Reference health
Targeting Pathogenic Postischemic Self-Recognition by Natural IgM to Protect Against Posttransplantation Cardiac Reperfusion Injury
32/32 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 32 checked references that resolve
resolves10.1084/jem.183.5.2343
Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.
resolves10.1073/pnas.0400347101
Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury
resolves10.4049/jimmunol.1102132
Pathogenic Natural Antibodies Propagate Cerebral Injury Following Ischemic Stroke in Mice
resolves10.4049/jimmunol.0803980
Pathogenic Natural Antibodies Recognizing Annexin IV Are Required to Develop Intestinal Ischemia-Reperfusion Injury
resolves10.1172/JCI25208
Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection
resolves10.4049/jimmunol.175.1.541
Mannose-Binding Lectin Is a Regulator of Inflammation That Accompanies Myocardial Ischemia and Reperfusion Injury
resolves10.4049/jimmunol.1001504
Targeted Complement Inhibitors Protect against Posttransplant Cardiac Ischemia and Reperfusion Injury and Reveal an Important Role for the Alternative Pathway of Complement Activation
resolves10.4049/jimmunol.160.9.4553
Blockade of Antibody-Induced Glomerulonephritis with Crry-Ig, a Soluble Murine Complement Inhibitor
resolves10.1016/j.expneurol.2006.01.033
Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome after traumatic brain injury
resolves10.1097/01.ASN.0000051597.27127.A1
Administration of a Soluble Recombinant Complement C3 Inhibitor Protects Against Renal Disease in MRL/lpr Mice
resolves10.1002/art.10591
Mechanisms of effects of complement inhibition in murine collagen‐induced arthritis
resolves10.1073/pnas.96.5.2250
Innate and acquired humoral immunities to influenza virus are mediated by distinct arms of the immune system
resolves10.1016/j.yjmcc.2006.02.006
The role of natural IgM in myocardial ischemia–reperfusion injury
resolves10.1093/cvr/cvq141
Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction
resolves10.4049/jimmunol.173.11.7055
Anti-Phospholipid Antibodies Restore Mesenteric Ischemia/Reperfusion-Induced Injury in Complement Receptor 2/Complement Receptor 1-Deficient Mice
resolves10.1084/jem.175.1.121
Distinct receptor and regulatory properties of recombinant mouse complement receptor 1 (CR1) and Crry, the two genetic homologues of human CR1.
resolves10.1016/j.jtcvs.2007.12.062
Pexelizumab in ischemic heart disease: A systematic review and meta-analysis on 15,196 patients
resolves10.1111/j.1600-6143.2011.03561.x
Anti-Complement Component C5 mAb Synergizes with CTLA4Ig to Inhibit Alloreactive T cells and Prolong Cardiac Allograft Survival in Mice
resolves10.1097/TP.0b013e31818a68e2
Posttransplant Ischemia-Reperfusion Injury In Transplanted Heart Is Prevented By A Minibody to the Fifth Component of Complement
resolves10.1111/j.1600-6143.2009.02723.x
Primed CD8+ T-Cell Responses to AllogeneicEndothelial Cells Are Controlled by Local Complement Activation
resolves10.1172/JCI61019
Antigen-presenting cell–derived complement modulates graft-versus-host disease
resolves10.1681/ASN.2005101116
Therapeutic Strategy with a Membrane-Localizing Complement Regulator to Increase the Number of Usable Donor Organs after Prolonged Cold Storage
resolves10.1161/circulationaha.112.000784
Donor Brain Death Exacerbates Complement-Dependent Ischemia/Reperfusion Injury in Transplanted Hearts
resolves10.1007/978-1-4614-4118-2_9
CR2-Mediated Targeting of Complement Inhibitors: Bench-to-Bedside Using a Novel Strategy for Site-Specific Complement Modulation
resolves10.1016/j.molimm.2006.06.015
MBL and C1q compete for interaction with human endothelial cells
resolves10.1002/j.1460-2075.1991.tb08025.x
Epstein Barr virus/complement C3d receptor is an interferon alpha receptor.
resolves10.1016/j.molimm.2006.07.003
Complement receptor 2, natural antibodies and innate immunity: Inter-relationships in B cell selection and activation
resolves10.1084/jem.170.6.1931
Mapping of the Epstein-Barr virus and C3dg binding sites to a common domain on complement receptor type 2.
The 1 reference without a DOI — listed, not checked
no DOI — not checkedSilverman GJ, Grönwall C, Vas J, Chen Y. Natural autoantibodies to apoptotic cell membranes regulate fundamental innate immune functions and suppress inflammation. Discov Med. 2009;8:151–156.
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