Every reference with a DOI in the deposited reference list resolved to a known
work in Crossref or DataCite at the dated check, and none carried a retraction,
withdrawal, or removal notice.
The 66 checked references that resolve
resolves10.1038/nri2194The Drosophila systemic immune response: sensing and signalling during bacterial and fungal infections
resolves10.1006/bbrc.1995.1477Signals from the IL-1 Receptor Homolog, Toll, Can Activate an Immune Response in a Drosophila Hemocyte Cell Line
resolves10.1016/S0092-8674(00)80172-5The Dorsoventral Regulatory Gene Cassette spätzle/Toll/cactus Controls the Potent Antifungal Response in Drosophila Adults
resolves10.1038/41131A human homologue of the Drosophila Toll protein signals activation of adaptive immunity
resolves10.1074/jbc.M313856200Differential Activation of the NF-κB-like Factors Relish and Dif in Drosophila melanogaster by Fungi and Gram-positive Bacteria
resolves10.1242/dmm.000307Models of infectious diseases in the fruit fly <i>Drosophila melanogaster</i>
resolves10.1126/science.1070216Requirement for a Peptidoglycan Recognition Protein (PGRP) in Relish Activation and Antibacterial Immune Responses in
<i>Drosophila</i>
resolves10.1038/nature734The Drosophila immune response against Gram-negative bacteria is mediated by a peptidoglycan recognition protein
resolves10.1038/nature735Functional genomic analysis of phagocytosis and identification of a Drosophila receptor for E. coli
resolves10.1038/ni1356PGRP-LC and PGRP-LE have essential yet distinct functions in the drosophila immune response to monomeric DAP-type peptidoglycan
resolves10.1073/pnas.0404952102<i>Drosophila</i>
peptidoglycan recognition protein LC (PGRP-LC) acts as a signal-transducing innate immune receptor
resolves10.1101/gad.203301Mutations in the
<i>Drosophila dTAK1</i>
gene reveal a conserved function for MAPKKKs in the control of rel/NF-κB-dependent innate immune responses
resolves10.1038/79801Role of Drosophila IKKγ in a Toll-independent antibacterial immune response
resolves10.1101/gad.856901The antibacterial arm of the <i>Drosophila</i> innate immune response requires an IκB kinase
resolves10.1016/S0960-9822(02)00873-4Inducible Expression of Double-Stranded RNA Reveals a Role for dFADD in the Regulation of the Antibacterial Response in Drosophila Adults
resolves10.1038/ni1388Host-pathogen interactions in drosophila: new tricks from an old friend
resolves10.1038/ni1019Entry is a rate-limiting step for viral infection in a Drosophila melanogaster model of pathogenesis
resolves10.1038/ni1237The Jak-STAT signaling pathway is required but not sufficient for the antiviral response of drosophila
resolves10.1128/AEM.02248-07<i>Drosophila melanogaster</i>
Mounts a Unique Immune Response to the Rhabdovirus
<i>Sigma virus</i>
resolves10.1038/ni1335Essential function in vivo for Dicer-2 in host defense against RNA viruses in drosophila
resolves10.1038/nature07712Antiviral immunity in Drosophila requires systemic RNA interference spread
resolves10.1038/ni.1664The DExD/H-box helicase Dicer-2 mediates the induction of antiviral activity in drosophila
resolves10.1159/000149489Cricket Paralysis Virus Replicates in Cultured Drosophila Cells
resolves10.1159/000149886The Isolation of Cricket Paralysis Virus from the Emperor Gum Moth, <i>Antheraea eucalypti</i> Scott, and its Infectivity towards a Range of Insect Species
resolves10.1101/gad.1482006The RNA silencing endonuclease Argonaute 2 mediates specific antiviral immunity in <i>Drosophila melanogaster</i>
resolves10.1038/sj.onc.1208156Increased expression of Drosophila tetraspanin, Tsp68C, suppresses the abnormal proliferation of ytr-deficient and Ras/Raf-activated hemocytes
resolves10.1099/0022-1317-35-2-393End-point Dilution and Plaque Assay Methods for Titration of Cricket Paralysis Virus in Cultured Drosophila Cells
resolves10.1534/genetics.107.085449Genetic Variation Affecting Host–Parasite Interactions: Different Genes Affect Different Aspects of Sigma Virus Replication and Transmission in <i>Drosophila melanogaster</i>
resolves10.1038/ng1314A complementary transposon tool kit for Drosophila melanogaster using P and piggyBac
resolves10.1096/fj.06-7907comInfection‐induced proteolysis of PGRP‐LC controls the IMD activation and melanization cascades in
<i>Drosophila</i>
resolves10.1098/rspb.1998.0324Phylogeny and PCR–based classification of Wolbachia strains using wsp gene sequences
resolves10.1006/viro.1996.0610Sequence Analysis of the Bicistronic Drosophila X Virus Genome Segment A and Its Encoded Polypeptides
resolves10.1073/pnas.97.7.3376The phytopathogenic bacteria
<i>Erwinia carotovora</i>
infects
<i>Drosophila</i>
and activates an immune response
resolves10.1016/j.cub.2006.11.026Psidin Is Required in Drosophila Blood Cells for Both Phagocytic Degradation and Immune Activation of the Fat Body
resolves10.1073/pnas.95.24.14337Analysis of the
<i>Drosophila</i>
host defense in
<i>domino</i>
mutant larvae, which are devoid of hemocytes
resolves10.1128/JVI.75.2.996-1003.2001Central Role of Hemocytes in
<i>Autographa californica M</i>
Nucleopolyhedrovirus Pathogenesis in
<i>Heliothis virescens</i>
and
<i>Helicoverpa zea</i>
resolves10.1534/genetics.107.083782Identification of Drosophila Mutants Altering Defense of and Endurance to <i>Listeria monocytogenes</i> Infection
resolves10.1016/S1534-5807(01)00059-4Drosophila Immune Deficiency (IMD) Is a Death Domain Protein that Activates Antibacterial Defense and Can Promote Apoptosis
resolves10.1042/bj3590099A Drosophila haemocyte-specific protein, hemolectin, similar to human von Willebrand factor
The 6 references without a DOI — listed, not checked
no DOI — not checkedA nonoccluded virus of the field crickets <italic>Teleogryllus oceanicus</italic> and <italic>T. commodus</italic> (Orthoptera: Grylllidae).
no DOI — not checkedFields' virology.
no DOI — not checkedIdentification, Pathology, Structure, and Replication of Insect Picornaviruses.
no DOI — not checkedPherokine-2 and -3.
no DOI — not checkedCell lines derived from late embryonic stages of Drosophila melanogaster.
no DOI — not checkedA simple method of estimating fifty percent endpoints.
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