Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 146 checked references that resolve
resolves10.1038/1831654a0Canalization of Development and Genetic Assimilation of Acquired Characters
resolves10.1016/j.cell.2016.12.002The International Human Epigenome Consortium: A Blueprint for Scientific Collaboration and Discovery
resolves10.1038/nature11606EZH2 inhibition as a therapeutic strategy for lymphoma with EZH2-activating mutations
resolves10.1038/nrc.2016.83Maintaining cell identity: PRC2-mediated regulation of transcription and cancer
resolves10.1016/j.molcel.2013.02.013Occupying Chromatin: Polycomb Mechanisms for Getting to Genomic Targets, Stopping Transcriptional Traffic, and Staying Put
resolves10.1038/nrg2752Chromatin structure and the inheritance of epigenetic information
resolves10.1038/nrc3130A decade of exploring the cancer epigenome — biological and translational implications
resolves10.1016/j.ccr.2010.12.014Oncometabolite 2-Hydroxyglutarate Is a Competitive Inhibitor of α-Ketoglutarate-Dependent Dioxygenases
resolves10.18632/oncotarget.14150AML associated oncofusion proteins PML-RARA, AML1-ETO and CBFB-MYH11 target RUNX/ETS-factor binding sites to modulate H3ac levels and drive leukemogenesis
resolves10.1038/ng.2928Small cell carcinoma of the ovary, hypercalcemic type, displays frequent inactivating germline and somatic mutations in SMARCA4
resolves10.1038/nature13981Intracellular α-ketoglutarate maintains the pluripotency of embryonic stem cells
resolves10.1038/ng.2443Epigenomic analysis detects widespread gene-body DNA hypomethylation in chronic lymphocytic leukemia
resolves10.1038/ng.3291Whole-genome fingerprint of the DNA methylome during human B cell differentiation
resolves10.1038/leu.2014.252A B-cell epigenetic signature defines three biologic subgroups of chronic lymphocytic leukemia with clinical impact
resolves10.1038/ncomms11938Chromatin accessibility maps of chronic lymphocytic leukaemia identify subtype-specific epigenome signatures and transcription regulatory networks
resolves10.1016/j.ccr.2009.12.045PML/RARα Targets Promoter Regions Containing PU.1 Consensus and RARE Half Sites in Acute Promyelocytic Leukemia
resolves10.1002/pbc.26251DNA methylation profiling of pediatric B‐cell lymphoblastic leukemia with <i>KMT2A</i> rearrangement identifies hypomethylation at enhancer sites
resolves10.1038/nm.3832DOT1L inhibits SIRT1-mediated epigenetic silencing to maintain leukemic gene expression in MLL-rearranged leukemia
resolves10.1016/j.leukres.2013.07.016The expression of histone deacetylase 4 is associated with prednisone poor-response in childhood acute lymphoblastic leukemia
resolves10.1172/JCI78440Musashi2 sustains the mixed-lineage leukemia–driven stem cell regulatory program
resolves10.4161/epi.27322Redistribution of H3K27me3 and acetylated histone H4 upon exposure to azacitidine and decitabine results in de-repression of the AML1/ETO target gene
<i>IL3</i>
resolves10.1182/blood-2012-05-429050ERG and FLI1 binding sites demarcate targets for aberrant epigenetic regulation by AML1-ETO in acute myeloid leukemia
resolves10.1182/blood.V91.9.3134Expression of a Knocked-In AML1-ETO Leukemia Gene Inhibits the Establishment of Normal Definitive Hematopoiesis and Directly Generates Dysplastic Hematopoietic Progenitors
resolves10.1128/MCB.15.4.1974The t(8;21) Fusion Protein Interferes with AML-1B-Dependent Transcriptional Activation
resolves10.1016/j.celrep.2016.08.082The Hematopoietic Transcription Factors RUNX1 and ERG Prevent AML1-ETO Oncogene Overexpression and Onset of the Apoptosis Program in t(8;21) AMLs
resolves10.1016/j.celrep.2014.08.024Identification of a Dynamic Core Transcriptional Network in t(8;21) AML that Regulates Differentiation Block and Self-Renewal
resolves10.1038/sj.leu.2403396Expression of AML1-ETO in human myelomonocytic cells selectively inhibits granulocytic differentiation and promotes their self-renewal
resolves10.1038/86515AML1–ETO downregulates the granulocytic differentiation factor C/EBPα in t(8;21) myeloid leukemia
resolves10.1182/blood-2013-03-487884Global methylation analysis identifies prognostically important epigenetically inactivated tumor suppressor genes in multiple myeloma
resolves10.1101/gr.180240.114Whole-epigenome analysis in multiple myeloma reveals DNA hypermethylation of B cell-specific enhancers
resolves10.1182/blood-2010-04-279539Aberrant global methylation patterns affect the molecular pathogenesis and prognosis of multiple myeloma
resolves10.1182/blood-2010-07-298349The MMSET histone methyl transferase switches global histone methylation and alters gene expression in t(4;14) multiple myeloma cells
resolves10.1158/1535-7163.MCT-12-0721Development of Gene Expression–Based Score to Predict Sensitivity of Multiple Myeloma Cells to DNA Methylation Inhibitors
resolves10.1182/blood-2015-09-665018Panobinostat plus bortezomib and dexamethasone in previously treated multiple myeloma: outcomes by prior treatment
resolves10.18632/oncotarget.10033Epimutational profile of hematologic malignancies as attractive target for new epigenetic therapies
resolves10.1371/journal.pone.0111840Synergistic Anti-Tumor Activity of EZH2 Inhibitors and Glucocorticoid Receptor Agonists in Models of Germinal Center Non-Hodgkin Lymphomas
resolves10.5306/wjco.v5.i3.509Preventing breast cancer in LMICs via screening and/or early detection: The real and the surreal
resolves10.1038/ncomms6899Methylome sequencing in triple-negative breast cancer reveals distinct methylation clusters with prognostic value
resolves10.4161/epi.22561<i>BRCA1</i>epigenetic inactivation predicts sensitivity to platinum-based chemotherapy in breast and ovarian cancer
resolves10.1200/JCO.2010.30.1010<i>BRCA1</i> CpG Island Hypermethylation Predicts Sensitivity to Poly(Adenosine Diphosphate)- Ribose Polymerase Inhibitors
resolves10.1186/1471-2407-9-217Prognostic relevance of Wnt-inhibitory factor-1 (WIF1) and Dickkopf-3 (DKK3) promoter methylation in human breast cancer
resolves10.1007/s10549-007-9620-xQuantitative hypermethylation of a small panel of genes augments the diagnostic accuracy in fine-needle aspirate washings of breast lesions
resolves10.1038/srep33435DNA methylation profile of triple negative breast cancer-specific genes comparing lymph node positive patients to lymph node negative patients
resolves10.1158/0008-5472.CAN-08-3907Global Histone Modifications in Breast Cancer Correlate with Tumor Phenotypes, Prognostic Factors, and Patient Outcome
resolves10.1007/s10549-014-3089-1Association of H3K9me3 and H3K27me3 repressive histone marks with breast cancer subtypes in the Nurses’ Health Study
resolves10.2217/epi-2016-0015H3K4 Acetylation, H3K9 Acetylation and H3K27 Methylation in Breast Tumor Molecular Subtypes
resolves10.1186/s13148-016-0201-xGlobal histone modification profiling reveals the epigenomic dynamics during malignant transformation in a four-stage breast cancer model
resolves10.1038/ncomms10751RUNX1 prevents oestrogen-mediated AXIN1 suppression and β-catenin activation in ER-positive breast cancer
resolves10.1186/gb-2007-8-10-r214MicroRNA expression profiling of human breast cancer identifies new markers of tumor subtype
resolves10.1186/1471-2164-14-643Integrated genomic analysis of triple-negative breast cancers reveals novel microRNAs associated with clinical and molecular phenotypes and sheds light on the pathways they control
resolves10.1038/nature12108The shaping and functional consequences of the microRNA landscape in breast cancer
resolves10.1186/bcr3127Expression profiling of cancerous and normal breast tissues identifies microRNAs that are differentially expressed in serum from patients with (metastatic) breast cancer and healthy volunteers
resolves10.1007/s13402-015-0239-3Identification of miR-10b, miR-26a, miR-146a and miR-153 as potential triple-negative breast cancer biomarkers
resolves10.1186/bcr2257MicroRNA signatures predict oestrogen receptor, progesterone receptor and HER2/neureceptor status in breast cancer
resolves10.1038/onc.2014.298Progesterone downregulation of miR-141 contributes to expansion of stem-like breast cancer cells through maintenance of progesterone receptor and Stat5a
resolves10.3892/etm.2013.915miR-342 is associated with estrogen receptor-α expression and response to tamoxifen in breast cancer
resolves10.1007/s10549-014-3037-0Exosomal miR-221/222 enhances tamoxifen resistance in recipient ER-positive breast cancer cells
resolves10.2144/000113837Quantitative Analysis of Micrornas in Tissue Microarrays by in Situ Hybridization
resolves10.1038/jhg.2016.89Recent trends in microRNA research into breast cancer with particular focus on the associations between microRNAs and intrinsic subtypes
resolves10.1200/JCO.2013.55.0491Prognostic Value of Tumor-Infiltrating Lymphocytes in Triple-Negative Breast Cancers From Two Phase III Randomized Adjuvant Breast Cancer Trials: ECOG 2197 and ECOG 1199
resolves10.1258/ebm.2011.011007Tumor-infiltrating immune cells and prognosis: the potential link between conventional cancer therapy and immunity
resolves10.1038/nrc3239The blockade of immune checkpoints in cancer immunotherapy
resolves10.1016/j.immuni.2016.10.021Transcriptional Landscape of Human Tissue Lymphocytes Unveils Uniqueness of Tumor-Infiltrating T Regulatory Cells
resolves10.1126/science.aah6893PI3K pathway regulates ER-dependent transcription in breast cancer through the epigenetic regulator KMT2D
resolves10.1038/bjc.2011.156A phase II study of the histone deacetylase inhibitor vorinostat combined with tamoxifen for the treatment of patients with hormone therapy-resistant breast cancer
resolves10.1080/2162402X.2016.1219008The interplay of epigenetic therapy and immunity in locally recurrent or metastatic estrogen receptor-positive breast cancer: Correlative analysis of ENCORE 301, a randomized, placebo-controlled phase II trial of exemestane with or without entinostat
resolves10.1016/j.ajhg.2010.01.013Germline Nonsense Mutation and Somatic Inactivation of SMARCA4/BRG1 in a Family with Rhabdoid Tumor Predisposition Syndrome
resolves10.1097/PAS.0b013e3182196a39Nonsense Mutation and Inactivation of SMARCA4 (BRG1) in an Atypical Teratoid/Rhabdoid Tumor Showing Retained SMARCB1 (INI1) Expression
resolves10.1002/gcc.2870020308Malignant rhabdoid tumor: A highly malignant childhood tumor with minimal karyotypic changes
resolves10.1073/pnas.250492697Haploinsufficiency of Snf5 (integrase interactor 1) predisposes to malignant rhabdoid tumors in mice
resolves10.1038/sj.onc.1205706Re-expression of hSNF5/INI1/BAF47 in pediatric tumor cells leads to G1arrest associated with induction of p16ink4a and activation of RB
resolves10.1158/0008-5472.CAN-05-1896Loss of the
<i>hSNF5</i>
Gene Concomitantly Inactivates p21CIP/WAF1 and p16INK4a Activity Associated with Replicative Senescence in A204 Rhabdoid Tumor Cells
resolves10.1073/pnas.0509014102Inactivation of the Snf5 tumor suppressor stimulates cell cycle progression and cooperates with p53 loss in oncogenic transformation
resolves10.1097/NEN.0b013e31822146cap16<sup>INK4A</sup>and p14<sup>ARF</sup>Tumor Suppressor Pathways Are Deregulated in Malignant Rhabdoid Tumors
resolves10.1038/nm.2251Loss of the tumor suppressor Snf5 leads to aberrant activation of the Hedgehog-Gli pathway
resolves10.1038/onc.2013.37Activation of β-catenin/TCF targets following loss of the tumor suppressor SNF5
resolves10.1073/pnas.1303800110Durable tumor regression in genetically altered malignant rhabdoid tumors by inhibition of methyltransferase EZH2
resolves10.1002/pbc.26218Initial testing (stage 1) of tazemetostat (EPZ‐6438), a novel EZH2 inhibitor, by the Pediatric Preclinical Testing Program
resolves10.1172/JCI64400A remarkably simple genome underlies highly malignant pediatric rhabdoid cancers
resolves10.1002/pbc.24315Absence of oncogenic canonical pathway mutations in aggressive pediatric rhabdoid tumors
resolves10.1016/S1470-2045(15)70114-2Molecular subgroups of atypical teratoid rhabdoid tumours in children: an integrated genomic and clinicopathological analysis
resolves10.1016/j.ccell.2016.02.001Atypical Teratoid/Rhabdoid Tumors Are Comprised of Three Epigenetic Subgroups with Distinct Enhancer Landscapes
resolves10.1038/nmeth.1557Development and applications of single-cell transcriptome analysis
resolves10.1038/nbt.2282Full-length mRNA-Seq from single-cell levels of RNA and individual circulating tumor cells
resolves10.1038/nrg3542Single-cell sequencing-based technologies will revolutionize whole-organism science
resolves10.1186/s13059-016-0944-xSingle-cell epigenomics: powerful new methods for understanding gene regulation and cell identity
resolves10.1126/science.1169786The Nuclear DNA Base 5-Hydroxymethylcytosine Is Present in Purkinje Neurons and the Brain
resolves10.1186/s13059-017-1177-3cepip: context-dependent epigenomic weighting for prioritization of regulatory variants and disease-associated genes
resolves10.1186/s13072-015-0050-4Making sense of GWAS: using epigenomics and genome engineering to understand the functional relevance of SNPs in non-coding regions of the human genome
resolves10.1136/bmjopen-2015-010243Other side of the coin for personalised medicine and healthcare: content analysis of ‘personalised’ practices in the literature
resolves10.1101/gr.6665407Prediction of individual genetic risk to disease from genome-wide association studies
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