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Placental Growth Factor Contributes to Bronchial Neutrophilic Inflammation and Edema in Allergic Asthma

https://doi.org/10.1165/rcmb.2011-0152oc
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45/45 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

4 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 45 checked references that resolve
resolves10.1038/nm1105
Vascular endothelial growth factor (VEGF) induces remodeling and enhances TH2-mediated sensitization and inflammation in the lung
resolves10.1067/mai.2001.115628
Vascular endothelial growth factor in patients with acute asthma
resolves10.1046/j.1365-2222.2003.01576.x
Increased levels of vascular endothelial growth factor in induced sputum in asthmatic patients
resolves10.1067/mai.2001.115626
Gene expression of vascular endothelial growth factor and its receptors and angiogenesis in bronchial asthma
resolves10.1136/thx.2004.023986
Enhanced bronchial expression of vascular endothelial growth factor and receptors (Flk-1 and Flt-1) in patients with chronic obstructive pulmonary disease
resolves10.1182/blood-2002-05-1516
A critical role of placental growth factor in the induction of inflammation and edema formation
resolves10.1136/thx.2007.087155
Increased expression of placenta growth factor in COPD
resolves10.1073/pnas.88.20.9267
Isolation of a human placenta cDNA coding for a protein related to the vascular permeability factor.
resolves10.3109/08977199709002113
Placenta Growth Factor and Vascular Endothelial Growth Factor are Co-Expressed During Early Embryonic Development
resolves10.1046/j.1538-7836.2003.00263.x
Placental growth factor and its receptor, vascular endothelial growth factor receptor-1: novel targets for stimulation of ischemic tissue revascularization and inhibition of angiogenic and inflammatory disorders
resolves10.1074/jbc.274.49.35172
Placenta Growth Factor and Vascular Endothelial Growth Factor B and C Expression in Microvascular Endothelial Cells and Pericytes
resolves10.1038/nm731
Revascularization of ischemic tissues by PlGF treatment, and inhibition of tumor angiogenesis, arthritis and atherosclerosis by anti-Flt1
resolves10.1046/j.1523-1747.2000.00085.x
Placenta Growth Factor is Induced in Human Keratinocytes during Wound Healing
resolves10.1007/s00125-006-0539-2
Placental growth factor-1 and epithelial haemato–retinal barrier breakdown: potential implication in the pathogenesis of diabetic retinopathy
resolves10.1152/ajplung.00075.2006
Cyclic stretch induces PlGF expression in bronchial airway epithelial cells via nitric oxide release
resolves10.1073/pnas.90.22.10705
Inhibition of vascular endothelial cell growth factor activity by an endogenously encoded soluble receptor.
resolves10.1016/S1071-55760300048-0
Expression and Function of Placenta Growth Factor: Implications for Abnormal Placentation
resolves10.1152/ajpheart.00922.2007
Placental growth factor is a potent vasodilator of rat and human resistance arteries
resolves10.1074/jbc.271.30.17629
The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological Activities
resolves10.1182/blood-2002-11-3423
Mechanism of monocyte activation and expression of proinflammatory cytochemokines by placenta growth factor
resolves10.1038/labinvest.3700053
Vascular endothelial growth factor VEGF189 induces human neutrophil chemotaxis in extravascular tissue via an autocrine amplification mechanism
resolves10.1165/rcmb.2003-0314OC
Expression and Function of the Vascular Endothelial Growth Factor Receptor FLT-1 in Human Eosinophils
resolves10.1002/jcp.1121
Expression of vascular endothelial growth factor receptors in smooth muscle cells
resolves10.1074/jbc.272.38.23659
Differential Transcriptional Regulation of the Two Vascular Endothelial Growth Factor Receptor Genes
resolves10.1038/87904
Synergism between vascular endothelial growth factor and placental growth factor contributes to angiogenesis and plasma extravasation in pathological conditions
resolves10.1016/S0006-291X(02)00677-0
Loss of placental growth factor protects mice against vascular permeability in pathological conditions
resolves10.1016/j.ejca.2005.07.022
Placenta growth factor is over-expressed and has prognostic value in human breast cancer
resolves10.1016/j.cell.2007.08.038
Anti-PlGF Inhibits Growth of VEGF(R)-Inhibitor-Resistant Tumors without Affecting Healthy Vessels
resolves10.1186/1465-9921-7-135
IL-17 mRNA in sputum of asthmatic patients: linking T cell driven inflammation and granulocytic influx?
resolves10.1006/cyto.1998.0426
QUANTIFICATION OF MURINE CYTOKINE mRNAs USING REAL TIME QUANTITATIVE REVERSE TRANSCRIPTASE PCR
resolves10.1046/j.1365-2222.2001.01081.x
Eosinophilic rhinitis accompanies the development of lower airway inflammation and hyper‐reactivity in sensitized mice exposed to aerosolized allergen
resolves10.1111/j.1365-2222.2006.02527.x
Aggravation of bronchial eosinophilia in mice by nasal and bronchial exposure to <i>Staphylococcus aureus</i> enterotoxin B
resolves10.1165/rcmb.2009-0425OC
Selective Nasal Allergen Provocation Induces Substance P–Mediated Bronchial Hyperresponsiveness
resolves10.1152/ajplung.00157.2005
Immunological determinants of ventilatory changes induced in mice by dermal sensitization and respiratory challenge with toluene diisocyanate
resolves10.1002/(SICI)1521-4141(199804)28:04<1262::AID-IMMU1262>3.0.CO;2-G
Natural truncation of RANTES abolishes signaling through the CC chemokine receptors CCR1 and CCR3, impairs its chemotactic potency and generates a CC chemokine inhibitor
resolves10.1111/j.1398-9995.2009.02313.x
<i>Staphylococcus aureus</i> enterotoxin B augments granulocyte migration and survival via airway epithelial cell activation
resolves10.1165/rcmb.4832
Interleukin-17 Orchestrates the Granulocyte Influx into Airways after Allergen Inhalation in a Mouse Model of Allergic Asthma
resolves10.1016/j.imlet.2004.01.014
Interleukin-17 augments tumor necrosis factor-α-induced elaboration of proangiogenic factors from fibroblasts
resolves10.1111/j.1398-9995.2010.02369.x
A viral PAMP double‐stranded RNA induces allergen‐specific Th17 cell response in the airways which is dependent on VEGF and IL‐6
resolves10.1159/000049511
Role of Interleukin-17 and the Neutrophil in Asthma
resolves10.1016/S0014-2999(03)01341-4
Pharmacological modulation of interleukin-17-induced GCP-2-, GRO-α- and interleukin-8 release in human bronchial epithelial cells
resolves10.4049/jimmunol.181.9.6117
IL-17-Producing Alveolar Macrophages Mediate Allergic Lung Inflammation Related to Asthma
resolves10.4049/jimmunol.170.4.2106
IL-17, Produced by Lymphocytes and Neutrophils, Is Necessary for Lipopolysaccharide-Induced Airway Neutrophilia: IL-15 as a Possible Trigger
resolves10.1084/jem.20101376
A novel subset of CD4+ TH2 memory/effector cells that produce inflammatory IL-17 cytokine and promote the exacerbation of chronic allergic asthma
resolves10.4049/jimmunol.1001713
Distinct Roles of Vascular Endothelial Growth Factor Receptor-1– and Receptor-2–Mediated Signaling in T Cell Priming and Th17 Polarization to Lipopolysaccharide-Containing Allergens in the Lung
The 4 references without a DOI — listed, not checked
no DOI — not checkedSerum levels of VEGF and bFGF in hypoxic patients with exacerbated COPD
no DOI — not checkedPlacenta growth factor gene expression is induced by hypoxia in fibroblasts: a central role for metal transcription factor–1
no DOI — not checkedFlt-1 but not KDR/Flk-1 tyrosine kinase is a receptor for placenta growth factor, which is related to vascular endothelial growth factor
no DOI — not checkedMarkedly increased amounts of messenger RNAs for vascular endothelial growth factor and placenta growth factor in renal cell carcinoma associated with angiogenesis
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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