Reference health
Neurodegeneration in Multiple Sclerosis: Relationship to Neurological Disability
56/56 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
6 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 56 checked references that resolve
resolves10.1002/ana.410360710
Nervous system-immune system interactions and their role in multiple sclerosis
resolves10.1002/ana.410360735
Central nervous system remyelination: Studies in chronically damaged tissue
resolves10.1093/brain/59.4.445
OBSERVATIONS ON THE HISTOPATHOLOGY OF THE CEREBRAL LESIONS IN DISSEMINATED SCLEROSIS
resolves10.1073/pnas.87.4.1561
Precursor of amyloid protein in Alzheimer disease undergoes fast anterograde axonal transport.
resolves10.1172/JCI115102
Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions.
resolves10.1002/ana.410360515
Induction of nitric oxide synthase in demyelinating regions of multiple sclerosis brains
resolves10.1093/brain/120.5.865
Biotechnological agents for the immunotherapy of multiple sclerosis. Principles, problems and perspectives
resolves10.1002/ana.410330402
Acute motor axonal neuropathy: A frequent cause of acute flaccid paralysis in China
resolves10.1007/BF02284784
Early nodal changes in the acute motor axonal neuropathy pattern of the Guillain-Barré syndrome
resolves10.1523/JNEUROSCI.16-16-05095.1996
Oligodendroglia Regulate the Regional Expansion of Axon Caliber and Local Accumulation of Neurofilaments during Development Independently of Myelin Formation
resolves10.1523/JNEUROSCI.14-11-06392.1994
Regional modulation of neurofilament organization by myelination in normal axons
resolves10.1016/0092-8674(92)90183-D
Local modulation of neurofilament phosphorylation, axonal caliber, and slow axonal transport by myelinating Schwann cells
resolves10.1016/0092-8674(92)90591-Y
Mouse P0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons
resolves10.1523/JNEUROSCI.17-12-04545.1997
Structural Abnormalities and Deficient Maintenance of Peripheral Nerve Myelin in Mice Lacking the Gap Junction Protein Connexin 32
resolves10.1523/JNEUROSCI.18-06-01953.1998
Myelin-Associated Glycoprotein Is a Myelin Signal that Modulates the Caliber of Myelinated Axons
resolves10.1126/science.280.5369.1610
Axonal Swellings and Degeneration in Mice Lacking the Major Proteolipid of Myelin
resolves10.1083/jcb.104.4.957
The amino acid sequences of the myelin-associated glycoproteins: homology to the immunoglobulin gene superfamily
resolves10.1073/pnas.84.2.600
Molecular cloning and primary structure of myelin-associated glycoprotein.
resolves10.1073/pnas.84.12.4337
Two forms of 1B236/myelin-associated glycoprotein, a cell adhesion molecule for postnatal neural development, are produced by alternative splicing.
resolves10.1073/pnas.76.3.1510
Myelin-associated glycoprotein demonstrated immunocytochemically in myelin and myelin-forming cells of developing rat.
resolves10.1083/jcb.109.5.2417
The myelin-associated glycoprotein is enriched in multivesicular bodies and periaxonal membranes of actively myelinating oligodendrocytes.
resolves10.1016/0896-6273(94)90472-3
Mice deficient for the glycoprotein show subtle abnormalities in myelin
resolves10.1016/0896-6273(94)90247-X
Identification of myelin-associated glycoprotein as a major myelin-derived inhibitor of neurite growth
resolves10.1016/0896-6273(94)90042-6
A novel role for myelin-associated glycoprotein as an inhibitor of axonal regeneration
resolves10.1002/(SICI)1097-4547(19961115)46:4<404::AID-JNR2>3.0.CO;2-K
Myelin-associated glycoprotein inhibits neurite/axon growth and causes growth cone collapse
resolves10.1111/j.1750-3639.1995.tb00603.x
Neuropathology and Genetics of Pelizaeus‐Merzbacher Disease
resolves10.1111/j.1750-3639.1995.tb00604.x
Transgenic and Natural Mouse Models of Proteolipid Protein (PLP)‐Related Dysmyelination and Demyelination
resolves10.1073/pnas.84.17.6287
Abnormal compact myelin in the myelin-deficient rat: absence of proteolipid protein correlates with a defect in the intraperiod line.
resolves10.1016/0896-6273(93)90148-K
A proteolipid protein gene family: Expression in sharks and rays and possible evolution from an ancestral gene encoding a pore-forming polypeptide
resolves10.1002/ana.410320609
Using gadolinium‐enhanced magnetic resonance imaging lesions to monitor disease activity in multiple sclerosis
resolves10.1177/135245859600200406
MRI studies of multiple sclerosis: Implications for the natural history of the disease and for monitoring effectiveness of experimental therapies
resolves10.1002/ana.410430114
Magnetic resonance studies of intramuscular interferon β–1a for relapsing multiple sclerosis
resolves10.1097/00001756-199103000-00005
Immunohistochemical localization of N-acetylaspartate in rat brain
resolves10.1016/0306-4522(91)90101-S
Immunocytochemical localization of N-acetyl-aspartate with monoclonal antibodies
resolves10.1002/ana.410360115
Use of proton magnetic resonance spectroscopy for monitoring disease progression in multiple sclerosis
resolves10.1002/ana.410410314
Imaging of axonal damage in multiple sclerosis: Spatial distribution of magnetic resonance imaging lesions
resolves10.1212/WNL.49.4.1138
Axonal dysfunction and disability in a relapse of multiple sclerosis: Longitudinal study of a patient
resolves10.1093/brain/119.6.2009
Progressive cerebral atrophy in multiple sclerosis A serial MRI study
resolves10.1093/brain/119.3.715
Assessment of lesion pathology in multiple sclerosis using quantitative MRI morphometry and magnetic resonance spectroscopy
resolves10.1093/brain/117.1.49
Serial proton magnetic resonance spectroscopy in acute multiple sclerosis lesions
resolves10.1002/mrm.1910340511
Reversible decreases in <i>N</i>‐acetylaspartate after acute brain injury
resolves10.1002/ana.410380610
Chemical pathology of acute demyelinating lesions and its correlation with disability
resolves10.1093/brain/121.1.103
Imaging axonal damage of normal-appearing white matter in multiple sclerosis
resolves10.1093/brain/118.6.1583
Persistent functional deficit in multiple sclerosis and autosomal dominant cerebellar ataxia is associated with axon loss
resolves10.1002/mus.880100603
Recent views on amyotrophic lateral sclerosis with emphasis on electrophysiological studies
The 6 references without a DOI — listed, not checked
no DOI — not checkedatypb3
no DOI — not checkedatypb35
no DOI — not checkedatypb54
no DOI — not checkedatypb56
no DOI — not checkedatypb59
no DOI — not checkedatypb60
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work were clean at the dated check: each resolved to a known work in a public registry, and none
carried a retraction notice at that time. It says nothing about the quality, findings,
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