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Blood Transcriptomics Predicts Progression of Pulmonary Fibrosis and Associated Natural Killer Cells

https://doi.org/10.1164/rccm.202008-3093oc
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37/37 checkable references clean · checked 2026-07-25

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 37 checked references that resolve
resolves10.1164/rccm.2009-040GL
An Official ATS/ERS/JRS/ALAT Statement: Idiopathic Pulmonary Fibrosis: Evidence-based Guidelines for Diagnosis and Management
resolves10.1164/rccm.201105-0840OC
Forced Vital Capacity in Patients with Idiopathic Pulmonary Fibrosis: Test Properties and Minimal Clinically Important Difference
resolves10.1056/NEJMp1500526
Forced Vital Capacity in Idiopathic Pulmonary Fibrosis — FDA Review of Pirfenidone and Nintedanib
resolves10.1164/rccm.201508-1546OC
Predictors of Mortality Poorly Predict Common Measures of Disease Progression in Idiopathic Pulmonary Fibrosis
resolves10.1126/scitranslmed.3005964
Peripheral Blood Mononuclear Cell Gene Expression Profiles Predict Poor Outcome in Idiopathic Pulmonary Fibrosis
resolves10.1016/S2213-2600(17)30349-1
Validation of a 52-gene risk profile for outcome prediction in patients with idiopathic pulmonary fibrosis: an international, multicentre, cohort study
resolves10.1371/journal.pone.0037708
The Peripheral Blood Transcriptome Identifies the Presence and Extent of Disease in Idiopathic Pulmonary Fibrosis
resolves10.1155/2016/4759040
Comparative Study of Circulating MMP-7, CCL18, KL-6, SP-A, and SP-D as Disease Markers of Idiopathic Pulmonary Fibrosis
resolves10.1016/S2213-2600(18)30185-1
Prognostic and predictive biomarkers for patients with idiopathic pulmonary fibrosis treated with pirfenidone: post-hoc assessment of the CAPACITY and ASCEND trials
resolves10.1152/ajplung.00139.2012
Periostin promotes fibrosis and predicts progression in patients with idiopathic pulmonary fibrosis
resolves10.1164/ajrccm.165.2.ats01
American Thoracic Society/European Respiratory Society International Multidisciplinary Consensus Classification of the Idiopathic Interstitial Pneumonias
resolves10.3155/1047-3289.59.9.1032
Estimating Precision Using Duplicate Measurements
resolves10.1002/sim.1335
Power and sample size for DNA microarray studies
resolves10.1111/j.2517-6161.1995.tb02031.x
Controlling the False Discovery Rate: A Practical and Powerful Approach to Multiple Testing
resolves10.18637/jss.v033.i01
Regularization Paths for Generalized Linear Models via Coordinate Descent
resolves10.18637/jss.v039.i05
Regularization Paths for Cox's Proportional Hazards Model via Coordinate Descent
resolves10.1111/j.1467-9868.2011.01004.x
Strong Rules for Discarding Predictors in Lasso-Type Problems
resolves10.1186/1471-2105-12-77
pROC: an open-source package for R and S+ to analyze and compare ROC curves
resolves10.18637/jss.v061.i08
<b>OptimalCutpoints</b>: An<i>R</i>Package for Selecting Optimal Cutpoints in Diagnostic Tests
resolves10.1164/rccm.201202-0314OC
A Placebo-Controlled Randomized Trial of Warfarin in Idiopathic Pulmonary Fibrosis
resolves10.1056/NEJMoa1113354
Prednisone, Azathioprine, and <i>N</i> -Acetylcysteine for Pulmonary Fibrosis
resolves10.1056/NEJMoa1002110
A Controlled Trial of Sildenafil in Advanced Idiopathic Pulmonary Fibrosis
resolves10.1056/NEJMoa1402582
A Phase 3 Trial of Pirfenidone in Patients with Idiopathic Pulmonary Fibrosis
resolves10.1056/NEJMoa1402584
Efficacy and Safety of Nintedanib in Idiopathic Pulmonary Fibrosis
resolves10.1164/ajrccm.185.12.1329
Plasma Proteins for Risk Prediction in Idiopathic Pulmonary Fibrosis
resolves10.1164/rccm.201804-0761OC
Prognosticating Outcomes in Interstitial Lung Disease by Mediastinal Lymph Node Assessment. An Observational Cohort Study with Independent Validation
resolves10.1097/01.LAB.0000032380.82232.67
Abnormal Re-epithelialization and Lung Remodeling in Idiopathic Pulmonary Fibrosis: The Role of ΔN-p63
resolves10.3389/fimmu.2016.00591
The Role of Lymphocytes in Radiotherapy-Induced Adverse Late Effects in the Lung
resolves10.1016/S2213-2600(17)30430-7
An epithelial biomarker signature for idiopathic pulmonary fibrosis: an analysis from the multicentre PROFILE cohort study
resolves10.3389/fimmu.2018.01869
Natural Killer Cells: Development, Maturation, and Clinical Utilization
resolves10.1016/j.cell.2020.01.022
Tissue Determinants of Human NK Cell Development, Function, and Residence
resolves10.1172/JCI16861
Regulation of pulmonary fibrosis by chemokine receptor CXCR3
resolves10.1016/j.cyto.2013.12.003
Peripheral depletion of NK cells and imbalance of the Treg/Th17 axis in idiopathic pulmonary fibrosis patients
resolves10.1007/s00439-009-0666-1
MICA polymorphisms and decreased expression of the MICA receptor NKG2D contribute to idiopathic pulmonary fibrosis susceptibility
resolves10.1513/AnnalsATS.201502-088AW
Viruses in Idiopathic Pulmonary Fibrosis. Etiology and Exacerbation
resolves10.1164/rccm.201607-1525OC
Microbes Are Associated with Host Innate Immune Response in Idiopathic Pulmonary Fibrosis
resolves10.3389/fimmu.2017.00301
Immunoregulatory Role of NK Cells in Tissue Inflammation and Regeneration
The 2 references without a DOI — listed, not checked
no DOI — not checkedShort-term longitudinal gene expression changes predict forced vital capacity decline in idiopathic pulmonary fibrosis [abstract]
no DOI — not checkedLinear models and empirical bayes methods for assessing differential expression in microarray experiments
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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