Reference health

Vitamin A at the interface of host–commensal–pathogen interactions

https://doi.org/10.1371/journal.ppat.1007750
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24/24 checkable references clean · checked 2026-07-23

Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.

1 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.

The 24 checked references that resolve
resolves10.3390/nu10091128
Modulation of Intestinal Immune and Barrier Functions by Vitamin A: Implications for Current Understanding of Malnutrition and Enteric Infections in Children
resolves10.3390/nu9010068
Retinoic Acid and Its Role in Modulating Intestinal Innate Immunity
resolves10.1084/jem.20070590
A functionally specialized population of mucosal CD103+ DCs induces Foxp3+ regulatory T cells via a TGF-β– and retinoic acid–dependent mechanism
resolves10.1016/j.immuni.2011.03.003
Essential Role for Retinoic Acid in the Promotion of CD4+ T Cell Effector Responses via Retinoic Acid Receptor Alpha
resolves10.1038/mi.2017.91
Intestinal epithelial cell-specific RARα depletion results in aberrant epithelial cell homeostasis and underdeveloped immune system
resolves10.1126/scitranslmed.aal4069
The effects of micronutrient deficiencies on bacterial species from the human gut microbiota
resolves10.1016/j.immuni.2014.01.003
The Cytokine IL-22 Promotes Pathogen Colonization by Suppressing Related Commensal Bacteria
resolves10.1084/jem.20180427
IgA regulates the composition and metabolic function of gut microbiota by promoting symbiosis between bacteria
resolves10.1038/nature26157
A new class of synthetic retinoid antibiotics effective against bacterial persisters
resolves10.1016/j.immuni.2018.11.018
Commensals Suppress Intestinal Epithelial Cell Retinoic Acid Synthesis to Regulate Interleukin-22 Activity and Prevent Microbial Dysbiosis
resolves10.1016/S0021-9258(19)83875-9
Transcriptional regulation of plasma protein synthesis during inflammation.
resolves10.1016/0167-5699(94)90137-6
The acute phase response
resolves10.1016/S0022-2275(20)42007-3
Effects of acute inflammation on plasma retinol, retinol-binding protein, and its mRNA in the liver and kidneys of vitamin A-sufficient rats
resolves10.7554/eLife.03206
Serum amyloid A is a retinol binding protein that transports retinol during bacterial infection
resolves10.1016/j.cell.2009.09.033
Induction of Intestinal Th17 Cells by Segmented Filamentous Bacteria
resolves10.1128/IAI.00201-15
Vitamin A-Deficient Hosts Become Nonsymptomatic Reservoirs of Escherichia coli-Like Enteric Infections
resolves10.1126/science.1247606
Adaptation of Innate Lymphoid Cells to a Micronutrient Deficiency Promotes Type 2 Barrier Immunity
resolves10.3945/jn.113.186569
Streptococcus pneumoniae–Induced Pneumonia and Citrobacter rodentium–Induced Gut Infection Differentially Alter Vitamin A Concentrations in the Lung and Liver of Mice
resolves10.1128/IAI.02191-14
Retinoid Levels Influence Enterohemorrhagic Escherichia coli Infection and Shiga Toxin 2 Susceptibility in Mice
resolves10.1038/ni.3734
Vitamin A mediates conversion of monocyte-derived macrophages into tissue-resident macrophages during alternative activation
resolves10.1086/521195
Vitamin A Supplementation Has Divergent Effects on Norovirus Infections and Clinical Symptoms among Mexican Children
resolves10.1038/srep25835
Antiviral effect of vitamin A on norovirus infection via modulation of the gut microbiome
resolves10.1038/nature13158
Maternal retinoids control type 3 innate lymphoid cells and set the offspring immunity
resolves10.1126/science.aad2571
The maternal microbiota drives early postnatal innate immune development
The 1 reference without a DOI — listed, not checked
no DOI — not checkedVitamin A supplementation for preventing morbidity and mortality in children from six months to five years of age
What this badge says. CiteStamped means the CHECKABLE references of this work were clean at the dated check: each resolved to a known work in a public registry, and none carried a retraction notice at that time. It says nothing about the quality, findings, or importance of the work itself, and nothing about references deposited without a DOI.

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