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CD82 Restrains Pathological Angiogenesis by Altering Lipid Raft Clustering and CD44 Trafficking in Endothelial Cells
42/42 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
The 42 checked references that resolve
resolves10.1016/j.ceb.2008.06.007
αvβ3 integrin and angiogenesis: a moody integrin in a changing environment
resolves10.1016/j.tcb.2009.06.004
Tetraspanin-enriched microdomains: a functional unit in cell plasma membranes
resolves10.1042/BJ20060259
Gangliosides play an important role in the organization of CD82-enriched microdomains
resolves10.1074/jbc.M611407200
Ganglioside GM2-Tetraspanin CD82 Complex Inhibits Met and Its Cross-talk with Integrins, Providing a Basis for Control of Cell Motility through Glycosynapse
resolves10.1096/fj.08-123414
CD82 endocytosis and cholesterol‐dependent reorganization of tetraspanin webs and lipid rafts
resolves10.1002/pros.20386
Characterization of an antibody that can detect the Kai1/CD82 murine metastasis suppressor
resolves10.1002/art.22450
Analysis of vascular gene expression in arthritic synovium by laser‐mediated microdissection
resolves10.1016/j.bbrc.2011.03.017
HIF-2 directly activates CD82 gene expression in endothelial cells
resolves10.1128/JVI.00790-13
Clustering and Mobility of HIV-1 Env at Viral Assembly Sites Predict Its Propensity To Induce Cell-Cell Fusion
resolves10.1093/nar/23.24.5080
A<i>cre</i>-transgenic mouse strain for the ubiquitous deletion of<i>loxP</i>-flanked gene segments including deletion in germ cells
resolves10.1158/0008-5472.CAN-07-2140
Hyaluronic Acid Synthase-1 Expression Regulates Bladder Cancer Growth, Invasion, and Angiogenesis through CD44
resolves10.1074/jbc.M102273200
Differential Involvement of the Hyaluronan (HA) Receptors CD44 and Receptor for HA-mediated Motility in Endothelial Cell Function and Angiogenesis
resolves10.1182/blood-2009-04-219204
A CD44v6 peptide reveals a role of CD44 in VEGFR-2 signaling and angiogenesis
resolves10.1083/jcb.201002119
Clathrin-independent carriers form a high capacity endocytic sorting system at the leading edge of migrating cells
resolves10.1074/jbc.M103481200
Hyaluronan Enters Keratinocytes by a Novel Endocytic Route for Catabolism
resolves10.1016/j.bpj.2009.05.039
On the Use of Ripley's K-Function and Its Derivatives to Analyze Domain Size
resolves10.1074/jbc.M111.320747
Cytoskeletal Modulation of Lipid Interactions Regulates Lck Kinase Activity
resolves10.1371/journal.pgen.1002575
Temporal Transcriptional Profiling of Somatic and Germ Cells Reveals Biased Lineage Priming of Sexual Fate in the Fetal Mouse Gonad
resolves10.1161/circresaha.111.240622
Laminin-Binding Integrins Induce Dll4 Expression and Notch Signaling in Endothelial Cells
resolves10.1038/nrc3023
CD44: can a cancer-initiating cell profit from an abundantly expressed molecule?
resolves10.1074/jbc.M006498200
CD44 Interaction with c-Src Kinase Promotes Cortactin-mediated Cytoskeleton Function and Hyaluronic Acid-dependent Ovarian Tumor Cell Migration
resolves10.1182/blood-2002-04-1237
Src and phosphatidylinositol 3–kinase mediate soluble E-selectin–induced angiogenesis
resolves10.1182/blood.V90.3.1150
CD44 Is Involved in Tumor Angiogenesis; an Activation Antigen on Human Endothelial Cells
resolves10.1002/(SICI)1097-0215(19960529)66:5<664::AID-IJC14>3.0.CO;2-4
Evidence of involvement of CD44 in endothelial cell proliferation, migration and angiogenesisin vitro
resolves10.1101/gad.14.2.163
Cell surface-localized matrix metalloproteinase-9 proteolytically activates TGF-β and promotes tumor invasion and angiogenesis
resolves10.1016/S1534-5807(02)00145-4
GPI-Anchored Proteins Are Delivered to Recycling Endosomes via a Distinct cdc42-Regulated, Clathrin-Independent Pinocytic Pathway
resolves10.1073/pnas.0804374105
Plasma membranes are poised for activation of raft phase coalescence at physiological temperature
resolves10.1074/jbc.M505630200
A Specific Microdomain (“Glycosynapse 3”) Controls Phenotypic Conversion and Reversion of Bladder Cancer Cells through GM3-mediated Interaction of α3β1 Integrin with CD9
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