Reference health
Renal sodium and magnesium reabsorption are not coupled in a mouse model of Gordon syndrome
30/30 checkable references clean · checked 2026-07-22
Every reference with a DOI in the deposited reference list resolved to a known work in Crossref or DataCite at the dated check, and none carried a retraction, withdrawal, or removal notice.
2 without a DOI — not checked. A reference deposited without a DOI is never matched by title or guessed at; it stays outside the checked set, and this line discloses that.
The 30 checked references that resolve
resolves10.1371/journal.pgen.1004267
CNNM2 Mutations Cause Impaired Brain Development and Seizures in Patients with Hypomagnesemia
resolves10.1152/ajprenal.00493.2013
STE20/SPS1-related proline/alanine-rich kinase (SPAK) is critical for sodium reabsorption in isolated, perfused thick ascending limb
resolves10.1152/ajpcell.00074.2010
On the substrate recognition and negative regulation of SPAK, a kinase modulating Na<sup>+</sup>-K<sup>+</sup>-2Cl<sup>−</sup> cotransport activity
resolves10.1152/ajprenal.00508.2012
Regulation of the renal Na<sup>+</sup>-Cl<sup>−</sup>cotransporter by phosphorylation and ubiquitylation
resolves10.1111/imj.1970.19.4.287
Hypertension and Severe Hyperkalaemia Associated with Suppression of Renin and Aldosterone and Completely Reversed by Dietary Sodium Restriction
resolves10.1172/JCI78558
Integrated compensatory network is activated in the absence of NCC phosphorylation
resolves10.1681/ASN.2016090948
Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules
resolves10.1016/0002-9343(86)90335-9
Thiazide treatment of hypertension: Effects of thiazide diuretics on serum potassium, magnesium, and ventricular ectopy
resolves10.1152/ajpcell.00289.2011
Human gene <i>SLC41A1</i> encodes for the Na<sup>+</sup>/Mg<sup>2+</sup> exchanger
resolves10.1006/meth.2001.1262
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method
resolves10.1210/jcem.87.7.8449
Pseudohypoaldosteronism Type II: Marked Sensitivity to Thiazides, Hypercalciuria, Normomagnesemia, and Low Bone Mineral Density
resolves10.1016/j.cmet.2011.07.009
A SPAK Isoform Switch Modulates Renal Salt Transport and Blood Pressure
resolves10.1074/jbc.M510042200
WNK1 Regulates Phosphorylation of Cation-Chloride-coupled Cotransporters via the STE20-related Kinases, SPAK and OSR1
resolves10.1172/JCI24134
Enhanced passive Ca2+ reabsorption and reduced Mg2+ channel abundance explains thiazide-induced hypocalciuria and hypomagnesemia
resolves10.1242/jcs.025312
Activation of the thiazide-sensitive Na+-Cl– cotransporter by the WNK-regulated kinases SPAK and OSR1
resolves10.1186/1471-2199-7-3
The RIN: an RNA integrity number for assigning integrity values to RNA measurements
resolves10.1074/jbc.273.44.29150
Phenotype Resembling Gitelman's Syndrome in Mice Lacking the Apical Na+-Cl− Cotransporter of the Distal Convoluted Tubule
resolves10.1038/ng0196-24
Gitelman's variant of Barter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na–Cl cotransporter
resolves10.1016/j.ajhg.2011.02.005
CNNM2, Encoding a Basolateral Protein Required for Renal Mg2+ Handling, Is Mutated in Dominant Hypomagnesemia
resolves10.1016/j.coph.2014.12.012
Regulation of blood pressure and renal function by NCC and ENaC: lessons from genetically engineered mice
resolves10.1073/pnas.1304230110
<i>WNK1</i>
-related Familial Hyperkalemic Hypertension results from an increased expression of L-WNK1 specifically in the distal nephron
resolves10.1681/ASN.2009121295
SPAK-Knockout Mice Manifest Gitelman Syndrome and Impaired Vasoconstriction
The 2 references without a DOI — listed, not checked
no DOI — not checkedA new familial disorder characterized by hypokalemia and hypomagnesemia
no DOI — not checkedWith no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo
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