Reference health
Age-induced NLRP3 Inflammasome Over-activation Increases Lethality of SARS-CoV-2 Pneumonia in Elderly Patients
45/45 checkable references clean · checked 2026-07-24
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 45 checked references that resolve
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Clinical course and risk factors for mortality of adult inpatients with COVID-19 in Wuhan, China: a retrospective cohort study
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The Role of Cytokines including Interleukin-6 in COVID-19 induced Pneumonia and Macrophage Activation Syndrome-Like Disease
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COVID-19 illness in native and immunosuppressed states: A clinical–therapeutic staging proposal
resolves10.1172/JCI32285
IL-1R1/MyD88 signaling and the inflammasome are essential in pulmonary inflammation and fibrosis in mice
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SARS-Coronavirus Open Reading Frame-8b triggers intracellular stress pathways and activates NLRP3 inflammasomes
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The NLRP3 inflammasome: molecular activation and regulation to therapeutics
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The Roles of the NLRP3 Inflammasome in Neurodegenerative and Metabolic Diseases and in Relevant Advanced Therapeutic Interventions
resolves10.1038/s41419-019-1413-8
Recent advances in the mechanisms of NLRP3 inflammasome activation and its inhibitors
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The NLRP3 Inflammasome: An Overview of Mechanisms of Activation and Regulation
resolves10.1016/j.virol.2015.08.010
Severe acute respiratory syndrome coronavirus E protein transports calcium ions and activates the NLRP3 inflammasome
resolves10.3390/ijms21093179
SARS Unique Domain (SUD) of Severe Acute Respiratory Syndrome Coronavirus Induces NLRP3 Inflammasome-Dependent CXCL10-Mediated Pulmonary Inflammation
resolves10.1371/journal.ppat.1005155
The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis
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SARS-Coronavirus Open Reading Frame-3a drives multimodal necrotic cell death
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Severe Acute Respiratory Syndrome Coronavirus Viroporin 3a Activates the NLRP3 Inflammasome
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Severe acute respiratory syndrome Coronavirus ORF3a protein activates the NLRP3 inflammasome by promoting TRAF3‐dependent ubiquitination of ASC
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Systematic Comparison of Two Animal-to-Human Transmitted Human Coronaviruses: SARS-CoV-2 and SARS-CoV
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Computational Inference of Selection Underlying the Evolution of the Novel Coronavirus, Severe Acute Respiratory Syndrome Coronavirus 2
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resolves10.1136/thoraxjnl-2016-209313
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A Vicious Circle of Alveolar Macrophages and Fibroblasts Perpetuates Pulmonary Fibrosis via CCL18
resolves10.1165/rcmb.2015-0222OC
Age-Dependent Susceptibility to Pulmonary Fibrosis Is Associated with NLRP3 Inflammasome Activation
resolves10.1126/science.1112125
Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging
resolves10.1038/nature10992
Mitochondrial DNA that escapes from autophagy causes inflammation and heart failure
resolves10.1038/ni.1980
Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome
resolves10.1016/j.ejim.2010.03.005
Targeting IL-1β in disease; the expanding role of NLRP3 inflammasome
resolves10.1038/nm1578
Telomere dysfunction induces environmental alterations limiting hematopoietic stem cell function and engraftment
resolves10.1016/j.celrep.2018.02.071
Telomere Dysfunction Disturbs Macrophage Mitochondrial Metabolism and the NLRP3 Inflammasome through the PGC-1α/TNFAIP3 Axis
resolves10.1189/jlb.4A0214-093RR
Characterization of lung inflammation and its impact on macrophage function in aging
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Identification of Oxidative Stress and Toll-like Receptor 4 Signaling as a Key Pathway of Acute Lung Injury
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Dampened NLRP3-mediated inflammation in bats and implications for a special viral reservoir host
The 2 references without a DOI — listed, not checked
no DOI — not checkedMadsbad S (2020). COVID-19 Infection in People with Diabetes. Touch Endocrinology (in press)
no DOI — not checkedConti P, Ronconi G, Caraffa A, Gallenga C, Ross R, Frydas I, et al (2020). Induction of Pro-Inflammatory Cytokines (IL-1 and IL-6) and Lung Inflammation by Coronavirus-19 (COVI-19 or SARS-CoV-2): Anti-Inflammatory Strategies. J Biol Regul Homeost Agents, 14;34(2):1
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