Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 40 checked references that resolve
resolves10.1038/356063a0Prevention of experimental autoimmune encephalomyelitis by antibodies against α4βl integrin
resolves10.1002/ana.20858Immune surveillance in multiple sclerosis patients treated with natalizumab
resolves10.1002/eji.201142108Natalizumab treatment perturbs memory‐ and marginal zone‐like B‐cell homing in secondary lymphoid organs in multiple sclerosis
resolves10.1038/bmt.2009.381CD49d blockade by natalizumab in patients with multiple sclerosis affects steady-state hematopoiesis and mobilizes progenitors with a distinct phenotype and function
resolves10.1093/cid/cit376Central Nervous System Herpes Simplex and Varicella Zoster Virus Infections in Natalizumab-Treated Patients
resolves10.1177/1352458514538111Effects of natalizumab on oligoclonal bands in the cerebrospinal fluid of multiple sclerosis patients: A longitudinal study
resolves10.1177/1352458512463483Cerebrospinal fluid parameters of B cell-related activity in patients with active disease during natalizumab therapy
resolves10.1002/ana.24153Cerebrospinal fluid JC virus antibody index for diagnosis of natalizumab‐associated progressive multifocal leukoencephalopathy
resolves10.1136/jnnp.57.8.897Cerebrospinal fluid in the diagnosis of multiple sclerosis: a consensus report.
resolves10.1136/mp.48.2.M93Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils
resolves10.4049/jimmunol.152.8.3760Adhesion through the LFA-1 (CD11a/CD18)-ICAM-1 (CD54) and the VLA-4 (CD49d)-VCAM-1 (CD106) pathways prevents apoptosis of germinal center B cells.
resolves10.1111/j.1750-3639.2004.tb00049.xDetection of Ectopic B‐cell Follicles with Germinal Centers in the Meninges of Patients with Secondary Progressive Multiple Sclerosis
resolves10.1016/j.jneuroim.2011.03.001Natalizumab therapy decreases surface expression of both VLA-heterodimer subunits on peripheral blood mononuclear cells
resolves10.1016/j.jneuroim.2007.11.007Natalizumab alters transcriptional expression profiles of blood cell subpopulations of multiple sclerosis patients
resolves10.1128/JVI.03221-13Lymphocyte Gene Expression and JC Virus Noncoding Control Region Sequences Are Linked with the Risk of Progressive Multifocal Leukoencephalopathy
resolves10.1016/j.jneuroim.2008.05.007Response to Linberg et al. Natalizumab alters transcriptional expression profiles of blood cell subpopulations of multiple sclerosis patients
resolves10.1177/1352458510385834CD34+ progenitor cells mobilized by natalizumab are not a relevant reservoir for JC virus
resolves10.1001/jamaneurol.2014.63JC Virus in CD34<sup>+</sup>and CD19<sup>+</sup>Cells in Patients With Multiple Sclerosis Treated With Natalizumab
resolves10.1002/ana.24148JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis
resolves10.1093/infdis/jir256Sequencing and Analysis of JC Virus DNA From Natalizumab-Treated PML Patients
resolves10.1093/infdis/jir198Progressive Multifocal Leukoencephalopathy (PML) Development Is Associated With Mutations in JC Virus Capsid Protein VP1 That Change Its Receptor Specificity
resolves10.1111/j.1750-3639.2011.00538.xFrequency and Large T (LT) Sequence of JC Polyomavirus DNA in Oligodendrocytes, Astrocytes and Granular Cells in Non‐PML Brain
resolves10.1212/WNL.82.10_supplement.S31.004B Cell-Specific Signal In Whole Blood And Serum Of Natalizumab-Treated PML Patients Is Revealed Using Transcript And Protein Profiling (S31.004)
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