Reference health
Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein
1 of 62 checkable references need attention · checked 2026-07-22
At the dated check, the references listed below either did not resolve in Crossref or DataCite, or carried a retraction notice. Each one is shown with the registry record that put it there.
References needing attention
marked retracted — notice via Crossref, record curated by Retraction Watch10.1038/nature04533
RETRACTED ARTICLE: A specific amyloid-β protein assembly in the brain impairs memory
The 61 checked references that resolve
resolves10.1016/S0306-4522(02)00289-0
Localized decrease in serotonin transporter-immunoreactive axons in the prefrontal cortex of depressed subjects committing suicide
resolves10.1073/pnas.0911829107
Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein
resolves10.1038/sj.emboj.7601510
Lethal recessive myelin toxicity of prion protein lacking its central domain
resolves10.1037/h0023681
Novelty, arousal, and the reinforcement of diversive exploration in the rat.
resolves10.1523/JNEUROSCI.2055-09.2009
Aβ Immunotherapy Protects Morphology and Survival of Adult-Born Neurons in Doubly Transgenic APP/PS1 Mice
resolves10.1523/JNEUROSCI.19-12-04867.1999
Expression of Human Apolipoprotein E3 or E4 in the Brains of<i>Apoe<sup>−/−</sup></i>Mice: Isoform-Specific Effects on Neurodegeneration
resolves10.1523/JNEUROSCI.1697-05.2005
β-Amyloid Immunotherapy Prevents Synaptic Degeneration in a Mouse Model of Alzheimer's Disease
resolves10.1038/6374
Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic mice
resolves10.1038/35050103
A learning deficit related to age and β-amyloid plaques in a mouse model of Alzheimer's disease
resolves10.1126/science.1110837
Anchorless Prion Protein Results in Infectious Amyloid Disease Without Clinical Scrapie
resolves10.1523/JNEUROSCI.0277-04.2004
Fyn Kinase Modulates Synaptotoxicity, But Not Aberrant Sprouting, in Human Amyloid Precursor Protein Transgenic Mice
resolves10.1038/nn1372
Natural oligomers of the amyloid-β protein specifically disrupt cognitive function
resolves10.1016/0022-510X(87)90057-8
A quantitative morphometric analysis of the neuronal and synaptic content of the frontal and temporal cortex in patients with Alzheimer's disease
resolves10.1523/JNEUROSCI.4650-09.2009
Rho-Associated Kinase II (ROCKII) Limits Axonal Growth after Trauma within the Adult Mouse Spinal Cord
resolves10.1523/JNEUROSCI.21-13-04691.2001
Age-Related Impairment of Synaptic Transmission But Normal Long-Term Potentiation in Transgenic Mice that Overexpress the Human APP695SWE Mutant Form of Amyloid Precursor Protein
resolves10.1016/j.jneumeth.2005.12.021
Semi-automated quantification of axonal densities in labeled CNS tissue
resolves10.1016/j.nbd.2003.11.011
Normal induction but accelerated decay of LTP in APP + PS1 transgenic mice
resolves10.1073/pnas.0813349106
Reduction of cholesterol synthesis in the mouse brain does not affect amyloid formation in Alzheimer's disease, but does extend lifespan
resolves10.1073/pnas.96.6.3228
Plaque-independent disruption of neural circuits in Alzheimer’s disease mouse models
resolves10.1093/hmg/ddh019
Mutant presenilins specifically elevate the levels of the 42 residue β-amyloid peptide in vivo: evidence for augmentation of a 42-specific γ secretase
resolves10.1016/j.neuron.2004.10.015
Nogo-66 Receptor Prevents Raphespinal and Rubrospinal Axon Regeneration and Limits Functional Recovery from Spinal Cord Injury
resolves10.1523/JNEUROSCI.23-09-03837.2003
Conditional Expression in Corticothalamic Efferents Reveals a Developmental Role for Nicotinic Acetylcholine Receptors in Modulation of Passive Avoidance Behavior
resolves10.1073/pnas.95.11.6448
Diffusible, nonfibrillar ligands derived from Aβ
<sub>1–42</sub>
are potent central nervous system neurotoxins
resolves10.1016/S0006-8993(99)01698-4
Alterations in synaptic transmission and long-term potentiation in hippocampal slices from young and aged PDAPP mice
resolves10.1038/nature07761
Cellular prion protein mediates impairment of synaptic plasticity by amyloid-β oligomers
resolves10.1038/sj.emboj.7601507
Neonatal lethality in transgenic mice expressing prion protein with a deletion of residues 105–125
resolves10.1523/JNEUROSCI.2828-04.2004
Blockade of Nogo-66, Myelin-Associated Glycoprotein, and Oligodendrocyte Myelin Glycoprotein by Soluble Nogo-66 Receptor Promotes Axonal Sprouting and Recovery after Spinal Injury
resolves10.1016/j.mcn.2004.12.008
Transgenic inhibition of Nogo-66 receptor function allows axonal sprouting and improved locomotion after spinal injury
resolves10.1016/j.neuron.2009.01.017
An Unbiased Expression Screen for Synaptogenic Proteins Identifies the LRRTM Protein Family as Synaptic Organizers
resolves10.1523/JNEUROSCI.4218-08.2008
Amyloid Pathology Is Associated with Progressive Monoaminergic Neurodegeneration in a Transgenic Mouse Model of Alzheimer's Disease
resolves10.1126/science.1090187
Depleting Neuronal PrP in Prion Infection Prevents Disease and Reverses Spongiosis
resolves10.1523/JNEUROSCI.20-02-00771.2000
BDNF Promotes the Regenerative Sprouting, But Not Survival, of Injured Serotonergic Axons in the Adult Rat Brain
resolves10.1007/BF02780662
129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normal
resolves10.1097/00005072-199207000-00003
Three-Dimensional Analysis of the Relationship Between Synaptic Pathology and Neuropil Threads in Alzheimer Disease
resolves10.1523/JNEUROSCI.5215-08.2009
Amyloid β-Induced Neuronal Hyperexcitability Triggers Progressive Epilepsy
resolves10.1074/jbc.274.10.6483
Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain
resolves10.1016/0165-0270(84)90007-4
Developments of a water-maze procedure for studying spatial learning in the rat
resolves10.1016/S0002-9440(10)64839-0
Astroglial Expression of Human α1-Antichymotrypsin Enhances Alzheimer-like Pathology in Amyloid Protein Precursor Transgenic Mice
resolves10.1523/JNEUROSCI.20-11-04050.2000
High-Level Neuronal Expression of Aβ<sub>1–42</sub>in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation
resolves10.1016/S0166-4328(01)00367-9
Perspectives on object-recognition memory following hippocampal damage: lessons from studies in rats
resolves10.1038/387500a0
Impaired learning and LTP in mice expressing the carboxy terminus of the Alzheimer amyloid precursor protein
resolves10.1016/j.neuron.2007.07.025
Aberrant Excitatory Neuronal Activity and Compensatory Remodeling of Inhibitory Hippocampal Circuits in Mouse Models of Alzheimer's Disease
resolves10.1523/JNEUROSCI.4504-06.2006
Subcutaneous Nogo Receptor Removes Brain Amyloid-β and Improves Spatial Memory in Alzheimer's Transgenic Mice
resolves10.1073/pnas.0609621104
Cellular prion protein regulates β-secretase cleavage of the Alzheimer's amyloid precursor protein
resolves10.1126/science.1141736
Reducing Endogenous Tau Ameliorates Amyloid ß-Induced Deficits in an Alzheimer's Disease Mouse Model
resolves10.1016/S0306-4522(03)00381-6
Electrophysiological studies on the hippocampus and prefrontal cortex assessing the effects of amyloidosis in amyloid precursor protein 23 transgenic mice
resolves10.1016/0197-4580(90)90059-9
Quantitative assessment of cortical synaptic density in Alzheimer's disease
resolves10.1038/nbt969
Time-controlled transcardiac perfusion cross-linking for the study of protein interactions in complex tissues
resolves10.1016/j.neurobiolaging.2004.10.004
Prion protein (PrPc) promotes β-amyloid plaque formation
resolves10.1038/nm1782
Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
resolves10.1002/ana.20101
Progressive age‐related development of Alzheimer‐like pathology in APP/PS1 mice
resolves10.1016/j.neurobiolaging.2008.08.005
Episodic memory deficits are not related to altered glutamatergic synaptic transmission and plasticity in the CA1 hippocampus of the APPswe/PS1ΔE9-deleted transgenic mice model of β-amyloidosis
resolves10.1038/nprot.2006.116
Morris water maze: procedures for assessing spatial and related forms of learning and memory
resolves10.1038/416535a
Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo
resolves10.1089/neu.2007.0464
Ibuprofen Enhances Recovery from Spinal Cord Injury by Limiting Tissue Loss and Stimulating Axonal Growth
What this badge says. CiteStamped means the CHECKABLE references of this
work were clean at the dated check: each resolved to a known work in a public registry, and none
carried a retraction notice at that time. It says nothing about the quality, findings,
or importance of the work itself, and nothing about references deposited without a DOI.
checked 2026-07-22 — re-checked daily as this page is visited; titles and statuses come from Crossref and DataCite and are not part of the signed record
Embed this badge
Both snippets point at the live badge image and link back to this page. The badge re-renders from the daily check, so an embed never goes stale by more than a day of visits.
<a href="https://citestamp.com/citestamped/10.1523/jneurosci.0395-10.2010"><img src="https://citestamp.com/citestamped/10.1523/jneurosci.0395-10.2010/badge.svg" alt="CiteStamped reference-health badge" width="460" height="64"></a>
[](https://citestamp.com/citestamped/10.1523/jneurosci.0395-10.2010)