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Upregulation of miR-205 induces CHN1 expression, which is associated with the aggressive behaviour of cervical cancer cells and correlated with lymph node metastasis

https://doi.org/10.1186/s12885-020-07478-w

40/40 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 40 checked references that resolve

resolves10.3322/caac.21551

Cancer statistics, 2019

resolves10.1097/GCO.0000000000000241

Global strategies for cervical cancer prevention

resolves10.1016/S0092-8674(04)00045-5

MicroRNAs

resolves10.1016/j.cell.2004.12.035

Conserved Seed Pairing, Often Flanked by Adenosines, Indicates that Thousands of Human Genes are MicroRNA Targets

resolves10.7314/APJCP.2014.15.2.577

Insights into the Diverse Roles of miR-205 in Human Cancers

resolves10.1007/s12032-013-0751-0

MicroRNA-361-5p facilitates cervical cancer progression through mediation of epithelial-to-mesenchymal transition

resolves10.1002/jcb.24726

MicroRNA‐590 Promotes Cervical Cancer Cell Growth and Invasion by Targeting CHL1

resolves10.25011/cim.v37i3.21380

MicroRNA-222 promotes the proliferation and migration of cervical cancer cells

resolves10.1038/bjc.2013.829

MicroRNA-196a promotes cervical cancer proliferation through the regulation of FOXO1 and p27Kip1

resolves10.1016/j.canlet.2012.05.022

MicroRNA-10a targets CHL1 and promotes cell growth, migration and invasion in human cervical cancer cells

resolves10.1371/journal.pone.0046990

miR-205 Expression Promotes Cell Proliferation and Migration of Human Cervical Cancer Cells

resolves10.1177/1010428317701308

MiR-205 serves as a prognostic factor and suppresses proliferation and invasion by targeting insulin-like growth factor receptor 1 in human cervical cancer

resolves10.7150/ijms.8880

MicroRNA-218 Enhances the Radiosensitivity of Human Cervical Cancer via Promoting Radiation Induced Apoptosis

resolves10.3892/mmr.2014.2335

MicroRNA-26a inhibits cell proliferation and invasion of cervical cancer cells by targeting protein tyrosine phosphatase type IVA 1

resolves10.1074/jbc.M111.337642

MicroRNA-214 Suppresses Growth and Invasiveness of Cervical Cancer Cells by Targeting UDP-N-acetyl-α-d-galactosamine:Polypeptide N-Acetylgalactosaminyltransferase 7

resolves10.3892/ol.2013.1404

MicroRNA-92 regulates cervical tumorigenesis and its expression is upregulated by human papillomavirus-16 E6 in cervical cancer cells

resolves10.1126/scisignal.2005825

Dysregulation of microRNA biogenesis and gene silencing in cancer

resolves10.1177/1470320316663327

miR-205 mediates the inhibition of cervical cancer cell proliferation using olmesartan

resolves10.1038/cr.2009.18

Suppression of cell growth and invasion by miR-205 in breast cancer

resolves10.1158/0008-5472.CAN-08-2894

miR-205 Exerts Tumor-Suppressive Functions in Human Prostate through Down-regulation of Protein Kinase Cε

resolves10.1002/cncr.25488

MicroRNA‐205–directed transcriptional activation of tumor suppressor genes in prostate cancer

resolves10.1073/pnas.0803992105

MicroRNA-184 antagonizes microRNA-205 to maintain SHIP2 levels in epithelia

resolves10.1007/s10059-013-2154-7

MicroRNA-205 Directly Regulates the Tumor Suppressor, Interleukin-24, in Human KB Oral Cancer Cells

resolves10.1186/1741-7007-8-58

Ultra-high throughput sequencing-based small RNA discovery and discrete statistical biomarker analysis in a collection of cervical tumours and matched controls

resolves10.1186/s12935-014-0081-0

Serum microRNA-205 as a novel biomarker for cervical cancer patients

resolves10.1042/BJ20061750

Chimaerins: GAPs that bridge diacylglycerol signalling and the small G-protein Rac

resolves10.1128/MCB.16.9.5069

The GTPase-Activating Protein <i>n</i> -Chimaerin Cooperates with Rac1 and Cdc42Hs To Induce the Formation of Lamellipodia and Filopodia

resolves10.1016/j.cell.2008.09.043

Rac Activation and Inactivation Control Plasticity of Tumor Cell Movement

resolves10.1038/nrc1299

A census of human cancer genes

resolves10.1074/jbc.M111.227611

miRNA-205 Suppresses Melanoma Cell Proliferation and Induces Senescence via Regulation of E2F1 Protein

resolves10.1016/j.jtcvs.2007.08.055

MicroRNA expression profiles of esophageal cancer

resolves10.1186/1479-5876-9-30

MiRNA-205 modulates cellular invasion and migration via regulating zinc finger E-box binding homeobox 2 expression in esophageal squamous cell carcinoma cells

resolves10.1002/ijc.24071

Dysregulated microRNAs and their predicted targets associated with endometrioid endometrial adenocarcinoma in Hong Kong women

resolves10.1016/j.bbrc.2007.03.201

MicroRNA expression profiles in head and neck cancer cell lines

resolves10.1158/0008-5472.CAN-07-1936

MicroRNA Signatures in Human Ovarian Cancer

resolves10.1038/onc.2012.20

Altered microRNA expression associated with chromosomal changes contributes to cervical carcinogenesis

resolves10.1126/science.1149460

Switching from Repression to Activation: MicroRNAs Can Up-Regulate Translation

resolves10.3892/or.2013.2400

miR-205 promotes tumor proliferation and invasion through targeting ESRRG in endometrial carcinoma

resolves10.3892/or.2014.3142

Human papilloma virus, DNA methylation and microRNA expression in cervical cancer (Review)

resolves10.1096/fj.10-157404

MicroRNA‐205 promotes keratinocyte migration <i>via</i> the lipid phosphatase SHIP2

The 2 references without a DOI — listed, not checked

no DOI — not checkedHou LJ, Zhai JJ. Aberrant expression profile of translationally controlled tumor protein and tumor-suppressive microRNAs in cervical cancer. J BUON. 2015;20(6):1504–9.

no DOI — not checkedKinsel LB, Szabo E, Greene GL, Konrath J, Leight GS, McCarty KS Jr. Immunocytochemical analysis of estrogen receptors as a predictor of prognosis in breast cancer patients: comparison with quantitative biochemical methods. Cancer Res. 1989;49(4):1052–6.

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