves10.1111/j.1365-2443.2012.01600.x
HORMAD1‐dependent checkpoint/surveillance mechanism eliminates asynaptic oocytes
resolves10.1095/biolreprod.112.106773
Mouse HORMAD1 Is a Meiosis I Checkpoint Protein That Modulates DNA Double-Strand Break Repair During Female Meiosis1
resolves10.1016/j.canlet.2012.07.001
Biological significance of HORMA domain containing protein 1 (HORMAD1) in epithelial ovarian carcinoma
resolves10.1016/j.molcel.2009.09.029
Regulation of Meiotic Recombination via Mek1-Mediated Rad54 Phosphorylation
resolves10.1016/S0960-9822(99)80142-0
Mouse Rad54 affects DNA conformation and DNA-damage-induced Rad51 foci formation
resolves10.1177/1947601913486344
Inactivating Mutations in GT198 in Familial and Early-Onset Breast and Ovarian Cancers
resolves10.1038/modpathol.2012.244
Comparison of basal-like triple-negative breast cancer defined by morphology, immunohistochemistry and transcriptional profiles
resolves10.1093/bioinformatics/btl117
Pvclust: an R package for assessing the uncertainty in hierarchical clustering
resolves10.1002/ijc.24017
The gene expression signature of genomic instability in breast cancer is an independent predictor of clinical outcome
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