Every reference with a DOI in the deposited reference list resolved to a known
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The 33 checked references that resolve
resolves10.1002/eji.1830010406Suppression of experimental allergic encephalomyelitis by a synthetic polypeptide
resolves10.1002/eji.1830030506<i>In vivo and in vitro</i> immunological cross‐reactions between basic encephalitogen and synthetic basic polypeptides capable of suppressing experimental allergic encephalomyelitis
resolves10.1073/pnas.88.21.9528Cross-reactions and specificities of monoclonal antibodies against myelin basic protein and against the synthetic copolymer 1.
resolves10.1001/jamaneurol.2014.1472The Effect of Glatiramer Acetate Therapy on Functional Properties of B Cells From Patients With Relapsing-Remitting Multiple Sclerosis
resolves10.1016/j.clim.2014.02.014Identification of a B cell-dependent subpopulation of multiple sclerosis by measurements of brain-reactive B cells in the blood
resolves10.1038/srep14265The brain antigen-specific B cell response correlates with glatiramer acetate responsiveness in relapsing-remitting multiple sclerosis patients
resolves10.3389/fneur.2018.01150The Effect of Disease Modifying Therapies on Disability Progression in Multiple Sclerosis: A Systematic Overview of Meta-Analyses
resolves10.1177/1352458516687402Assessing long-term prognosis improvement as a consequence of treatment pattern changes in MS
resolves10.1136/jnnp-2016-313976Highly active immunomodulatory therapy ameliorates accumulation of disability in moderately advanced and advanced multiple sclerosis
resolves10.1001/jama.2018.20588Association of Initial Disease-Modifying Therapy With Later Conversion to Secondary Progressive Multiple Sclerosis
resolves10.1080/23744235.2016.1269190Positive predictive value of ELISpot in BAL and pleural fluid from patients with suspected pulmonary tuberculosis
resolves10.1371/journal.pone.0197825Novel M. tuberculosis specific IL-2 ELISpot assay discriminates adult patients with active or latent tuberculosis
resolves10.1073/pnas.2011249117Autoantibodies against central nervous system antigens in a subset of B cell–dominant multiple sclerosis patients
resolves10.1016/j.clim.2019.108297JNK and phosphorylated Bcl-2 predict multiple sclerosis clinical activity and glatiramer acetate therapeutic response
resolves10.2217/pgs.14.26Comparative Pharmacogenetics of Multiple Sclerosis: IFN-β Versus Glatiramer Acetate
resolves10.1093/brain/awm038Meningeal B-cell follicles in secondary progressive multiple sclerosis associate with early onset of disease and severe cortical pathology
resolves10.1093/brain/awr182Meningeal inflammation is widespread and linked to cortical pathology in multiple sclerosis
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