Reference health
Modulation of post-stroke degenerative and regenerative processes and subacute protection by site-targeted inhibition of the alternative pathway of complement
39/39 checkable references clean · checked 2026-07-23
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 39 checked references that resolve
resolves10.4049/jimmunol.1201904
The Alternative Complement Pathway Propagates Inflammation and Injury in Murine Ischemic Stroke
resolves10.1161/CIRCULATIONAHA.112.103051
Targeting Mannose-Binding Lectin Confers Long-Lasting Protection With a Surprisingly Wide Therapeutic Window in Cerebral Ischemia
resolves10.1371/journal.pone.0008433
Genetically-Defined Deficiency of Mannose-Binding Lectin Is Associated with Protection after Experimental Stroke in Mice and Outcome in Human Stroke
resolves10.2174/156720211794520260
The Contribution of Mannose Binding Lectin to Reperfusion Injury after Ischemic Stroke
resolves10.1186/1742-2094-9-137
Complement activation in the injured central nervous system: another dual-edged sword?
resolves10.1007/s12975-011-0104-2
The Neuroprotective Effect of Genetic Mannose-Binding Lectin Deficiency Is Not Sustained in the Subacute Phase of Stroke
resolves10.1161/01.RES.0000232544.90675.42
Complement Component C3 Mediates Inflammatory Injury Following Focal Cerebral Ischemia
resolves10.4049/jimmunol.177.10.7266
Complement-Dependent P-Selectin Expression and Injury following Ischemic Stroke
resolves10.1038/sj.emboj.7601004
Complement: a novel factor in basal and ischemia‐induced neurogenesis
resolves10.4049/jimmunol.181.11.8068
A Novel Targeted Inhibitor of the Alternative Pathway of Complement and Its Therapeutic Application in Ischemia/Reperfusion Injury
resolves10.1172/JCI25208
Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection
resolves10.4049/jimmunol.160.9.4553
Blockade of Antibody-Induced Glomerulonephritis with Crry-Ig, a Soluble Murine Complement Inhibitor
resolves10.1097/00004647-199804000-00004
A Reproducible Model of Middle Cerebral Artery Occlusion in Mice: Hemodynamic, Biochemical, and Magnetic Resonance Imaging
resolves10.1016/j.bbr.2008.10.029
Evaluation of spatial memory of C57BL/6J and CD1 mice in the Barnes maze, the Multiple T-maze and in the Morris water maze
resolves10.1161/01.STR.17.6.1304
Evaluation of 2,3,5-triphenyltetrazolium chloride as a stain for detection and quantification of experimental cerebral infarction in rats.
resolves10.1038/nbt1385
Direct multiplexed measurement of gene expression with color-coded probe pairs
resolves10.1016/S0306-4522(01)00117-8
Proliferation and differentiation of progenitor cells in the cortex and the subventricular zone in the adult rat after focal cerebral ischemia
resolves10.1002/stem.306
Subventricular Zone-Derived Neural Progenitor Cells Migrate Along a Blood Vessel Scaffold Toward The Post-stroke Striatum
resolves10.1371/journal.pone.0038664
Complement Inhibition Promotes Endogenous Neurogenesis and Sustained Anti-Inflammatory Neuroprotection following Reperfused Stroke
resolves10.1073/pnas.182296499
Vascular endothelial growth factor (VEGF) stimulates neurogenesis
<i>in vitro</i>
and
<i>in vivo</i>
resolves10.1161/01.STR.0000109041.89959.4C
Preventive Antibacterial Treatment Improves the General Medical and Neurological Outcome in a Mouse Model of Stroke
resolves10.1186/1742-2094-5-5
Complement anaphylatoxin C5a neuroprotects through regulation of glutamate receptor subunit 2 in vitro and in vivo
resolves10.1002/(SICI)1097-4644(19990601)73:3<303::AID-JCB2>3.0.CO;2-2
Complement-derived anaphylatoxin C5a protects against glutamate-mediated neurotoxicity
resolves10.1097/00001756-200102120-00022
Complement anaphylatoxin C3a is selectively protective against NMDA-induced neuronal cell death
resolves10.1096/fj.13-230011
Receptor for complement peptide C3a: a therapeutic target for neonatal hypoxic‐ischemic brain injury
resolves10.1523/JNEUROSCI.5249-09.2010
Complement Component C1q Mediates Mitochondria-Driven Oxidative Stress in Neonatal Hypoxic–Ischemic Brain Injury
resolves10.1002/stem.225
Complement-Derived Anaphylatoxin C3a Regulates In Vitro Differentiation and Migration of Neural Progenitor Cells
resolves10.1177/1753425910369271
Review: Soluble innate immune pattern-recognition proteins for clearing dying cells and cellular components: implications on exacerbating or resolving inflammation
resolves10.2174/187152711794488601
Activation and Control of CNS Innate Immune Responses in Health and Diseases: A Balancing Act Finely Tuned by Neuroimmune Regulators (NIReg)
resolves10.1073/pnas.0408835103
Drusen complement components C3a and C5a promote choroidal neovascularization
resolves10.1523/JNEUROSCI.3617-10.2011
<i>Complement Receptor 2</i>Is Expressed in Neural Progenitor Cells and Regulates Adult Hippocampal Neurogenesis
The 1 reference without a DOI — listed, not checked
no DOI — not checkedKomotar RJ, Kim GH, Otten ML, Hassid B, Mocco J, Sughrue ME, et al. The role of complement in stroke therapy. Adv Exp Med Biol. 2008;632:23–33.
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