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PM2.5 exposure aggravates oligomeric amyloid beta-induced neuronal injury and promotes NLRP3 inflammasome activation in an in vitro model of Alzheimer’s disease

https://doi.org/10.1186/s12974-018-1178-5
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Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 43 checked references that resolve
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Long-Term Exposure to Concentrated Ambient PM <sub>2.5</sub> Increases Mouse Blood Pressure through Abnormal Activation of the Sympathetic Nervous System: A Role for Hypothalamic Inflammation
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resolves10.3389/fphar.2015.00262
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The NALP3 inflammasome is involved in the innate immune response to amyloid-β
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NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice
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Inflammasomes as therapeutic targets for <scp>A</scp>lzheimer's disease
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The airway epithelium nucleotide-binding domain and leucine-rich repeat protein 3 inflammasome is activated by urban particulate matter
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The Nucleotide-Binding Domain, Leucine-Rich Repeat Protein 3 Inflammasome/IL-1 Receptor I Axis Mediates Innate, but Not Adaptive, Immune Responses after Exposure to Particulate Matter under 10 μm
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Alzheimer’s Disease Risk Gene CD33 Inhibits Microglial Uptake of Amyloid Beta
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Microglia Activated with the Toll-Like Receptor 9 Ligand CpG Attenuate Oligomeric Amyloid β Neurotoxicity in in Vitro and in Vivo Models of Alzheimer’s Disease
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resolves10.1111/cns.12144
Antiepileptics Topiramate and Levetiracetam Alleviate Behavioral Deficits and Reduce Neuropathology in <scp>APP</scp>swe/<scp>PS</scp>1dE9 Transgenic Mice
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resolves10.1016/j.jes.2016.10.013
Particulate matter (PM2.5) exposure season-dependently induces neuronal apoptosis and synaptic injuries
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N-methyl-d-aspartate receptor-mediated calcium overload and endoplasmic reticulum stress are involved in interleukin-1beta-induced neuronal apoptosis in rat hippocampus
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Mechanism and Regulation of NLRP3 Inflammasome Activation
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Differential requirement for the activation of the inflammasome for processing and release of IL-1β in monocytes and macrophages
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Non-canonical inflammasome activation targets caspase-11
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NLRP3 inflammasome: From a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases
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Predictive and Prognostic Biomarkers of Respiratory Diseases due to Particulate Matter Exposure
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resolves10.1016/j.chemosphere.2015.09.023
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resolves10.1007/s00204-015-1610-x
Progression and inflammation of human myeloid leukemia induced by ambient PM2.5 exposure
The 1 reference without a DOI — listed, not checked
no DOI — not checkedSong L, Pei L, Yao S, Wu Y, Shang Y. NLRP3 inflammasome in neurological diseases, from functions to therapies. Front Cell Neurosci. 2017;11:63.
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